scholarly journals Effect of Corticosterone Administration on Small Intestinal Weight and Expression of Small Intestinal Nutrient Transporter mRNA of Broiler Chickens

2009 ◽  
Vol 23 (2) ◽  
pp. 175-181 ◽  
Author(s):  
X. F. Hu ◽  
Y. M. Guo ◽  
B. Y. Huang ◽  
L. B. Zhang ◽  
S. Bun ◽  
...  
Keyword(s):  
Broiler Chickens ◽  
Nutrient Transporter ◽  
Small Intestinal ◽  
Intestinal Weight

Effects of early posthatch feeding on growth, organ development, and blood biochemical profile in broiler chickens

2019 ◽  
Vol 99 (2) ◽  
pp. 418-424
Author(s):  
H.K. Kang ◽  
H.T. Bang ◽  
C.H. Kim ◽  
J.J. Jeon ◽  
H.S. Kim ◽  
...  
Keyword(s):  
Body Weight ◽  
Weight Gain ◽  
Broiler Chickens ◽  
Body Weight Gain ◽  
Feeding Time ◽  
Biochemical Profile ◽  
Early Feeding ◽  
Blood Biochemical ◽  
Small Intestinal ◽  
Intestinal Weight

The effects of early feeding time on growth performance, organ weight, blood biochemical, and leukocyte profile were investigated in posthatch broiler chickens to 35 d of age. From 7 to 21 d, early feeding (3 h after hatching) had a significant (p < 0.01) effect on body weight gain, and at 14 d, chicks fed 3 and 12 h after hatching were significantly heavier (p < 0.01) than those fed later. At 7 d, feed intake (FI) was significantly higher (p < 0.01) among chicks fed 3 and 12 h after hatching. At 21 d, chicks fed 3 h after hatching showed significantly higher FI (p < 0.01) than those fed later. At 3 d, gizzard weight was significantly heavier (p < 0.05) in chicks fed 24, 36, and 48 h after hatching than in those fed earlier, and small intestinal weight was also significantly higher (p < 0.05). At 35 d of age, alanine aminotransferase was higher (p < 0.01) in chicks fed 36 and 48 h after hatching, and the eosinophil level was significantly higher (p < 0.01) in chicks fed 3, 24, 36, and 48 h after hatching.

scholarly journals Interactions between starch source and gelatinisation degree on performance and small intestinal digestion in broiler chickens

2021 ◽  
pp. 1-11
Author(s):  
K. Itani ◽  
J. Ø. Hansen ◽  
B. Kierończyk ◽  
A. Benzertiha ◽  
P. P. Kurk ◽  
...  
Keyword(s):  
Broiler Chickens ◽  
Intestinal Digestion ◽  
Small Intestinal

scholarly journals INFECTIOUS STUNTING SYNDROME ASSOCIATED WITH DISTURBANCES OF MINERAL METABOLISM AND BONE DEVELOPMENT IN BROILER CHICKENS

1999 ◽  
Vol 47 (3) ◽  
pp. 361-378 ◽  
Author(s):  
Glávits ◽  
G. Sályi ◽  
R. Glávits
Keyword(s):  
Growth Retardation ◽  
Broiler Chickens ◽  
Neuronal Degeneration ◽  
Bone Development ◽  
Mineral Metabolism ◽  
Digestive Enzyme ◽  
Small Intestinal Mucosa ◽  
Vacuolar Degeneration ◽  
Leg Weakness ◽  
Small Intestinal

On a broiler farm with a rearing capacity of about 200,000 chickens, a disease characterised by growth retardation, variability in chick size, 'leg weakness', diarrhoea and increased mortality at 3 weeks of age occurred repeatedly, in several successive broiler flocks. Gross and histopathological findings were dominated by widening of the hypertrophic and ossification layers of the physes of long bones as well as by thickening, unevenness and defective calcification of the cartilage trabeculae. In the parathyroid gland, vacuolar degeneration of the cytoplasm of glandular epithelial cells, connective tissue proliferation and, here and there, cyst formation were seen. Additional findings included severe cerebellar oedema and neuronal degeneration. The pancreatic, myocardial and intestinal changes typical of infectious stunting syndrome (ISS) occurred only in a mild form. Four-week-old chickens exhibiting 'leg weakness' had significantly lower blood inorganic phosphate concentration and tibial ash content as compared to healthy chickens. The disease was successfully transmitted by oral administration of small intestinal homogenate from affected chickens. In a second experiment, however, the disease could not be transmitted with intestinal homogenate sterilized by irradiation. Large doses of vitamin D3reduced the rate of growth retardation and defective calcification of bones. The digestive enzyme activities of the pancreas and small intestinal mucosa of 'infected' chickens were decreased as is typical of ISS.

Intestinal growth is associated with elevated levels of glucagon-like peptide 2 in diabetic rats

1997 ◽  
Vol 273 (4) ◽  
pp. E815-E820 ◽  
Author(s):  
Kirk D. Fischer ◽  
Savita Dhanvantari ◽  
Daniel J. Drucker ◽  
Patricia L. Brubaker
Keyword(s):  
Experimental Diabetes ◽  
Plasma Concentrations ◽  
Diabetic Rats ◽  
Diabetic Rat ◽  
Villus Height ◽  
Intestinal Growth ◽  
Glucagon Like Peptide ◽  
Small Intestinal ◽  
The Relationship ◽  
Intestinal Weight

Glucagon-like peptide 2 (GLP-2) has recently been identified as a novel intestinal growth factor. Because experimental diabetes is associated with bowel growth, we examined the relationship between GLP-2 and intestinal growth in rats made diabetic by streptozotocin (STZ) injection and treated with or without insulin for 3 wk. Ileal concentrations of the intestinal proglucagon-derived peptides, i.e., glicentin + oxyntomodulin, and GLPs 1 and 2, were increased by 57 ± 20% above those of controls in untreated STZ diabetes ( P < 0.05–0.001). Similar increases in plasma concentrations of glicentin + oxyntomodulin (77 ± 15% above controls, P < 0.01) and GLP-2 (91 ± 32% above controls, P < 0.05) were seen in untreated STZ diabetes. Both wet and dry small intestinal weight increased by 74 ± 20% above controls ( P < 0.01) in STZ diabetes, and macromolecular analysis indicated parallel increases in both protein ( P < 0.001) and lipid ( P < 0.05) content. Villus height ( P < 0.001) and crypt depth ( P < 0.01) were also increased in untreated diabetic rat intestine. Insulin therapy prevented the changes in plasma GLP-2 and intestinal mass seen in untreated STZ diabetes. Thus STZ diabetes is associated with both increased production of GLP-2 and enhanced bowel weight, thereby suggesting a role for GLP-2 in diabetes-associated bowel growth.

scholarly journals Maternal supplementation of seaweed-derived polysaccharides improves intestinal health and immune status of suckling piglets

2015 ◽  
Vol 4 ◽  
Author(s):  
G. Heim ◽  
J. V. O'Doherty ◽  
C. J. O'Shea ◽  
D. N. Doyle ◽  
A. M. Egan ◽  
...  
Keyword(s):  
Gene Expression ◽  
Dietary Treatment ◽  
Intestinal Morphology ◽  
Cytokine Gene Expression ◽  
Cytokine Gene ◽  
Lps Challenge ◽  
Nutrient Transporter ◽  
Suckling Piglets ◽  
Small Intestinal ◽  
Weaning Piglets

AbstractThe experiment investigated the effect of maternal dietary supplementation of seaweed-derived polysaccharides (SDP) (–SDPv.+SDP,n   20) from day 83 of gestation until weaning (day 28) on selected sow faeces and piglet digesta microbiota populations, piglet small-intestinal morphology, and intestinal nutrient transporter and inflammatory cytokine gene expression at birth, 48 h after birth and weaning. The effect of maternal dietary treatment on the piglet gene expression profile of inflammatory cytokines in the colon following a lipopolysaccharide (LPS) challenge was also investigated. Dietary SDP reduced sow faecal Enterobacteriaceae gene numbers at parturition. Small-intestinal morphology, nutrient transporter and cytokine gene expression in newborn piglets did not differ between maternal dietary treatments (P > 0·10). At 48 h after birth, sodium–glucose-linked transporter 1 gene expression was down-regulated in the ileum of piglets suckling the SDP-supplemented sows compared with those suckling the basal sows (P = 0·050). There was a SDP × LPS challenge interaction onIL-1andIL-6gene expression in the colon of piglets (P < 0·05). The gene expression ofIL-1andIL-6was down-regulated in the LPS-challenged colon of piglets suckling the SDP sows compared with those suckling the basal sows (P < 0·05). However, there was no difference inIL-1andIL-6gene expression in the unchallenged colon between treatment groups. At weaning, piglets suckling the SDP-supplemented sows had increased villus height in the jejunum and ileum compared with those suckling the basal-fed sows (P < 0·05). In conclusion, maternal dietary SDP supplementation enhanced the immune response of suckling piglets and improved gut morphology, making them more immune competent to deal with post-weaning adversities.

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