scholarly journals Risk Factors Regarding Portal Vein Thrombosis in Chronic Liver Disease

2020 ◽  
Vol 25 (4) ◽  
pp. 38-41
Author(s):  
Liliana Vecerzan ◽  
Romeo Gabriel Mihăilă
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Hepatic Cirrhosis ◽  
Vascular Diseases ◽  
High Rate ◽  
Imaging Method ◽  
Vein Thrombosis ◽  
Common Causes ◽  
Portosystemic Collateral ◽  
Collateral Vessels

Abstract The portal vein thrombosis (PVT) is one of the most frequent vascular diseases of the liver, with a high rate of morbidity and mortality. The most common causes of the PVT are hepatic cirrhosis, hepatobiliary neoplasms, inflammatory and infectious abdominal diseases, and myeloproliferative syndromes.(1,2) The natural progress of the PVT has as a result portal hypertension which leads to splenomegaly and the formation of portosystemic collateral vessels, as well as gastroesophageal, duodenal and jejunal varices. Ultrasonography, especially Doppler ultrasound, is the most widely used imaging method to asses, supervise and diagnose PVT in patients with hepatopathies. The purpose of acute PVT treatment is to re-permeabilize the obstructed vessels; the endoscopic ligature of the varices in the eventuality of their rupture is safe and extremely efficient in chronic PVT. To conclude, PVT is the most common hepatic vascular disorder, and its prevalence has increased particularly among the patients with chronic hepatopathies.(3)

scholarly journals The ABCD of portal vein thrombosis: a systematic approach

2020 ◽  
Vol 53 (6) ◽  
pp. 424-429
Author(s):  
Alexandre Makoto Minoda ◽  
Raissa Brito Fernandes Cadete ◽  
Sara Reis Teixeira ◽  
Valdair Francisco Muglia ◽  
Jorge Elias Junior ◽  
...  
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Systematic Approach ◽  
Careful Analysis ◽  
Venous System ◽  
Portal Venous System ◽  
Vein Thrombosis ◽  
Case Files ◽  
Collateral Vessels ◽  
And Diffusion

Abstract Portal vein thrombosis refers to complete or partial obstruction of the portal venous system, in the intrahepatic or extrahepatic venous tract or even in the splenic or superior mesenteric veins. This common and potentially fatal condition can develop in various clinical contexts, especially those of liver cirrhosis, hepatocellular carcinoma, and other solid tumors. Certain characteristics, such as the time since the onset of the thrombus (acute or chronic), its biology (hematic or tumoral), the presence of collateral vessels, and the magnetic resonance imaging aspects, are important components of a thorough, careful analysis, as well as informing decisions regarding the appropriate therapeutic strategy. Here, we present a brief review of the anatomy of the portal venous system and a systematic approach to analyzing the condition, using a mnemonic (ABCD, for age, biology, collaterals, and diffusion). We discuss the various imaging methods and illustrate our discussion with images selected from the case files archived at our facility.

scholarly journals Low Molecular Weight Heparin in Portal Vein Thrombosis of Cirrhotic Patients: Only Therapeutic Purposes?

2014 ◽  
Vol 2014 ◽  
pp. 1-6 ◽  
Author(s):  
Raffaele Licinio ◽  
Mariabeatrice Principi ◽  
Giuseppe Losurdo ◽  
Nicola Maurizio Castellaneta ◽  
Enzo Ierardi ◽  
...  
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
High Rate ◽  
Low Molecular Weight ◽  
Vein Thrombosis ◽  
If Current ◽  
General Feeling ◽  
Cirrhotic Patients ◽  
Current Guidelines ◽  
Hepatic Synthesis

Cirrhosis has always been regarded as hemorrhagic coagulopathy caused by the reduction in the hepatic synthesis of procoagulant proteins. However, with the progression of liver disease, the cirrhotic patient undergoes a high rate of thrombotic phenomena in the portal venous system. Although the progression of liver failure produces a reduction in the synthesis of anticoagulant molecules, a test able to detect the patients with hemostatic balance shifting towards hypercoagulability has not yet been elaborated. The need of treatment and/or prophylaxis of cirrhotic patients is demonstrated by the increased mortality, the risk of bleeding from esophageal varices, and the mortality of liver transplantation, when portal vein thrombosis (PVT) occurs even if current guidelines do not give indications about PVT treatment in cirrhosis. In view of the general feeling that the majority of cirrhotic patients at an advanced stage may be in a procoagulant condition (suggested by the sharp increase in the prevalence of PVT), it is presumable that a prophylaxis of this population could be of benefit. The safety and the efficacy of prophylaxis and treatment with enoxaparin in patients with cirrhosis demonstrated by a single paper suggest this option only in controlled trials and, currently, there are no sufficient evidences for a recommendation in the clinical practice.

Chronic Portal Vein Thrombosis: Transcapsular Hepatic Collateral Vessels and Communicating Ectopic Varices

Radiology ◽  
2010 ◽  
Vol 257 (2) ◽  
pp. 568-578 ◽  
Author(s):  
Marcus Seeger ◽  
Rainer Günther ◽  
Holger Hinrichsen ◽  
Marcus Both ◽  
Ulf Helwig ◽  
...  
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Vein Thrombosis ◽  
Ectopic Varices ◽  
Collateral Vessels

scholarly journals Portal vein thrombosis due to Candida albicans associated with hepatic cirrhosis.

1993 ◽  
Vol 69 (809) ◽  
pp. 234-236
Author(s):  
G. Torres ◽  
L. A. Gil Grande ◽  
B. Boixeda ◽  
C. Martin-de-Argila ◽  
R. Barcena ◽  
...  
Keyword(s):  
Candida Albicans ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Hepatic Cirrhosis ◽  
Vein Thrombosis

Portal vein thrombosis in Fontan-associated liver disease

2020 ◽  
Vol 30 (6) ◽  
pp. 883-885
Author(s):  
Renate Kaulitz ◽  
Ludger Sieverding ◽  
Michael Hofbeck
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Collateral Vessel ◽  
Portal Vein Branch ◽  
Vein Thrombosis ◽  
Portosystemic Collateral ◽  
Branch Development ◽  
The Right

AbstractA 25-year-old patient with signs of cirrhosis on ultrasound and CT presented with portal vein thrombosis on routine follow-up examinations; retrograde hepatic wedge angiography demonstrated only the right-sided portal vein branch. Development of a portosystemic collateral vessel to the left-sided renal vein prevented signs of hypersplenism. This unique complication of portal vein thrombosis should be considered during long-term surveillance.

CLINICAL CASE OF PORTAL VEIN THROMBOSIS IN PATIENT WITH HEPATIC CIRRHOSIS

Pharmateca ◽  
2018 ◽  
Vol 2_2018 ◽  
pp. 83-87
Author(s):  
T.V. Ermolova () Ermolova ◽  
E.G. Bykova () Bykova ◽  
B.N. Kotiv () Kotiv ◽  
A.L. Petrov () Petrov ◽  
A.O. Nesterko () Nesterko ◽  
...  
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Clinical Case ◽  
Hepatic Cirrhosis ◽  
Vein Thrombosis

scholarly journals Duodeno-Jejunal Varicosities Following Extrahepatic Portal Vein Thrombosis

HPB Surgery ◽  
1992 ◽  
Vol 5 (2) ◽  
pp. 147-153 ◽  
Author(s):  
Nick Varsamidakis ◽  
Brian R. Davidson ◽  
Kenneth Hobbs
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Portal Pressure ◽  
Duodenal Atresia ◽  
Left Lobe ◽  
Jejunal Loop ◽  
Anastomotic Site ◽  
Vein Thrombosis ◽  
Pass Through ◽  
Collateral Vessels

A 31 year old man, under investigation for melena, was found at endoscopy to have varicosities at the site of a duodeno-jejunostomy which had been performed for duodenal atresia when he was three days old. Angiography revealed an occluded portal vein with an extensive collateral circulation. At laparotomy some of the collateral vessels were found to pass through the anastomotic site and directly into the left lobe of the liver. The portal pressure was found to be minimally elevated. Resection of the anastomotic segment was performed with reconstruction using a Roux en Y jejunal loop. Bleeding from collateral vessels passing through an anastomosis site in a patient with extrahepatic portal vein thrombosis has not previously been reported.

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