scholarly journals Monoamine Depletion by Reuptake Inhibitors [Expression of Concern]

2020 ◽  
Vol Volume 12 ◽  
pp. 57-58
Author(s):  
Marty Hinz ◽  
Alvin Stein ◽  
Tom Uncini
Keyword(s):  
Author(s):  
Alexander Neumeister ◽  
Nicole Praschak-Rieder ◽  
Matthäus Willeit ◽  
Jürgen Stastny ◽  
Siegfried Kasper

Author(s):  
Michael M. Morgan ◽  
MacDonald J. Christie ◽  
Thomas Steckler ◽  
Ben J. Harrison ◽  
Christos Pantelis ◽  
...  
Keyword(s):  

1996 ◽  
Vol 5 (3) ◽  
pp. 241-249 ◽  
Author(s):  
Francesco Fornai ◽  
Lucia Bassi ◽  
Maria Tilde Torracca ◽  
Maria Grazia Alessandrı̀ ◽  
Vera Scalori ◽  
...  

2003 ◽  
Vol 8 (12) ◽  
pp. 951-973 ◽  
Author(s):  
L Booij ◽  
A J W Van der Does ◽  
W J Riedel
Keyword(s):  

2016 ◽  
Vol 31 (5) ◽  
pp. 1095-1104 ◽  
Author(s):  
Katarzyna Socała ◽  
Dorota Nieoczym ◽  
Mateusz Pieróg ◽  
Agnieszka Szuster-Ciesielska ◽  
Elżbieta Wyska ◽  
...  

1979 ◽  
Vol 57 (1) ◽  
pp. 16-23 ◽  
Author(s):  
M. T. Lin

The thermal responses of three groups of control, 6-hydroxydopamine (6-OHDA) treated and 5,7-dihydroxytryptamine (5,7-DHT) treated rabbits to the administration of chlorpromazine (CPZ) were assessed at three different ambient temperatures (Ta: 2, 22, and 32 °C). Depleting catecholamines (CA) in brain with 6-OHDA produced a decrease in metabolic rate, in respiratory evaporative heat loss, and in ear blood flow at both Ta's of 2 and 22 °C, while depleting 5-hydroxytryptamine (5-HT) contents in brain with 5,7-DHT produced the opposite responses at the same Ta's. However, these amine-depleted animals maintained their rectal temperatures within normal limits over a wide range of Ta's tested. Furthermore, intraperitoneal administration of CPZ produced hypothermia at both Ta's of 2 and 22 °C. The major cause of the CPZ-induced hypothermia was an inhibition of metabolic heat production at Ta of 2 °C. At Ta of 22 °C, the CPZ-induced hypothermia was due to both a decrease in heat production and an increase in ear blood flow. However, CPZ hypothermia was attenuated in the CA-depleted animals, but was potentiated in the 5-HT-depleted animals. The data indicate that brain monoamines are involved in the central mechanisms of CPZ-induced hypothermia.


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