EDUKASI GIZI PADA IBU HAMIL DI KELURAHAN PISANG  KECAMATAN PAUH KOTA PADANG

Desmawati Desmawati; Abdiana Abdiana; Edison Edison; Wahyu Kodri A; Rian Rizki Ananda; Pandu Prakoso Moendanoe; Zikra Fadhilah; Karolina Karolina; Doa Vami; Rahmeidia Audya Yusmi; Qorirah Summayah Indrapati; Nurul Fadhilah; Mona Indah Putriani

doi:10.25077/bina.v4i2.290

EDUKASI GIZI PADA IBU HAMIL DI KELURAHAN PISANG KECAMATAN PAUH KOTA PADANG

BULETIN ILMIAH NAGARI MEMBANGUN ◽

10.25077/bina.v4i2.290 ◽

2021 ◽

Vol 4 (2) ◽

pp. 106-113

Author(s):

Desmawati Desmawati ◽

Abdiana Abdiana ◽

Edison Edison ◽

Wahyu Kodri A ◽

Rian Rizki Ananda ◽

...

Keyword(s):

Growth Retardation ◽

Uterine Growth ◽

Post Test ◽

Intra Uterine Growth Retardation

Kekurangan Energi Kronis merupakan masalah pada ibu hamil yang prevalensinya fluktuasi dan dapat menimbulkan dampak negatif pada bayi yang akan dilahirkannya. Kegiatan ini bertujuan untuk memberikan penyuluhan dan edukasi kepada ibu hamil dengan Kurang Energi Kronis yang berada di wilayah kerja Puskesmas Pauh tepatnya Kelurahan Pisang untuk mengetahui, memahami dan mengaplikasikan pengetahuan tentang asupan gizi yang baik untuk ibu hamil sehingga dapat menurunkan insidensi intra-uterine growth retardation yang mampu berujung pada status gizi anak yang buruk. Kegiatan ini dilakukan di Kelurahan Pisang Kecamatan Pauh Kota Padang dengan melibatkan seluruh sektor yang terkait. Kegiatan ini berupa serangkaian acara berupa pelatihan kader dan penyuluhan pada ibu hamil dengan kekurangan energi kronis yang didahului dengan pretest, launching kelas ibu hamil, demonstrasi memasak dan konsultasi kehamilan. Hasil kegiatan setelah acara dilakukan post-test untuk menilai peningkatan pengetahuan ibu hamil dan kader. Peningkatan tingkat pengetahuan pada ibu hamil setelah dilakukan penyuluhan gizi yang baik pada kehamilan dari 54,5 % menjadi 63,6 % tergolong baik, 36,3 % ke 27,2 % tergolong cukup dan yang memiliki pengetahuan kurang sebesar 9 %. Program bantu siaga asupan gizi mama kekurangan energi kronik terbukti dapat meningkatkan pengetahuan ibu hamil dan kader tentang pentingnya asupan zat gizi dalam mencegah terjadinya kekurangan energi kronis pada ibu hamil, serta pentingnya nutrisi pada ibu hamil dengan kekurangan energi kronis agar tidak berdampak pada bayi yang dikandungnya.

Download Full-text

Intra-Uterine Growth Retardation in the Pig. I. Organ Size and Cellular Development at Birth and after Growth to Maturity

Neonatology ◽

10.1159/000240427 ◽

1971 ◽

Vol 19 (4-6) ◽

pp. 329-340 ◽

Cited By ~ 77

Author(s):

E.M. Widdowson

Keyword(s):

Growth Retardation ◽

Organ Size ◽

Uterine Growth ◽

Cellular Development ◽

Intra Uterine Growth Retardation

Download Full-text

Effects of prenatal glucocorticoid exposure on cardiac calreticulin and calsequestrin protein expression during early development and in adulthood

Biochemical Journal ◽

10.1042/bj20021771 ◽

2003 ◽

Vol 371 (1) ◽

pp. 61-69 ◽

Cited By ~ 21

Author(s):

Maria L. LANGDOWN ◽

Mark J. HOLNESS ◽

Mary C. SUGDEN

Keyword(s):

Cardiac Function ◽

Protein Expression ◽

Growth Retardation ◽

Premature Death ◽

Fetal Life ◽

Dexamethasone Treatment ◽

Increased Risk ◽

Uterine Growth ◽

Intra Uterine Growth Retardation ◽

Glucocorticoid Action

Overexpression of the conserved Ca2+-binding proteins calreticulin and calsequestrin impairs cardiac function, leading to premature death. Calreticulin is vital for embryonic development, but also impairs glucocorticoid action. Glucocorticoid overexposure during late fetal life causes intra-uterine growth retardation and programmed hypertension in adulthood. To determine whether intra-uterine growth retardation or programmed hypertension was associated with altered calreticulin or calsequestrin expression, effects of prenatal glucocorticoid overexposure (maternal dexamethasone treatment on days 15—21 of pregnancy) were examined during fetal life and postnatal development until adulthood (24 weeks). Dexamethasone (100 or 200μg/kg of maternal body weight) was administered via osmotic pump. Calreticulin was detected as a 55kDa band and calsequestrin as 55 and 63kDa bands in 21 day fetal hearts. Only the 55kDa calsequestrin band was detected postnatally. Prenatal glucocorticoid overexposure at the higher dose decreased calreticulin protein expression (26%; P<0.05) but increased calsequestrin protein expression, both 55 and 63kDa bands, by 87% (P<0.01) and 78% (P<0.01); only the 55kDa calsequestrin band was increased at the lower dose (66%; P<0.05). Offspring of dams treated at the lower dexamethasone dose were studied further. In control offspring, cardiac calreticulin protein expression declined between 2 and 3 weeks of age, and remained suppressed until adulthood. Cardiac calsequestrin protein expression increased 2-fold between fetal day 21 and postnatal day 1 and continued to increase until adulthood, at which time it was 3.4-fold higher (P<0.001). Prenatal dexamethasone exposure minimally affected postnatal calsequestrin protein expression, but the postnatal decline in calreticulin protein expression was abrogated and calreticulin protein expression in adulthood was 2.2-fold increased (P<0.001) compared with adult controls. In view of the known associations between cardiac calreticulin overexpression and impaired cardiac function, targeted up-regulation of calreticulin may contribute to the increased risk of adult heart disease introduced as a result of prenatal overexposure to glucocorticoids.

Download Full-text