The Schizophrenic Psychosis

Schizophrenia ◽  
2020 ◽  
pp. 28-60
Author(s):  
Andrew Crider
1992 ◽  
Vol 22 (3) ◽  
pp. 269-274 ◽  
Author(s):  
Mohamed Sabaawi ◽  
Jose Gutierrez-Nunez ◽  
M. Richard Fragala

A patient whose clinical presentation met criteria for schizophreniform disorder was ultimately found to have neurosarcoidosis, and the psychiatric symptoms responded to steroid treatment. The ongoing search for organic etiology was prompted by the presence of cognitive decline, perseveration and rare bizarre automatisms. This is virtually the first reported association between schizophreniform disorder and sarcoidosis. We reviewed the literature on neurologic involvement and psychiatric manifestations in sarcoidosis as well as the concurrence between organicity and schizophrenic psychosis. The importance of attending to all elements of the mental status examination in a patient with complex atypical findings is underscored.


1989 ◽  
Vol 155 (S5) ◽  
pp. 112-116 ◽  
Author(s):  
Kurt Hahlweg ◽  
Eli Feinstein ◽  
Ursula Müller ◽  
Matthias Dose

Hypotheses on the relationship of schizophrenia and family variables have changed considerably over the last 15 years: whereas speculations on the causal role of familial interaction for the onset of schizophrenic psychosis previously dominated the field of psychological theorising and psychotherapy (Bateson et al, 1956), it was not possible to confirm these theories empirically. In accordance with the research on Expressed Emotion (EE), a shift in emphasis to the influence of family variables on the further course of the illness has taken place. As a consequence, promising new techniques have been developed for the prevention or postponement of relapse.


1992 ◽  
Vol 161 (S18) ◽  
pp. 52-58 ◽  
Author(s):  
Pekka Tienari

It is accepted that schizophrenia runs in families, but whether this relates to genetic or psychosocial transmission is an unanswered question. Kendler (1988) has articulated four testable hypotheses: (a) a general liability to any psychiatric illness (b) a liability to schizotypal functioning — oddness, suspiciousness etc. (c) a liability to broadly defined schizophrenic psychosis, or any functional, non-affective psychosis and (d) a specific liability to narrowly defined schizophrenia, e.g. using DSM-III-R criteria (American Psychiatric Association, 1987). Kendler suggests that neither hypothesis (a) nor (d) is correct, and that the familial predisposition is neither completely non-specific nor highly specific; available results strongly support the second hypothesis and also provide some support in favour of the third.


2020 ◽  
Vol 288 ◽  
pp. 113001 ◽  
Author(s):  
M. Fischer ◽  
A.N. Coogan ◽  
F. Faltraco ◽  
J. Thome

The Lancet ◽  
1983 ◽  
Vol 321 (8332) ◽  
pp. 1048-1049 ◽  
Author(s):  
Irl Extein ◽  
A.L.C. Pottash ◽  
MarkS. Gold

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