scholarly journals A Surgical Case of a Ruptured Internal Carotid-Posterior Communicating Artery Aneurysm Forming Kissing Aneurysms with an Unruptured C2 Aneurysm

2021 ◽  
Vol 49 (6) ◽  
pp. 463-467
Author(s):  
Chihiro AKIYAMA ◽  
Mamoru TANE ◽  
Shinya KOBAYASHI ◽  
Koji HAYASAKI
Keyword(s):  
Internal Carotid ◽  
Artery Aneurysm ◽  
Posterior Communicating Artery ◽  
Surgical Case ◽  
Posterior Communicating Artery Aneurysm

An Operative Complication in a Patient with a True Posterior Communicating Artery Aneurysm: Case report and Review of the Literature

Neurosurgery ◽  
1990 ◽  
Vol 27 (4) ◽  
pp. 650-653 ◽  
Author(s):  
Tadashi Kudo
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Internal Carotid ◽  
Artery Aneurysm ◽  
Oculomotor Nerve ◽  
Posterior Communicating Artery ◽  
Review Of The Literature ◽  
Operative Complications ◽  
Posterior Communicating Artery Aneurysm ◽  
Aneurysmal Neck

Abstract Intraoperative oculomotor nerve injury in a patient with a true posterior communicating artery aneurysm is reported in detail. A comparison of internal carotid artery aneurysms at the posterior communicating artery junction with true posterior communicating artery aneurysms deserves special attention, because the vascular relationships of the aneurysm are more complex. A clip along the internal carotid artery does not occlude blood flow to the aneurysm, and the aneurysmal neck and the distal posterior communicating artery are closer to the oculomotor nerve. This is the 27th reported case of a true posterior communicating artery aneurysm. The incidence of true posterior communicating artery aneurysms ranges from 0.1 to 2.8% of all aneurysm patients. Such aneurysms constitute 4.6 and 11% of so-called posterior communicating aneurysms in two series. Difficulty associated with a preoperative diagnosis has been documented in at least 4 cases. An awareness of this rare aneurysm is stressed in order to avoid operative complications.

scholarly journals Unruptured internal carotid-posterior communicating artery aneurysm splitting the oculomotor nerve: A case report and literature review

2020 ◽  
Vol 11 ◽  
pp. 353
Author(s):  
Hirotaka Inoue ◽  
Akihito Hashiguchi ◽  
Koichi Moroki ◽  
Hajime Tokuda
Keyword(s):  
Internal Carotid ◽  
Artery Aneurysm ◽  
Underlying Mechanism ◽  
Nerve Fibers ◽  
Oculomotor Nerve ◽  
Posterior Communicating Artery ◽  
Age Related ◽  
Posterior Communicating Artery Aneurysm ◽  
Multiple Fibers ◽  
Loss Of Compactness

Background: Although it is well known that internal carotid-posterior communicating artery (ICA-PcomA) aneurysms compress the oculomotor nerve and cause nerve palsy, cases of ICA-PcomA aneurysms splitting the oculomotor nerve are extremely rare. Case Description: We present the rare case of an asymptomatic, growing, left-sided ICA-PcomA aneurysm that was confirmed to split the oculomotor nerve. We report the clinical course and discuss the underlying mechanism. The oculomotor nerve, which is an aggregate of multiple fibers, exhibits age-related loss of compactness in the arrangement of its nerve fibers. Conclusion: We speculate that injury to the nerve fibers by aneurysmal compression was avoided because of the rare phenomenon of splitting of the oculomotor nerve.

scholarly journals Delayed brain infarction caused by mechanical compression of the anterior choroidal artery after coil embolization for a large internal carotid-posterior communicating artery aneurysm: A case report

2020 ◽  
Vol 8 ◽  
pp. 2050313X2093375
Author(s):  
Naoki Wakuta ◽  
Satoshi Yamamoto
Keyword(s):  
Coil Embolization ◽  
Internal Carotid ◽  
Brain Infarction ◽  
Artery Aneurysm ◽  
Posterior Communicating Artery ◽  
Anterior Choroidal Artery ◽  
Mechanical Compression ◽  
Posterior Communicating Artery Aneurysm ◽  
Anterior Choroidal ◽  
Choroidal Artery

A 65-year-old male received coil embolization for a large internal carotid-posterior communicating artery aneurysm. Pre- and postoperative angiography at surgery demonstrated that the ipsilateral anterior choroidal artery branched from the internal carotid artery near the distal side of the aneurysm, and elevated and expanded on the aneurysmal dome, but was clearly visualized. Three days following endovascular treatment, the patient presented hemiparesis on the left side, with brain infarction in the territory of the right anterior choroidal artery despite antithrombotic therapy. The delayed brain infarction was likely caused by a reduction in anterior choroidal artery perfusion caused by mechanical compression following a postoperative increase in internal carotid-posterior communicating artery aneurysmal volume during intra-aneurysmal thrombosis. Transient volume expansion after coil embolization for intracranial aneurysms is rarely reported as a cause of brain infarction. It is important to recognize these arteries as potential postoperative complication risks, and consider the use of open surgery to avoid this risk.

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