scholarly journals Negative Pressure Pulmonary Edema After Oral and Maxillofacial Surgery

2009 ◽  
Vol 56 (2) ◽  
pp. 49-52 ◽  
Author(s):  
Hideki Mamiya ◽  
Tatsuya Ichinohe ◽  
Yuzuru Kaneko

Abstract Negative pressure pulmonary edema (NPPE) following upper airway obstruction (UAO) has been reported in several clinical situations. The main cause of NPPE is reported to be increased negative intrathoracic pressure. We present a case of NPPE that occurred after general anesthesia for plate removal after jaw deformity surgery. After completion of the surgery, administration of inhaled anesthetics was stopped and the patient opened his eyes on verbal command. Immediately after extubation, the patient stopped breathing and became cyanotic. Acute UAO following laryngospasm was suspected. Soon after reintubation, pink, frothy fluid came out of the endotracheal tube, and a tentative diagnosis of NPPE was made. Continuous positive airway pressure was applied. In addition, furosemide and dexamethasone were administered. By the next day, the symptoms had almost disappeared.

2021 ◽  
Vol 24 (5) ◽  
pp. 141-143
Author(s):  
Carmen Di Maria ◽  
Guido Prizio ◽  
Alessandro Triunfo ◽  
Flavio Quarantiello ◽  
Domenico Di Maria

Negative pressure pulmonary edema (NPPE) is a danger-ous and potentially fatal condition with a multifactorial pathogenesis. NPPE is frequently a manifestation of upper airway obstruction, indeed the large negative intrathoracic pressure generated by forced inspiration against an obstructed airway is thought to be the main mechanism involved. The early detection of the signs of this syndrome is vital to the treatment and to patient outcome. The paper presents a case of NPPE due to post-extubation laryngospasm in a 14-year-old patient.


2019 ◽  
Vol 2019 ◽  
pp. 1-4
Author(s):  
Evan Harmon ◽  
Sebastian Estrada ◽  
Ryan J. Koene ◽  
Sula Mazimba ◽  
Younghoon Kwon

Upper airway obstruction is a potentially life-threatening emergency often encountered in the acute care, perioperative, and critical care settings. One important complication of acute obstruction is negative-pressure pulmonary edema (NPPE). We describe two cases of acute upper airway obstruction, both of which resulted in flash pulmonary edema complicated by acute hypoxic respiratory failure. Though NPPE was suspected, these patients were also found to have Takotsubo syndrome (TTS). Neither patient had prior cardiac disease, and both subsequently had a negative ischemic workup. Because TTS is a condition triggered by hyperadrenergic states, the acute airway obstruction alone or in combination with NPPE was the likely explanation for TTS in each case. These cases highlight the importance of also considering cardiogenic causes of pulmonary edema in the setting of upper airway obstruction, which we suspect generates a profound catecholamine surge and places patients at increased risk of TTS development.


Diagnostics ◽  
2020 ◽  
Vol 10 (10) ◽  
pp. 749 ◽  
Author(s):  
Adrien Holzgreve ◽  
Matthias P. Fabritius ◽  
Philippe Conter

Negative pressure pulmonary edema (NPPE) is a rare, potentially life-threatening, and yet diagnostically challenging perioperative complication. Most cases of NPPE occur in the context of anesthetic procedures, mainly caused by upper airway obstruction, and are diagnosed during the recovery period. We present a case of fulminant NPPE in a patient during general anesthesia which illustrates the eye-catching CT findings that can occur in NPPE and eventually support diagnosis. With regard to the current pandemic, we include a discussion of the typical imaging patterns of COVID-19 as a radiological differential diagnosis of NPPE. A 42-year old male patient presented with sudden respiratory insufficiency during arthroscopic knee lavage and subsequently required highly invasive ventilation therapy and catecholamine administration. Postoperative CT imaging of the thorax exhibited extensive, centrally accentuated consolidations with surrounding ground-glass opacity in all lung lobes, suggestive of pulmonary edema. In view of the clinical course and the imaging findings, a negative pressure pulmonary edema (NPPE) was diagnosed.


2021 ◽  
Vol 49 (9) ◽  
pp. 030006052110477
Author(s):  
Qin Li ◽  
Liang Zhou

To date, only one case of pediatric type II negative pressure pulmonary edema (NPPE) caused by removal of an endobronchial foreign body has been documented. We report another case of type II NPPE that developed after extraction of inhaled peanuts. A 21-month-old boy who presented with wheezing and intermittent cough for 1 month after eating peanuts was admitted to our department. A chest computed tomographic scan showed foreign bodies lodged in the right main bronchus. Fiberoptic bronchoscopy was performed, and three pieces of peanuts were removed. Fifteen minutes after this procedure, the child grew restless and started coughing with frothy pink sputum. Tachypnea and rales were observed. A chest radiograph showed patchy opacification in both lungs, especially in the right lower zone, leading to the diagnosis of type II NPPE. Intravenous furosemide and dexamethasone were immediately administered, followed by non-invasive continuous positive airway pressure ventilation. Twelve hours later, the patient recovered uneventfully and was discharged home the following day. In conclusion, pediatric type II NPPE rapidly occurs following the relief of upper airway obstruction. Clinicians need to be aware of the acuteness and manifestations of type II NPPE to make an early diagnosis and initiate prompt treatment.


2021 ◽  
Vol 35 (1) ◽  
pp. 113-116
Author(s):  
İkbal Türker ◽  
Rıza Dinçer Yıldızdaş ◽  
Ozden Ozgur Horoz ◽  
Faruk Ekinci

2020 ◽  
Vol 58 (227) ◽  
Author(s):  
Anisha Budhathoki ◽  
Yawen Wu

Negative pressure pulmonary edema is an uncommon complication of the extubation of theendotracheal tube. An increase in intrathoracic pressure and negative pressure of the lung causedby acute laryngeal spasm results from acute upper respiratory obstruction causing life-threateningpulmonary edema by alveolar-capillary damage is called negative pressure pulmonary edema. Wehere describe 28-years old female case the preoperative diagnosis of pelvic inflammatory diseaseundergoing exploratory laporoscopy caused negative pressure pulmonary edema while extubation.With the immediate treatment, the patient was discharged without any abnormalities.


2019 ◽  
Author(s):  
Jun Xiong ◽  
Yongxing Sun

Abstract Background: The negative pressure pulmonary edema is rare clinical situation which caused mainly by upper airway obstruction. However the present case was without obvious upper airway obstruction, so we suspected there were other culprits combined with the mild upper airway obstruction to produce the negative pressure pulmonary edema. Case presentation: This case was a 5-year-old girl with tumor on saddle area, her hormones level were abnormal preoperatively, such as cortison, adrenocorticotrophic hormone, free T4 and total T4. During the stage of induction, negative pressure pulmonary edema took place due to the mild upper airway obstruction. And the instant chest Computer tomography proved the diagnosis. After intensive care, mostly lung field of the girl recovered to the normal within 48 hours. Conclusion: The patient with abnormal hormone levels is vulnerable to pulmonary edema, although there is no obvious upper airway obstruction. Thus pre-operation hormones supplement is as important as keeping the upper airway unobstructed. Keywords: Negative pressure pulmonary edema, hypocortisolism, hypothyroidea, obstructive sleep apnea.


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