Case File: Cardiac amyloidosis using on routine hand-held ultrasound

A 64 year-old man presented to the Kingston General Hospital with cardiac arrest. At the time of EMS arrival, the ECG showed ventricular tachycardia. The patient was intubated and ventilated. Multiple defibrillations were required to convert the patient back to normal sinus rhythm.

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Ventricular Tachycardia and Ventricular Fibrillation Analysis Based on Jensen-Shannon Divergence Analysis

Applied Mechanics and Materials ◽

10.4028/www.scientific.net/amm.321-324.712 ◽

2013 ◽

Vol 321-324 ◽

pp. 712-715

Author(s):

Zheng Zhong Zheng ◽

Jun Chang Zhao ◽

Jun Wang

Keyword(s):

Ventricular Tachycardia ◽

Ventricular Fibrillation ◽

Sinus Rhythm ◽

Complexity Analysis ◽

Heart Function ◽

Normal Sinus Rhythm ◽

Normal Sinus ◽

Practice Of Medicine ◽

Cardiac Sudden Death ◽

Jensen Shannon Divergence

t is an important method for using electrocardiogram (ECG) to detect and diagnose heart function in clinical practice of medicine. Ventricular tachycardia (VT) and ventricular fibrillation (VF) are serious threats for peoples lives, they often bring about cardiac sudden death. In this paper, the complexity analysis method based on Jensen-Shannon Divergence was used to calculate the complexity of the normal sinus rhythm signals, VT, VF. The study found that the VF was highest, followed by that of VT, and that of normal sinus rhythm signals was minimum. The result can be used to assisted clinical diagnosis.

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Pulseless Ventricular Tachycardia During Office-Based Anesthetic in a Four-Year-Old Child

Anesthesia Progress ◽

10.2344/13-00032.1 ◽

2015 ◽

Vol 62 (4) ◽

pp. 162-165 ◽

Cited By ~ 3

Author(s):

Timothy M. Orr ◽

Daniel L. Orr

Keyword(s):

Ventricular Tachycardia ◽

Sinus Rhythm ◽

Life Support ◽

Normal Sinus Rhythm ◽

Advanced Cardiac Life Support ◽

Epinephrine Administration ◽

Normal Sinus ◽

Dental Office ◽

Hospital Emergency Room ◽

Dental Rehabilitation

A 4-year-old 16-kg boy presented for full mouth dental rehabilitation in a private pediatric dental office. The patient had no significant previous medical history. Upon sevoflurane induction by a dentist anesthesiologist, the patient converted from normal sinus rhythm to pulseless ventricular tachycardia. Advanced cardiac life support protocol was initiated. After 2 automatic external defibrillator shocks were delivered in conjunction with epinephrine administration, the patient returned to normal sinus rhythm. The patient was transported via emergency medical service paramedics to a local children's hospital emergency room where he was observed uneventfully for 24 hours prior to discharge.

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Adenosine deaminase and adenosine attenuate ventricular arrhythmias caused by norepinephrine

AJP Heart and Circulatory Physiology ◽

10.1152/ajpheart.1991.260.3.h979 ◽

1991 ◽

Vol 260 (3) ◽

pp. H979-H984 ◽

Cited By ~ 1

Author(s):

G. S. Friedrichs ◽

G. F. Merrill

Keyword(s):

Ventricular Tachycardia ◽

Sinus Rhythm ◽

Adenosine Deaminase ◽

Ventricular Arrhythmias ◽

Normal Sinus Rhythm ◽

Common Element ◽

Adenosine Infusion ◽

Normal Sinus ◽

Arrhythmogenic Effects

Twenty-five beagles weighing 9.1 +/- 0.4 kg were used to investigate the arrhythmogenic effects of divided doses of intracoronary norepinephrine (50-200 ng.kg-1.min-1) in the absence and the presence of adenosine deaminase (5 U.kg-1.min-1). A dose of norepinephrine (100 ng.kg-1.min-1) that caused 66 +/- 17% ectopy in the absence of adenosine deaminase caused only 16 +/- 14% ectopy (P less than 0.05) in the presence of the enzyme. Ventricular tachycardia caused by 200 ng.kg-1.min-1 norepinephrine was reduced from 1.2 +/- 0.3 to 0.1 +/- 0.1 bouts/10 cardiac cycles (P less than 0.05) by adenosine deaminase. In five additional dogs, intracoronary adenosine (0.11 mumol/min) terminated sustained norepinephrine-induced (200 ng.kg-1.min-1) ventricular tachycardia within 23 +/- 9 s (P less than 0.05). As long as the adenosine infusion was maintained, a normal sinus rhythm was observed. We conclude that both adenosine and adenosine deaminase significantly attenuate norepinephrine-induced ventricular arrhythmias. A common element beyond the deamination of adenosine, quite possibly ammonia, appears to account for these results.

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RECURRENT VENTRICULAR TACHYCARDIA IN AN INFANT—USE OF HIGH VOLTAGE DC SHOCK THERAPY IN MANAGEMENT

PEDIATRICS ◽

10.1542/peds.33.6.926 ◽

1964 ◽

Vol 33 (6) ◽

pp. 926-930

Author(s):

Ramon V. Canent ◽

Madison S. Spach ◽

James J. Morris ◽

Will L. London

Keyword(s):

Ventricular Tachycardia ◽

Drug Therapy ◽

Sinus Rhythm ◽

Cardiac Cycle ◽

Normal Sinus Rhythm ◽

Shock Therapy ◽

Right Atrial ◽

Qrs Complex ◽

T Wave ◽

Normal Sinus

The case of a 17-month-old infant with ventricular tachycardia has been presented. The diagnosis was confirmed by esophageal and right atrial electrocardiography. No cardiac abnormalities were demonstrated by bilane angiocardiography. Quinidine and steroid therapy were effective temporarily for conversion to normal sinus rhythm. Gradually, conversion became more difficult and sustained ventricular tachycardia became refractory to drug therapy. A Lown Cardioverter was employed with a single countershock discharge of 8 to 15 watt seconds (1,000 to 1,370 volts). The shock was electronically timed to occur at the peak of the QRS complex to avoid the vulnerable period of the cardiac cycle (T-wave), wherein the shock may initiate ventricular fibrillation. Normal sinus rhythm was established on five occasions with the administration of a single shock. Following this therapy, the patient remained well and maintained a normal sinus rhythm for ten months while on Quinaglute duratabs® and reserpine.

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