scholarly journals Giant Left Atrium and Atrial Fibrillation in Rheumatic Heart Disease Patient with Normally Functioning Mitral Valve Prosthesis

2021 ◽  
pp. 1-2
Author(s):  
Rohit Walia ◽  
◽  
Udit Chauhan ◽  
Author(s):  
Gëzim Berisha ◽  
Edmond Haliti ◽  
Gani Bajraktari

The giant left atrium (GLA) is a rare condition, commonly associated with rheumatic mitral valve disease, and very rarely with non rheumatic heart disease (nRHD). The triple valvular heart disease with involved mitral, aortic and tricuspid valves is quite uncommon. A 47 year female patient with a past medical history of rheumatic heart disease (RHD) and known severe mitral stenosis was with severe breathlessness (NYHA class IV). She had undergone mitral valve commissurotomy and tricuspid valve annuloplasty 12 years previously.  Transthoracic echocardiography revealed a giant left atrium, moderate to severe mitral valve restenosis, severe mitral regurgitation, moderate aortic regurgitation and severe tricuspid regurgitation, associated with severe secondary pulmonary hypertension and a markedly dilated right heart chambers. The patient was considered inoperable by the heart team, because of advanced pulmonary hypertension predicting a very high risk for open heart surgery. The final treatment decision was a difficult and complex issue.


2021 ◽  
Vol 07 (05) ◽  
Author(s):  
Soumia Faid ◽  

Giant left atrium is a condition characterized by an extreme enlargement of the left atrium with a diameter more than 80 mm and it is usually associated with long standing rheumatic mitral valve disease. We present a case of giant left atrium in a 47-year-old female patient who had a history of rheumatic heart disease, severe mitral disease, permanent atrial fibrillation and causing the compression of adjacent intra-thoracic structures. The patient underwent a successful mitral valve replacement with reduction of the enlarged sized left atrium with good outcomes.


1971 ◽  
Vol 174 (2) ◽  
pp. 194-201 ◽  
Author(s):  
J. PLASCHKES ◽  
J. B. BORMAN ◽  
G. MERIN ◽  
H. MILWIDSKY

2009 ◽  
Vol 4 (5) ◽  
pp. 435-437
Author(s):  
Gaspare Parrinello ◽  
Daniele Torres ◽  
Salvatore Paterna ◽  
Manuela Mezzero ◽  
Pietro Di Pasquale ◽  
...  

Heart ◽  
2016 ◽  
Vol 102 (15) ◽  
pp. 1206-1214 ◽  
Author(s):  
Ho Jin Kim ◽  
Joon Bum Kim ◽  
Sung-Ho Jung ◽  
Suk Jung Choo ◽  
Cheol Hyun Chung ◽  
...  

2016 ◽  
Vol 7 (3) ◽  
pp. 126-127
Author(s):  
Anshul Kumar Gupta ◽  
Sunil Dhondiram Shewale ◽  
Kanchanahalli Siddegowda Sadananda ◽  
Chollenahally Nanjappa Manjunath

2021 ◽  
Vol 10 (2) ◽  
pp. 83-88
Author(s):  
Yu. E. Teplova ◽  
I. N. Lyapina ◽  
V. A. Shaleva ◽  
O. K. Kuzmina ◽  
A. V. Evtushenko ◽  
...  

Aim. To study “the portrait” of patients with acquired mitral valve (MV) heart disease of various origins and pulmonary hypertension hospitalized for surgical correction of the defect.Methods. The study included 97 patients with acquired diseases of mitral valve and pulmonary hypertension. The assessment of demographic, clinical and anamnestic data, indicators of transthoracic echocardiography, quality of life before the correction of MV defect was carried out.Results. The studied cohort is mostly represented by female patients (n = 70; 72.2%). The most common cause of mitral valve disease was rheumatic heart disease (n = 40; 41.2%). Overweight, hypertension (n = 76; 78.4%) and atrial fibrillation (n = 62; 63.9%) were the most common comorbidities. The mean pressure level in the pulmonary artery according to echocardiography was 35.5 (29.0; 40.0) mm Hg, with no significant difference among the patients, regardless the mitral defect etiology. Less pronounced remodeling of the left ventricle was noted in patients with rheumatic heart disease, which is caused by a lesion of the MV by the type of stenosis in contrast to patients with connective tissue dysplasia syndrome or against the background of detachment of MV chords with MV damage in the form of its insufficiency. There were no significant differences in the systolic function of the right ventricle depending on the etiology of MV defect.Conclusion. The “portrait” of a patient with pulmonary hypertension associated with an acquired mitral valve defect before its correction is the predominance of female, overweight, with II or III functional class of chronic heart failure, more frequent rheumatic genesis of MV defect, the presence of concomitant pathology in the form of hypertension and persistent atrial fibrillation, and increased size of the left atrium and left ventricle, reduced systolic function of the right ventricle according to the data of Echocardiography. 


2021 ◽  
Vol 14 (11) ◽  
pp. e244270
Author(s):  
Mark Zachary Johnson ◽  
Nicholas James Damianopoulos ◽  
Felicity Lee ◽  
Gerald Yong

A 32-year-old, 11-week pregnant African woman with known rheumatic heart disease presented to the emergency department with worsening shortness of breath on exertion. She had undergone a double bioprosthetic valve replacement and left atrial appendage resection 8 years prior for severe mitral stenosis, moderate mitral regurgitation and moderate aortic regurgitation. A transo-oesophageal echocardiography at this presentation confirmed a morphologically calcified and stenosed mitral bioprosthesis, with moderate stenosis of her aortic bioprosthesis. Her multidisciplinary team, including cardiologists, cardiothoracic surgeons and obstetricians, came to a consensus decision to proceed with a transseptal transcatheter valve implantation within the mitral valve prosthesis (valve-in-valve implantation). Transthoracic echocardiography performed 2 months post procedure showed satisfactory mitral valve gradients and at 30 weeks’ gestation, she successfully delivered her fifth child. 2 years later, the valve in valve complex is still functioning well.


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