scholarly journals Factors Contributing to Acquire Muscle Weakness among Critical Ill Patients

2019 ◽  
Vol 10 (3) ◽  
pp. 295-314
Author(s):  
Mona Abd Elaty Atea Mohamed ◽  
Eman Fathy Amr ◽  
Manal Salah Hassan ◽  
Zeinab Hussein Bakr
Keyword(s):  
Muscle Weakness ◽  
Critical Ill Patients ◽  
Critical Ill

scholarly journals Risk Factors of ICU-Acquired Weakness in Critical Ill Patients with ECMO Support: A Retrospective Study

2021 ◽  
Author(s):  
xinyi chen ◽  
xiong lei ◽  
xin xu ◽  
Man Huang
Keyword(s):  
Risk Factors ◽  
Retrospective Study ◽  
Muscle Weakness ◽  
Ecmo Support ◽  
Case Report Form ◽  
Mechanical Ventilation Duration ◽  
Critical Ill Patients ◽  
Sum Score ◽  
Icu Acquired Weakness ◽  
Critical Ill

Abstract Background Muscle weakness is exceedingly common in critical illness patients and is well described. However, information on the risk factors of muscle weakness in critically ill patients who received extracorporeal membrane oxygenation (ECMO) support are lacking. Our study aims to clarify the risk factors of ICU-acquired weakness (ICU-AW) in ECMO patients to help early recognize potential weakness. Methods We conducted a retrospective study of critical ill patients who used ECMO during their ICU stay. ICU-AW was diagnosed at the time when patients discharged and had a muscle strength sum score < 48 out of a maximal score of 60. Baseline characters and therapy details were collected from the case report form and inspection report. Results 63 patients receiving ECMO support were divided to ICU-AW group and no ICU-AW group according to the Medical Research Council (MRC) score when discharged. A total of 50 of the 63 patients were diagnosed with ICU-AW. On logistic regression analysis, the use of adrenaline apparently increased the odds of developing ICU-AW (OR=6.8,95%CI 1.19-38.92, p=0.032), and it was independent of all other established risk factors for ICU-AW. Meanwhile, the occurrence of infection during ECMO support significantly increased the risks of ICU-AW (OR=7.3,95% CI 1.28-41.24, p=0.025) and was independently associated with the outcome of ICU-AW. Other factors such as mechanical ventilation duration, steroid use and neuromuscular blockade did not differ significantly between the groups in our analysis. Conclusion Infection during ECMO support and use of adrenaline were found independently associated with the development of ICU-AW.

scholarly journals FP263A COMPARISON BETWEEN BIOIMPEDANCE DATA, LUNG ULTRASONOGRAPHY AND FLUID ACCCUMULATION. THREE CARDS TO DETERMINATE THE STATE OF EUVOLEMIA AND FLUID OVERLOAD IN CRITICAL ILL PATIENTS

2018 ◽  
Vol 33 (suppl_1) ◽  
pp. i119-i119
Author(s):  
Francisco Javier Centellas Pérez ◽  
Mercedes Martínez Díaz ◽  
Angela Prado Mira ◽  
Agustín Ortega Cerrato ◽  
Jaime López Tendero ◽  
...  
Keyword(s):  
Fluid Overload ◽  
The State ◽  
Lung Ultrasonography ◽  
Critical Ill Patients ◽  
Critical Ill

Pantoprazole-induced Thrombocytopenia: Unresponsive to Corticosteroid and Thrombocyte Concentrate Transfusion

2021 ◽  
pp. 089719002110647
Author(s):  
Widyati ◽  
Nurul Latifah ◽  
Maya Ramadhani
Keyword(s):  
Platelet Count ◽  
Prophylactic Therapy ◽  
Immune Mechanism ◽  
Drug Induced ◽  
Stress Ulcers ◽  
Present Case Report ◽  
Once Daily ◽  
Mucosal Disease ◽  
Critical Ill Patients ◽  
Critical Ill

Introduction Pantoprazole is a proton pump inhibitor (PPI) class drug that is widely used in the treatment of SRMD (stress-related mucosal disease in critical ill patients. PPI are one class of drugs used commonly both for treatment and prophylactic therapy for stress ulcers in intensive care unit (ICU). Case We report a case of a 51-year old male who was referred to PKU Hospital. He was admitted to ICU with diagnosis of Hyperosmolar Hyperglymic State and bronchopneumonia. Thrombocytopenia was noted in admission. There was more than 70% decrease in platelet count after initiation of pantoprazole. Patient received Thrombocyte Concentrate (TC) transfusion and corticosteroid iv for several days, but only had minor increase in platelet count. The platelets recovered after stopping pantoprazole. Discussion In the present case report, another exposures to parenteral pantoprazole in a dose of 40 mg once daily reproduced the same adverse drug reaction. In comparison to lansoprazole, thrombocytopenia from pantoprazole is more severe that necessitate TC transfusion and corticosteroid trial. However, in the present case, TC transfusion and corticosteroid fail to escalate platelet count. This finding suggests probability of non-immune mechanism of pantoprazole-induced thrombocytopenia. Conclusion Pantoprazole may induce thrombocytopenia with new features that were immediately developed, resulting a decrease in platelet count >70%. The mechanism found in this case may be non-immune. Drug-induced thrombocytopenia is one of the rare complications that has to be kept in mind with the use of pantoprazole.

scholarly journals Ionized or Total Magnesium levels, what should we measure in critical ill patients?

2021 ◽  
Vol 23 (4) ◽  
Author(s):  
Giuliana Scarpati ◽  
Daniela Baldassarre ◽  
Filomena Oliva ◽  
Gabriele Pascale ◽  
Ornella Piazza
Keyword(s):  
Critical Ill Patients ◽  
Total Magnesium ◽  
Critical Ill

Critical ill patients and flight

2017 ◽  
Vol 10 (4) ◽  
pp. 1079
Author(s):  
Beuy Joob ◽  
Viroj Wiwanitkit
Keyword(s):  
Critical Ill Patients ◽  
Critical Ill

scholarly journals Secondary Bacterial Infections in Critical Ill Patients With Coronavirus Disease 2019

2020 ◽  
Vol 7 (6) ◽  
Author(s):  
Yiqi Fu ◽  
Qing Yang ◽  
Min Xu ◽  
Haishen Kong ◽  
Hongchao Chen ◽  
...  
Keyword(s):  
Intensive Care Unit ◽  
Mechanical Ventilation ◽  
Intensive Care ◽  
Nosocomial Infection ◽  
Bacterial Infection ◽  
Critically Ill Patients ◽  
Bacterial Infections ◽  
Critical Ill Patients ◽  
Prevention Of Nosocomial Infection ◽  
Critical Ill

Secondary bacterial infections occurred in 13.9% (5 of 36) of critical ill patients with coronavirus disease 2019. All 5 patients had been admitted to intensive care unit and received mechanical ventilation before developing bacterial infection. Active surveillance of culture should be performed for critically ill patients. Prevention of nosocomial infection should to be taken seriously.

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