scholarly journals Envenoming caused by a Portuguese man-o'-war (Physalia physalis) manifesting as purpuric papules

2012 ◽  
Vol 87 (4) ◽  
pp. 644-645 ◽  
Author(s):  
Yamin José Risk ◽  
João Luiz Costa Cardoso ◽  
Vidal Haddad Junior
Keyword(s):  
Initial Phase ◽  
Late Onset ◽  
Anomalous Reaction ◽  
Physalia Physalis

We report the case of a 42-year old woman who was envenomed by a Portuguese man-o'-war (Physalia physalis). She presented an anomalous reaction manifested by purpuric papules that appeared after the initial phase of envenoming (around 24 hours later), when linear erythematous and edematous papules were observed. Late-onset reactions in accidents involving cnidarians commonly include chronic eruptions and local pigmentation.

Wrodzony niedobór biotynidazy o późnym początku – opis przypadku

2018 ◽  
Vol 22 (2) ◽  
Author(s):  
Milena Lubowicz ◽  
Magdalena Łukasik ◽  
Katarzyna Dylewska ◽  
Andrzej Kurylak
Keyword(s):  
Hearing Loss ◽  
Initial Phase ◽  
Laboratory Tests ◽  
Clinical Symptoms ◽  
Late Onset ◽  
Carrier State ◽  
Biotinidase Deficiency ◽  
Psychomotor Development ◽  
Chromosome 3 ◽  
Biotinidase Activity

Biotinidase deficiency is a genetically conditioned congenital disorder of biotin metabolism. The disease is caused by mutations in the BTD-3p25 gene, located on the short strand of chromosome 3. The BTD gene conditions proper biotinidase synthesis. So far, approximately 150 mutations of this gene have been identified. The incidence proportion is one case per 61 000 births, and the carrier state – one case per 120 births. Biotin (vit. H, B7) is essential in numerous metabolic processes. The initial phase of the disease can be acute, chronic including exacerbations/remissions, progressive, and the symptoms can appear at any age. The diagnosis of patients with late-onset disease is particularly difficult, since the symptoms suggest disorders of different nature, especially neurologic. The diagnosis is based on the analysis of clinical symptoms and laboratory tests, including biotinidase activity. The supplementation of biotin is the treatment of choice. It leads to rapid resolution of symptoms and can protect against permanent consequences of the disease such as optic atrophy, hearing loss or retarded psychomotor development.

Late-Onset Anaphylaxis to Omalizumab Reported

2006 ◽  
Vol 37 (6) ◽  
pp. 6
Author(s):  
PATRICE WENDLING
Keyword(s):  
Late Onset

Familial Patterns of Risk in Very Late-Onset Alzheimer's Disease

2003 ◽  
Author(s):  
J. M. Silverman ◽  
C. J. Smith ◽  
D. B. Marin ◽  
R. C. Mohs ◽  
C. B. Propper
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Late Onset ◽  
Familial Patterns

Some changes during the initial phase of the unit system in a large mental hospital

1966 ◽  
Author(s):  
Robert P. Barrell ◽  
Alan S. DeWolfe ◽  
Fred E. Spaner
Keyword(s):  
Initial Phase ◽  
Mental Hospital ◽  
Unit System

Discriminating early and late onset aggression from early child behaviors and family conflict

2010 ◽  
Author(s):  
Lisabeth F. DiLalla ◽  
S. J. W. Biebl ◽  
S. S. Long ◽  
S. Gheyara ◽  
K. Otto
Keyword(s):  
Family Conflict ◽  
Late Onset ◽  
Child Behaviors ◽  
Early Child

Late-Onset Stress Symptomatology Scale

2007 ◽  
Author(s):  
Lynda A. King ◽  
Daniel W. King ◽  
Kristin Vickers ◽  
Eve H. Davison ◽  
Avron Spiro
Keyword(s):  
Late Onset
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