Upregulated Local Cytokine Production in Recurrent Tonsillitis Compared with Tonsillar Hypertrophy

1995 ◽  
Vol 115 (5) ◽  
pp. 689-696 ◽  
Author(s):  
Karin Ågren ◽  
Ulf Andersson ◽  
Britt Nordlander ◽  
Carl-Erik Nord ◽  
Annika Linde ◽  
...  
Keyword(s):  
Cytokine Production ◽  
Tonsillar Hypertrophy ◽  
Recurrent Tonsillitis ◽  
Local Cytokine

Comparison of histology between recurrent tonsillitis and tonsillar hypertrophy

2003 ◽  
Vol 28 (3) ◽  
pp. 235-239 ◽  
Author(s):  
P.C. Zhang ◽  
Y.T. Pang ◽  
K.S. Loh ◽  
D.Y. Wang
Keyword(s):  
Tonsillar Hypertrophy ◽  
Recurrent Tonsillitis

scholarly journals Glutathione S-transferase enzyme activity and protein expression in patients with recurrent tonsillitis and idiopathic tonsillar hypertrophy

2019 ◽  
Vol 163 (4) ◽  
pp. 349-354
Author(s):  
Sedat Aydin ◽  
Mehmet Gokhan Demir ◽  
Serpil Oguztuzun ◽  
Murat Kilic ◽  
Can Yilmaz ◽  
...  
Keyword(s):  
Enzyme Activity ◽  
Protein Expression ◽  
Tonsillar Hypertrophy ◽  
Glutathione S Transferase ◽  
Recurrent Tonsillitis

Microbiology of Obstructive Tonsillar Hypertrophy and Recurrent Tonsillitis

1989 ◽  
Vol 115 (6) ◽  
pp. 721-724 ◽  
Author(s):  
I. H. Kielmovitch ◽  
G. Keleti ◽  
C. D. Bluestone ◽  
E. R. Wald ◽  
C. Gonzalez
Keyword(s):  
Tonsillar Hypertrophy ◽  
Recurrent Tonsillitis

scholarly journals TCT-157 Increased Local Cytokine Production at Culprit Superficial Femoral Artery Plaques

2012 ◽  
Vol 60 (17) ◽  
pp. B45-B46
Author(s):  
Cameron Donaldson ◽  
David Schneider ◽  
Daniel Bertges ◽  
Julie Adams ◽  
Nader Elgharib ◽  
...  
Keyword(s):  
Femoral Artery ◽  
Superficial Femoral Artery ◽  
Cytokine Production ◽  
Local Cytokine

Actinomycosis in Obstructive Tonsillar Hypertrophy and Recurrent Tonsillitis

1991 ◽  
Vol 117 (8) ◽  
pp. 883-885 ◽  
Author(s):  
S. M. Pransky ◽  
J. I. Feldman ◽  
D. B. Kearns ◽  
A. B. Seid ◽  
G. F. Billman
Keyword(s):  
Tonsillar Hypertrophy ◽  
Recurrent Tonsillitis

Evaluation of iron and zinc levels in recurrent tonsillitis and tonsillar hypertrophy

2016 ◽  
Vol 37 (2) ◽  
pp. 116-119 ◽  
Author(s):  
Battal Tahsin Somuk ◽  
Emrah Sapmaz ◽  
Harun Soyalıç ◽  
Murat Yamanoğlu ◽  
Durali Mendil ◽  
...  
Keyword(s):  
Tonsillar Hypertrophy ◽  
Recurrent Tonsillitis ◽  
Iron And Zinc ◽  
Zinc Levels

Exacerbates kidney injury in mice subjected to sepsis: critical impact of used heparin

2018 ◽  
Vol 6 (4) ◽  
pp. 323-335
Author(s):  
Peter Weighardt ◽  
Niels Hayashi
Keyword(s):  
Oxidative Stress ◽  
Acute Kidney Injury ◽  
Cytokine Production ◽  
Multiorgan Failure ◽  
Kidney Injury ◽  
Tubular Dysfunction ◽  
Cytokine Storm ◽  
Peritubular Capillary ◽  
Inflammatory Reactions ◽  
Local Cytokine

Sepsis is the leading cause of death in critically ill patients, and the incidence of sepsis is increasing causes multiorgan failure, including acute kidney injury (AKI) and patients with both sepsis and AKI have an especially high mortality rate. Several different pathophysiological mechanisms have been proposed for sepsis-induced AKI: vasodilation-induced glomerular hypoperfusion, dysregulated circulation within the peritubular capillary network, inflammatory reactions by systemic cytokine storm or local cytokine production, and tubular dysfunction induced by oxidative stress animal sepsis models have been developed using LPS infusion. Renal dysfunction evaluated by serum creatinine and BUN was found in acute non-survivors (<24 hours) and decreased urine output in subacute non-survivors (24–96 hours). The study show that increased AKI in animal with blood collected heparin (P=0.002) compared to citrate, furthermore; sham mice that received heparin did not develop AKI.

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