Auriculotemporal syndrome (Frey's syndrome) presenting as tic douloureux

1990 ◽  
Vol 72 (6) ◽  
pp. 955-958 ◽  
Author(s):  
Giuseppe De Benedittis
Keyword(s):  
Treatment Modalities ◽  
Frey’S Syndrome ◽  
Content Type ◽  
Gustatory Sweating ◽  
Tic Douloureux ◽  
Auriculotemporal Syndrome ◽  
Frey's Syndrome ◽  
Fine Print ◽  
Auriculotemporal Nerve

✓ Two cases of auriculotemporal nerve syndrome (Frey's syndrome) presenting as trigeminal tic douloureux are reported. This condition, characterized by gustatory sweating and facial hyperemia, is occasionally associated with pain, which is usually described as aching or burning, and long-lasting. In these two cases, however, a tantalizing gustatory pain occurred in excruciating brief paroxysms. The pathophysiology of the syndrome, with particular reference to pain, and possible treatment modalities are discussed.

scholarly journals Frey's syndrome after condylar fracture: case report

2004 ◽  
Vol 15 (2) ◽  
pp. 159-162 ◽  
Author(s):  
Cassio Edvard Sverzut ◽  
Alexandre Elias Trivellato ◽  
Elis Cristina Souza Serra ◽  
Emanuela Prado Ferraz ◽  
Alexander Tadeu Sverzut
Keyword(s):  
Case Report ◽  
Rigid Fixation ◽  
Simple Test ◽  
Review Of The Literature ◽  
Frey’S Syndrome ◽  
Condylar Fracture ◽  
Gustatory Sweating ◽  
Auriculotemporal Syndrome ◽  
Frey's Syndrome ◽  
Auriculotemporal Nerve

Frey's syndrome is the occurrence of hyperesthesia, flushing and warmth or sweating over the distribution of the auriculotemporal nerve and/or greater auricular nerve while eating foods that produce a strong salivary stimulus. Frey's syndrome is also known as auriculotemporal syndrome and gustatory sweating. We present a case of Frey's syndrome after a condylar fracture and its treatment by internal rigid fixation. A review of the literature is provided along with mention of a simple test (Minor's test) that can help in the diagnosis of this syndrome.

Risk of late stroke and survival following carotid endarterectomy procedures for symptomatic patients

1990 ◽  
Vol 73 (2) ◽  
pp. 193-200 ◽  
Author(s):  
Dennis A. Turner ◽  
Jay Tracy ◽  
Stephen J. Haines
Keyword(s):  
Carotid Endarterectomy ◽  
Life Table ◽  
Stroke Risk ◽  
Neurological Symptoms ◽  
Treatment Modalities ◽  
Control Group ◽  
Content Type ◽  
Postoperative Stroke ◽  
Fine Print

✓ The long-term outcome following carotid endarterectomy for neurological symptoms was analyzed using a retrospective life-table approach in 212 patients who had undergone 243 endarterectomy procedures. The postoperative follow-up period averaged 38.9 ± 2.1 months (mean ± standard error of the mean). The endpoints of stroke and death were evaluated in these patients. Patient groups with the preoperative symptoms of amaurosis fugax, transient ischemic attack, and prior recovered stroke were similar in terms of life-table outcome over the follow-up period. Sixty-two percent of symptomatic patients were alive and free of stroke at 5 years. The late risk of stroke (after 30 days postoperatively) averaged 1.7% per year based on a linear approximation to the hazard at each life-table interval (1.3% per year for ipsilateral stroke). The trend of late stroke risk was clearly downward, however, and could be fitted more accurately by an exponential decay function with a half-life of 33 months. Thus, the risk of stroke following carotid endarterectomy for neurological symptoms was highest in the perioperative period, slowly declined with time, and occurred predominantly ipsilateral to the procedure. The definition of a prospective medical control group remains crucial for a critical analysis of treatment modalities following the onset of premonitory neurological symptoms. In the absence of an adequate control group for this series, the calculated perioperative and postoperative stroke risk from this study was compared to data obtained from the literature on stroke risk in medically treated symptomatic patients. This uncontrolled comparison of treatment modalities suggests the combined perioperative and postoperative stroke risk associated with carotid endarterectomy to be modestly improved over medical treatment alone.

Frey's syndrome: treatment with botulinum toxin

1997 ◽  
Vol 111 (9) ◽  
pp. 839-844 ◽  
Author(s):  
A. Bjerkhoel ◽  
O. Trobbe
Keyword(s):  
Botulinum Toxin ◽  
Botulinum Toxin A ◽  
Surgical Techniques ◽  
Frey’S Syndrome ◽  
Need For Treatment ◽  
Gustatory Sweating ◽  
Parotid Surgery ◽  
Frey's Syndrome ◽  
Before And After ◽  
Set Up

AbstractFrey's syndrome, i.e. gustatory sweating on the cheek, is a fairly common embarrassment after parotid gland surgery. New surgical techniques have been proposed to avoid this complication, but are not widely in use. Hence, there is need for treatment of Frey's syndrome. All surgical and topical treatments have drawbacks. This study was set up in order to evaluate a recently described treatment. One hundred and two patients were interviewed after parotidectomy. Thirty-one of them had noticed gustatory sweating and 15 patients underwent Minor's starch iodine test before, and after, treatment with intracutaneous injections of botulinum toxin A (Botox®, Allergan Inc., USA). Thirteen of the patients did not experience any gustatory sweating at follow-up (one to 13 months). Minor's starch test showed total disappearance of gustatory sweating in 12 of the 15 treated patients. The only side effect was a discreet, transitory affection of the orbicularis oris muscle in one patient. As this treatment is minimally invasive it could be an attractive treatment for Frey's syndrome if the effect is maintained. Complaints of local hypoaesthesia and pain were also common after parotid surgery.

Treatment of gustatory sweating (Frey's syndrome) with botulinum toxin A

Head & Neck ◽  
2003 ◽  
Vol 25 (8) ◽  
pp. 624-628 ◽  
Author(s):  
Andr� Eckardt ◽  
Christian Kuettner
Keyword(s):  
Botulinum Toxin ◽  
Botulinum Toxin A ◽  
Frey’S Syndrome ◽  
Toxin A ◽  
Gustatory Sweating ◽  
Frey's Syndrome

How should a subarachnoid hemorrhage grading scale be determined? A combinatorial approach based solely on the Glasgow Coma Scale

1999 ◽  
Vol 90 (4) ◽  
pp. 680-687 ◽  
Author(s):  
Kiyoshi Takagi ◽  
Akira Tamura ◽  
Tadayoshi Nakagomi ◽  
Hitoshi Nakayama ◽  
Osamu Gotoh ◽  
...  
Keyword(s):  
Subarachnoid Hemorrhage ◽  
Glasgow Coma Scale ◽  
Treatment Modalities ◽  
Combinatorial Approach ◽  
Content Type ◽  
Coma Scale ◽  
Break Points ◽  
Grading Scale ◽  
Gcs Score ◽  
Fine Print

Object. The purpose of this study was to present a combinatorial approach used to develop a subarachnoid hemorrhage (SAH) grading scale based on the patient's preoperative Glasgow Coma Scale (GCS) score.Methods. There are 4094 different combinations that can be used to compress the 13 scores of the GCS into two to 12 grades. Break points, the positions in the scale in which two adjacent scores connote a significantly different outcome, are obtained by a direct comparison of the GCS and the Glasgow Outcome Scale (GOS). Guided by the break points, the number of combinations to be considered can be limited. All possible combinations are statistically analyzed with respect to intergrade differences in outcome. Single combinations, with the maximum number of grades having maximum intergrade outcome differences for each corresponding set of adjacent grades, must be selected. The authors verified the validity of this combinatorial approach by retrospectively analyzing 1398 consecutive patients with aneurysmal SAH who underwent surgery within 7 days of the last hemorrhage episode. The patients' GCS scores were assessed just before surgery and their GOS scores were estimated 6 months post-SAH. The combinatorial approach yields only one acceptable grading scale: I (GCS Score 15); II (GCS Scores 11–14); III (GCS Scores 8–10); IV (GCS Scores 4–7); and V (GCS Score 3).Conclusions. The combinatorial approach, guided by the break points, is so simple and systematic that it can be used again in the future when revision of the grading scale becomes necessary after development of new and effective treatment modalities that improve patients' overall outcome.

Sudden loss of vision following alcohol block of the infraorbital nerve

1973 ◽  
Vol 38 (5) ◽  
pp. 655-657 ◽  
Author(s):  
James W. Markham
Keyword(s):  
Blood Vessels ◽  
Infraorbital Nerve ◽  
Unusual Complication ◽  
Predisposing Factor ◽  
Content Type ◽  
Sudden Loss ◽  
Loss Of Vision ◽  
Tic Douloureux ◽  
Retinal Artery ◽  
Fine Print

✓ The unusual complication of sudden ipsilateral loss of vision following alcohol block of the infraorbital nerve for tic douloureux is described and the possible physiopathology discussed. The author believes that spasm of the central retinal artery was the cause and that repeated injections may have been a predisposing factor. Review of other cases reported shows that the loss of vision has been attributed to embolization of injected agents via collateral blood vessels.

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