Short-Term Memory Loss Associated with Rosuvastatin

2006 ◽  
Vol 26 (8) ◽  
pp. 1190-1192 ◽  
Author(s):  
Laura Galatti ◽  
Giovanni Polimeni ◽  
Francesco Salvo ◽  
Marcello Romani ◽  
Aurelio Sessa ◽  
...  
Keyword(s):  
Short Term Memory ◽  
Memory Loss ◽  
Short Term ◽  
Term Memory

scholarly journals High methionine, low folate and low vitamin B6/B12 (HM-LF-LV) diet causes neurodegeneration and subsequent short-term memory loss

2018 ◽  
Vol 33 (6) ◽  
pp. 1923-1934 ◽  
Author(s):  
Mohammed Nuru ◽  
Nino Muradashvili ◽  
Anuradha Kalani ◽  
David Lominadze ◽  
Neetu Tyagi
Keyword(s):  
Vitamin B6 ◽  
Short Term Memory ◽  
Memory Loss ◽  
Short Term ◽  
Term Memory

104. Do patients with end-stage renal failure and short-term memory loss have Alzheimerʼs disease

1997 ◽  
Vol 18 (4) ◽  
pp. 320
Author(s):  
P. Lass ◽  
J. R. Buscombe ◽  
A. Davenport ◽  
S. Gacinovic ◽  
D. S. Thakrar ◽  
...  
Keyword(s):  
Renal Failure ◽  
Short Term Memory ◽  
Memory Loss ◽  
Short Term ◽  
Term Memory ◽  
End Stage Renal Failure ◽  
End Stage

Drug inhibition of memory formation in chickens II. Short-term memory

1971 ◽  
Vol 178 (1053) ◽  
pp. 455-464 ◽  
Keyword(s):  
Protein Synthesis ◽  
Sodium Pump ◽  
Short Term Memory ◽  
Memory Loss ◽  
Protein Synthesis Inhibitor ◽  
Protein Synthesis Inhibitors ◽  
Short Term ◽  
Synthesis Inhibitor ◽  
Term Memory

1. Memory in day-old-chickens during the first few hours after learning can be made to decline by the prior intracranial injection of two classes of drugs. 2. Sodium pump inhibitors in increasing doses cause increasingly rapid loss of memory. 3. Protein synthesis inhibitors in increasing doses attain a maximum potency in causing memory decline and the rate may not be further accelerated by higher doses. 4. Adding a sodium pump inhibitor to the inhibition of protein synthesis increases memory loss. 5. Adding a protein synthesis inhibitor to a sodium pump inhibitor causes no further loss. 6. Therefore within a few minutes of learning a short-term memory of limited time span but independent of protein synthesis becomes supplemented and eventually replaced by a long-term storage requiring protein synthesis. The amount of long-term store is set by the amount of short-term memory. 7. The short-term store could be directly dependent on post-activation enhancement of Na + extrusion from neurons. Some physiological mechanisms by which this could be achieved and how this might activate protein synthesis are discussed.

Tocainide: A Severe Adverse Reaction

1988 ◽  
Vol 22 (2) ◽  
pp. 142-145 ◽  
Author(s):  
James M. Dunn ◽  
Paul E. Groth ◽  
Anne DeSimone
Keyword(s):  
Adverse Reaction ◽  
Short Term Memory ◽  
Memory Loss ◽  
Uneventful Recovery ◽  
Physical Status ◽  
Short Term ◽  
Severe Adverse Reaction ◽  
Premature Ventricular Contractions ◽  
Term Memory ◽  
Cardioactive Drug

Tocainide is a primary analog of lidocaine with antiarrhythmic properties used to treat ventricular rhythm disorders. A 76-year-old man with benign paroxysmal premature ventricular contractions was treated with tocainide and developed a generalized maculopapular lupoid eruption, bleeding from the lips and gingivae, vertigo, gross tremors of the extremities, fever, and short-term memory loss, which required hospitalization. The patient recovered slowly over three months with no permanent sequelae after discontinuing the drug and receiving rigorous supportive care. His excellent physical status and absence of concomitant illness contributed to an uneventful recovery. Tocainide is a potent cardioactive drug with a long biological half-life and should be used with caution.

LGI1 antibody encephalitis and psychosis

2018 ◽  
Vol 26 (6) ◽  
pp. 612-614 ◽  
Author(s):  
Dahai Wang ◽  
Qinjian Hao ◽  
Lan He ◽  
Qiang Wang
Keyword(s):  
Magnetic Resonance Imaging ◽  
Short Term Memory ◽  
Background Activity ◽  
Early Recognition ◽  
Memory Loss ◽  
Psychotic Symptoms ◽  
Resonance Imaging ◽  
Short Term ◽  
Term Memory ◽  
Hippocampal Lesions

Objective: To describe a case of leucine-rich, glioma inactivated 1 antibody-encephalitis presenting with psychosis. Methods: Case report. Results: A young man with leucine-rich, glioma inactivated 1-antibody encephalitis initially presented with acute psychotic symptoms, short-term memory loss and faciobrachial dystonic seizures. Magnetic resonance imaging revealed hippocampal lesions. Electroencephalography revealed frontotemporal slowing of background activity. Conclusion: Increased awareness of leucine-rich, glioma inactivated 1-antibody encephalitis may promote early recognition and treatment.

scholarly journals Short-term memory loss over time without retroactive stimulus interference

2008 ◽  
Vol 15 (1) ◽  
pp. 230-235 ◽  
Author(s):  
N. COWAN ◽  
A. M. AUBUCHON
Keyword(s):  
Short Term Memory ◽  
Memory Loss ◽  
Short Term ◽  
Term Memory ◽  
Over Time

scholarly journals Clothianidin and Thiacloprid Mixture Administration Induces Degenerative Damage in the Dentate Gyrus and Alteration in Short-Term Memory in Rats

2021 ◽  
Vol 2021 ◽  
pp. 1-9
Author(s):  
Alejandra Mora-Gutiérrez ◽  
Jorge Guevara ◽  
Carmen Rubio ◽  
Minerva Calvillo-Velasco ◽  
Daniela Silva-Adaya ◽  
...  
Keyword(s):  
Dentate Gyrus ◽  
Short Term Memory ◽  
Lethal Dose ◽  
Memory Loss ◽  
Memory Test ◽  
Long Term Memory ◽  
Behavioral Alterations ◽  
Short Term ◽  
Term Memory ◽  
Discharge Patterns

Neonicotinoids are pesticides that act as agonists of nicotinic receptors for acetylcholine in insects’ central nervous system (CNS). Chronic exposure to neonicotinoids in humans is related to autism, memory loss, and finger tremor. In this article, we evaluate the effect of subchronic oral administration of two neonicotinoids in the same mixture: clothianidin and thiacloprid. Decreasing doses of both pesticides were administered to rats starting from the lethal dose 50 (LD50) reported by the manufacturer. Our results indicate that the administration of three doses of decreasing amounts of LD50 (5/10, 4/10, and 3/10 LD50) resulted in 100% death in all cases. Ten administration times of 2/10 LD50 of the mixture caused only 20% of death cases after twenty-seven days, which was determined as a subchronic administration scheme. The animals administered 2/10 LD50 showed behavioral alterations after the first and second administration. Electrographic studies showed abnormal discharge patterns in the CNS. 72 h after the tenth dose, learning and memory tests were performed in the Morris water maze. Our results revealed significant decreases in permanence at the quadrant and the number of crosses ( P = 0.0447 ,   P = 0.0193 , respectively), which represent alterations in the short-term memory test, but there were no significant changes in a long-term memory test. Likewise, the brains of these animals showed tissue architecture loss, nucleosomal retraction, and a significant increase in the pycnosis of the granular neurons of the dentate gyrus analyzed at 72 h after the last dose ( P = 0.0125 ). Toxic effects and cognitive deterioration that have been found in communities living near contaminated areas are probably related to the agricultural use of neonicotinoids.

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