scholarly journals Dysphagia caused by Anterior Cervical Osteophyte: A Rare Entity Revisited

2012 ◽  
Vol 3 (3) ◽  
pp. 168-171
Author(s):  
Prashanth Veerabhadraiah ◽  
Vishal Rao ◽  
Raghavendra Shankar ◽  
Naveen Shivappa ◽  
TM Nagaraj

ABSTRACT Large anterior cervical osteophytes can occur in degeneration of the cervical spine, cervical spondylosis or in diffuse idiopathic skeletal hyperostosis (DISH). Voluminous anterior cervical osteophytes which can develop from C3 to C7 can cause narrowing of the pharyngoesophageal segment by external compression and may cause dysphagia, which may be life threatening when it is associated with aspiration and or dyspnea. The objective of this case report is to highlight how commonly occurring anterior cervical osteophytes may become an unrecognized cause for life-threatening dysphagia. The clinical and radiographic findings in patient with dysphagia and ventral osteophytes of the cervical spine due to degeneration are demonstrated. The anterolateral approach for removal of these osteophytes is described. How to cite this article Veerabhadraiah P, Rao V, Shankar R, Shivappa N, Kumar P, Nagaraj TM. Dysphagia caused by Anterior Cervical Osteophyte: A Rare Entity Revisited. Int J Head and Neck Surg 2012;3(3):168-171.

Author(s):  
Uday Kiran Katari

<p class="abstract">Dysphagia may occur in various pathological, inflammatory diseases of esophagus. It may also occur due to motility disorders of esophagus, benign and malignant diseases of mediastinum, cervical spine diseases. Dysphagia secondary to compression of esophagus by a cervical osteophyte is rare. The most common causes of osteophyte (bony outgrowth) in the cervical spine are diffuse idiopathic skeletal hyperostosis (DISH), ankylosing spondylitis (AS), and cervical spondylosis. Patients with cervical osteophytes are mostly asymptomatic. Hence, when considering cervical osteophytes as a cause of dysphagia other pathologic entities in the esophagus (e.g. tumors, webs, rings, strictures) should be excluded. We present a 68 year female patient who presented with complaints of dysphagia and neck stiffness since 3 months. She has been evaluated and found that dysphagia is due to large anterior cervical osteophytes compressing pharynx at C2/C3 and esophagus at C5/C6 and C6/C7 vertebral levels respectively. The objective of this case report is to emphasize the importance of anterior cervical osteophyte as a cause of dysphagia in elderly.</p>


Head & Neck ◽  
2015 ◽  
Vol 37 (9) ◽  
pp. E115-E119 ◽  
Author(s):  
Yohan Song ◽  
Suzanne Tharin ◽  
Vasu Divi ◽  
Laura M. Prolo ◽  
Davud B. Sirjani

2020 ◽  
Vol 57 ◽  
pp. 37-40
Author(s):  
Mikołaj Dąbrowski ◽  
Adam Sulewski ◽  
Jacek Kaczmarczyk ◽  
Łukasz Kubaszewski

2019 ◽  
Vol 12 (9) ◽  
pp. e231035
Author(s):  
Anju Gupta ◽  
Nishkarsh Gupta

Vallecular cysts are a rare entity but can complicate the airway management due to the physical impediment and distortion of the laryngeal inlet. It can lead to fatal life-threatening airway obstruction postinduction of anaesthesia. Infancy compounds the risks due to lack of cooperation and physiological reserves. The literature review suggested most anaesthesiologists avoided muscle relaxants and aspiration of the cyst was used as a rescue technique. The present case report illustrates the successful airway management of an infant with a huge vallecular cyst using fibre-optic intubation by a ‘three-person’ technique after paraglossal videolaryngoscopy failed to secure the airway.


2020 ◽  
Vol 4 (4) ◽  
pp. 610-612
Author(s):  
Maddi Massa ◽  
Karla Newbold

Introduction: Gastric diverticula (GD) are uncommon. Most are asymptomatic and diagnosed incidentally. Symptoms range from reflux and epigastric discomfort to life-threatening bleeding and perforation. We describe a case of symptomatic GD presenting as hematemesis requiring surgical treatment. Case Report: A 57-year-old female presented to the emergency department (ED) with one day of epigastric pain and hematemesis. Hemoglobin was found to be stable, but blood urea nitrogen was elevated. Imaging revealed a fundal GD. Esophagogastroduodenoscopy did not show other etiology of hematemesis. The patient underwent partial gastric resection for GD removal and did well without further symptoms on follow-up. Conclusion: Although rare, GD needs to be included on a differential diagnosis when evaluating gastrointestinal symptoms in the ED. Patients may present with an array of complaints but can potentially develop serious complications. Providers should be familiar with the diagnostic options and treatment regimens available to better care for patients presenting with GD.


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