Focal necrosis of the caecum

2018 ◽  
Vol 0 (1) ◽  
pp. 95-98
Author(s):  
K. O. Vander ◽  
V. I. Lupaltsov
Keyword(s):  
1944 ◽  
Vol 100 (5) ◽  
pp. 519
Author(s):  
N. Mitchell ◽  
A. Angrist

2015 ◽  
Vol 23 (3) ◽  
pp. 141-148 ◽  
Author(s):  
Thangapalam Jawahar Abraham ◽  
Prakash Kumar Mallick ◽  
Harresh Adikesavalu ◽  
Sayani Banerjee

AbstractEdwardsiella tarda is one of the serious fish pathogens infecting both cultured and wild fish species. This study aimed to assess the phenotypic characterization and pathogenicity of E. tarda isolated from Clarias gariepinus (Burchell) with dropsy and histopathological alterations. The causative agent was identified with Vitek 2, and its pathogenicity was determined by intramuscular injection. The challenged catfish exhibited vertical hanging, frothing, excess mucus production, listing, swollen abdomen, anorexia, fin and tail rot, and reddish operculum. The LD50of E. tarda PBB and PBP strains was found to be 8.52 × 106and 1.68 × 107cells fish-1, respectively. Histopathological observations on catfish infected naturally revealed lymphocyte infiltration in muscle and focal necrosis, hyperplasia, edema, and swelling of the gill lamellar epithelium. The kidney of diseased fish exhibited ischemic type tubulopathy, necrosis of nephritic tubules, hyperplastic hematopoietic tissue, rupture of the tubular basement membrane, hydropic dystrophy of nephritic cells, neutrophil infiltration, fibrinoid necrosis of nephretic tubules, hemosiderin deposition, and edema. The liver sections revealed lymphocyte infiltration, dilation of hepatic sinusoids, expansion of space between hepatic sinusoids, and focal necrosis. The inflammatory responses observed in kidney and liver in the present study were presumably suppuration and were attributed to the potential virulence factors of E. tarda.


1999 ◽  
Vol 123 (6) ◽  
pp. 536-538
Author(s):  
Augusto F. Mesia ◽  
Peter Mo ◽  
Lourdes R. Ylagan

Abstract We describe an unusual, large atypical cellular chorangioma with abundant mitoses and focal necrosis. Other than premature birth, the prenatal and postpartum clinical course was unremarkable for both the mother and baby. Our case and a few similar cases reported in literature suggest that atypical cellular chorangioma is a benign tumor, despite its worrisome histopathologic features.


2020 ◽  
Vol 23 (1) ◽  
pp. 25-35
Author(s):  
MM Rahman ◽  
J Alam ◽  
MM Rahman ◽  
MAHNA Khan ◽  
MG Haider

This experiment was conducted to determine the pathology of pullorum disease of chickens and molecular identification of its pathogen. A total of 108 samples, including swabs from different organs were collected from different commercial layer farms of Gazipur district. The histopathological samples were collected in 10% formalin and the swabs were collected in tetrathionate broth. Samples were subjected to isolation and identification of the causal agent followed by gross and histopathological study of the affected visceral organs. Fifty eight out of 108 cloacal swabs (53.7%) were Salmonella positive. The percentage of Salmonella positive in liver swabs, spleen swabs, lung swabs and intestinal swabs from dead birds were 55.88%, 32.35%, 35.29% and 47.05%, respectively. On average, 52.94% livers of Salmonella affected birds were enlarged, congested and hemorrhagic and necrotic foci was present in 32.35% liver. Unabsorbed and coagulated yolk was found in 70.58% cases. From these 38.24% spleens were swollen and congested and 44.12 % kidneys were enlarged. At histopathology, 52.94% livers showed congestion, focal necrosis with multifocal infiltration of histiocytes in liver parenchyma. Focal necrosis and inflammatory cells were found in 70.58% spleen. Infiltration of heterophils in intestinal mucosa was found in 47.05% cases. 20.58% (7 out of 34) samples were PCR positive for Salmonella Pullorum organism. Ann. Bangladesh Agric. (2019) 23(1) : 25-35


The Lancet ◽  
1928 ◽  
Vol 211 (5464) ◽  
pp. 1012-1013
Author(s):  
C.E. Newman
Keyword(s):  

1982 ◽  
Vol 243 (2) ◽  
pp. F113-F120
Author(s):  
R. E. Cronin ◽  
E. R. Ferguson ◽  
W. A. Shannon ◽  
J. P. Knochel

Magnesium deficiency was induced in a setting of an otherwise adequate diet in adult beagle dogs. Despite the development of severe hypomagnesemia (from 1.5 +/- 0.2 to 0.5 +/- 0.2 meq/liter) during the 10-wk study, Mg content of skeletal muscle fell only modestly (from 3.8 +/- 0.2 to 3.1 +/- 0.4, P less than 0.005, at 7 wk and 3.5 +/- 0.4 mM/100 g FFDS, NS, at 10 wk). The most pronounced muscle compositional changes were a loss of phosphorus (from 29.5 +/- 1.8 to 22.0 +/- 1.6, P less than 0.001, at 7 wk and 24.8 +/- 2.8 mM/100 G FFDS, P less than 0.001, at 10 wk) and gains of calcium (from 0.64 +/- 0.11 to 0.93 +/- 0.17, P less than 0.05, at 7 wk, and 0.85 +/- 0.26 mM/100 g FFDS, P less than 0.05, at 10 wk), sodium (from 13.2 +/- 2.6 to 22.9 +/- 4.7, P less than 0.001 at 7 wk and 17.8 +/- 2.0 meq/100 g FFDS, P less than 0.005, at 10 wk), and chloride (from 5.8 +/- 0.8 to 8.2 +/- 1.6, P less than 0.001, at 7 wk and 6.8 +/- 0.6 meq/100 g FFDS, P less than 0.05, at 10 wk). Cellular potassium content did not change (from 35.9 +/- 1.9 to 33.0 +/- 4.1, NS, at 7 wk and 36.3 +/- 2.0 meq/100 g FFDS, NS, at 10 wk). Muscle cell electrical hyperpolarization developed after 10 wk of Mg depletion. Convulsive seizures developed in three animals. Frank rhabdomyolysis in three animals and focal necrosis in four animals were present on terminal biopsy, with only four animals having completely normal histology.


1970 ◽  
Vol 4 (2) ◽  
pp. 79-85 ◽  
Author(s):  
MM Islam ◽  
MG Haider ◽  
EH Chowdhury ◽  
M Kamruzzaman ◽  
MM Hossain

The present research work was conducted to study about the seroprevalence and pathology of Salmonella infections in layer chickens of Dhaka and Gazipur regions of Bangladesh and to isolate and characterize Salmonellae from layer chickens during the period from January to May 2006. The used materials were blood sample, cloacal and liver swabs from live and dead birds respectively, and visceral organs (liver, lungs, spleen and intestine). The used methods were serum plate agglutination (SPA) test; necropsy and histopathology; cultural, morphological and biochemical test. The overall seroprevalence was 43.4%. During necropsy, congested and enlarged liver with focal necrosis, haemorrhagic and discoloured ovary with stalk formation and mild haemorrhagic to catarrhal enteritis in intestine were detected. Microscopically, the liver showed congestion, focal necrosis with multifocal infiltration of histiocytes in liver parenchyma. In ovum, infiltration of leukocytes, bacterial colony, RE cell proliferation and fibrinoid necrosis were recorded. The intestinal mucosa exhibited infiltration of mononuclear cells in mucosa, submucosa as well as muscularis mucosa. A total of 33 (21.02%) Salmonellae from live and dead birds were isolated. The isolation rate of Salmonellae was higher in seronegative (31.6%) group than seropositive (3.2%) group. Out of 33 Salmonella isolates, 25 were S. pullorum, 3 were S. gallinarum and the rest 5 were motile Salmonellae. The isolates obtained in the study may be investigated for serotyping, pathogenesis study, antibiogram and vaccine production in future. Key words: Seroprevalence, histopathology, isolation, identification, Salmonella, chickensDOI = 10.3329/bjvm.v4i2.1288Bangl. J. Vet. Med. (2006). 4 (2): 79–85


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