scholarly journals A case of acute myocardial infarction in a patient with congenital atresia of the left coronary artery

2020 ◽  
Vol 11 (1) ◽  
pp. 46-49
Author(s):  
Anna G. Evdokimova ◽  
Vladimir V. Popov ◽  
Olga A. Shuiskaya ◽  
Irina S. Mikhailova
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Coronary Artery ◽  
Cytomegalovirus Infection ◽  
Left Coronary Artery ◽  
Clinical Laboratory ◽  
Congenital Atresia ◽  
Coronary Syndrome ◽  
Single Right Coronary Artery ◽  
Modern Technologies

A clinical example of a rare pathology is presented a congenital anomaly of the coronary arteries, namely, atresia of the trunk of the left coronary artery in a young patient of 40 years old. This pathology was asymptomatic and was suddenly manifested by the development of acute myocardial infarction in a patient with a single right coronary artery. Clinical, laboratory and instrumental research methods using modern technologies are described in detail. In the article, the authors discuss the genesis of the development of acute coronary syndrome, due to a possible connection with the activation of cytomegalovirus infection.

scholarly journals Acute myocardial infarction and syncope in an 18-year-old athlete with an abnormal origin of the left coronary artery: a case report

Cases Journal ◽  
2009 ◽  
Vol 2 (1) ◽  
pp. 8142 ◽  
Author(s):  
Ronan MG Berg ◽  
Bente Brendorp ◽  
Thomas Kristensen ◽  
Morten Helvind ◽  
Steffen Helqvist
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Case Report ◽  
Coronary Artery ◽  
Left Coronary Artery

scholarly journals Adverse Cardiac Remodelling after Acute Myocardial Infarction: Old and New Biomarkers

2020 ◽  
Vol 2020 ◽  
pp. 1-21 ◽  
Author(s):  
Alexander E. Berezin ◽  
Alexander A. Berezin
Keyword(s):  
Myocardial Infarction ◽  
Heart Failure ◽  
Acute Myocardial Infarction ◽  
Acute Coronary Syndrome ◽  
Coronary Artery ◽  
New Technologies ◽  
Early Period ◽  
Cardiac Remodelling ◽  
Complete Revascularization ◽  
Coronary Syndrome

The prevalence of heart failure (HF) due to cardiac remodelling after acute myocardial infarction (AMI) does not decrease regardless of implementation of new technologies supporting opening culprit coronary artery and solving of ischemia-relating stenosis with primary percutaneous coronary intervention (PCI). Numerous studies have examined the diagnostic and prognostic potencies of circulating cardiac biomarkers in acute coronary syndrome/AMI and heart failure after AMI, and even fewer have depicted the utility of biomarkers in AMI patients undergoing primary PCI. Although complete revascularization at early period of acute coronary syndrome/AMI is an established factor for improved short-term and long-term prognosis and lowered risk of cardiovascular (CV) complications, late adverse cardiac remodelling may be a major risk factor for one-year mortality and postponded heart failure manifestation after PCI with subsequent blood flow resolving in culprit coronary artery. The aim of the review was to focus an attention on circulating biomarker as a promising tool to stratify AMI patients at high risk of poor cardiac recovery and developing HF after successful PCI. The main consideration affects biomarkers of inflammation, biomechanical myocardial stress, cardiac injury and necrosis, fibrosis, endothelial dysfunction, and vascular reparation. Clinical utilities and predictive modalities of natriuretic peptides, cardiac troponins, galectin 3, soluble suppressor tumorogenicity-2, high-sensitive C-reactive protein, growth differential factor-15, midregional proadrenomedullin, noncoding RNAs, and other biomarkers for adverse cardiac remodelling are discussed in the review.

scholarly journals Case report of a 66-year-old woman with atypical takotsubo syndrome and concomitant coronary artery disease

2020 ◽  
Vol 13 (2) ◽  
pp. e230164 ◽  
Author(s):  
David Niederseer ◽  
Jelena Rima Ghadri ◽  
Robert Manka ◽  
Christian Templin
Keyword(s):  
Myocardial Infarction ◽  
Coronary Artery Disease ◽  
Acute Myocardial Infarction ◽  
Coronary Artery ◽  
Coronary Artery Stenosis ◽  
Takotsubo Syndrome ◽  
Elevated Troponin ◽  
Coronary Syndrome ◽  
Artery Disease ◽  
Concomitant Coronary Artery Disease

Takotsubo syndrome is increasingly recognised worldwide. As both, takotsubo syndrome and acute myocardial infarction can present with similar findings, including chest pain, elevated troponin and creatine kinase, it is often difficult to differentiate these conditions. Here, we present a challenging case that illustrates (1) difficulties to diagnose takotsubo syndrome in the presence of a significant coronary artery stenosis; (2) how takotsubo syndrome could be misdiagnosed as acute coronary syndrome if diagnostic workup does not include echocardiography or left ventriculography; (3) the importance of cardiac MRI which can contribute to the diagnosis of takotsubo syndrome.

scholarly journals Case Report of an Acute Myocardial Infarction as a Result of Spontaneous Coronary Artery Dissection in a Patient with Fibromuscular Dysplasia

2019 ◽  
Vol 2019 ◽  
pp. 1-4
Author(s):  
A. Kalinskaya ◽  
D. Skrypnik ◽  
A. Kostin ◽  
E. Vasilieva ◽  
A. Shpektor
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Coronary Artery ◽  
Fibromuscular Dysplasia ◽  
Artery Dissection ◽  
Spontaneous Coronary Artery Dissection ◽  
Coronary Artery Dissection ◽  
Coronary Syndrome ◽  
Case Summary ◽  
Acute Mi

Background. Spontaneous coronary artery dissection (SCAD) is an underdiagnosed and rare cause of myocardial infarction (MI). SCAD is defined as the separation of the coronary artery wall by hemorrhage with or without intimal tear. It causes acute coronary syndrome in 1.7% to 4% of cases. Case Summary. We report a case of a patient with acute MI caused by SCAD with marked progression of dissection within 4 days and spontaneous healing in 2 months. Fibromuscular dysplasia (FMD) of the arteries is an associated condition of SCAD that was found in our patient. Conclusion. In young women admitted to the clinic with signs of acute myocardial infarction, SCAD should be suspected. FMD as an associated condition that should be ruled out in every SCAD patient. Conservative treatment of SCAD is the most preferable strategy.

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