scholarly journals Recurrent Bell’s Palsy During Takeoff on a Commercial Flight: A Case Report

2021 ◽  
Vol 5 (1) ◽  
Author(s):  
Gayle Galletta
Keyword(s):  
Case Report ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Commercial Flight

Late Surgical Decompression of Facial Nerve in Bell's Palsy: A Case Report

2012 ◽  
Vol 73 (S 02) ◽  
Author(s):  
L. M. Marques ◽  
J. Pimentel ◽  
P. Escada ◽  
G. Neto D'Almeida
Keyword(s):  
Case Report ◽  
Facial Nerve ◽  
Bell’S Palsy ◽  
Surgical Decompression ◽  
Bell's Palsy

scholarly journals COMPLETE MANAGEMENT OF ARDITA (BELL’S PALSY) AS PER AYURVEDA CHIKITSA SUTRA – A CASE REPORT

2021 ◽  
Vol 09 (3) ◽  
pp. 650-656
Author(s):  
Ram Lakhan Meena ◽  
Santoshkumar Bhatted ◽  
Nilam Meena
Keyword(s):  
Case Report ◽  
Adverse Effects ◽  
Facial Nerve ◽  
Present Report ◽  
Complete Recovery ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Contemporary Science ◽  
The Face ◽  
Grading Scale

Bell’s palsy, also known as acute idiopathic lower motor neuron facial paralysis, is characterized by sud-den onset paralysis or weakness of the muscles to one side of the face controlled by the facial nerve. In contemporary science, administration of steroids is the treatment of choice for complete facial palsy. Cer-tain Panchakarma procedures and internal Ayurvedic medicines have been proved to be beneficial in the management of Ardita vata. The present report deals with a case of 62-year-old male patient diagnosed as Ardita vata was treated with various Panchakarma procedures like Nasya, Shirobasti, Kukkutanda Swedana, Dashmoola Ksheer Dhoom, Gandoosh and oral Ayurveda medicines. Criteria of assessment was based on the scoring of House-Brackmann Facial Nerve Grading scale. After completion of Ayurveda treatment, the patient Shown almost complete recovery without any adverse effects. This case is an evi-dence to demonstrate the effectiveness of Ayurveda treatment in case of Ardita vata (Bell’s palsy).

Neural mobilization in Bell’s palsy: A case report

CRANIO® ◽  
2019 ◽  
pp. 1-4
Author(s):  
Faizan Zaffar Kashoo ◽  
Mazen Alqahtani ◽  
Mehrunnisha Ahmad
Keyword(s):  
Case Report ◽  
Bell’S Palsy ◽  
Bell's Palsy

Idiopathic facial paralysis (Bell’s palsy) in the immediate puerperium in a patient with mild preeclampsia: a case report

2005 ◽  
Vol 272 (3) ◽  
pp. 241-243 ◽  
Author(s):  
Ioannis Mylonas ◽  
Ralph Kästner ◽  
Christina Sattler ◽  
Franz Kainer ◽  
Klaus Friese
Keyword(s):  
Case Report ◽  
Facial Paralysis ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Mild Preeclampsia

Familal Bell's Palsy (Case Report)

1966 ◽  
Vol 80 (6) ◽  
pp. 628-630 ◽  
Author(s):  
Prem. K. Kakar ◽  
K. L. Sawhney ◽  
P. S. Saharia
Keyword(s):  
Case Report ◽  
Bell’S Palsy ◽  
Bell's Palsy

scholarly journals Familial Bell’s Palsy: A Case Report and Literature Review

2012 ◽  
Vol 2012 ◽  
pp. 1-3
Author(s):  
Mark Kubik ◽  
Liliana Robles ◽  
Doris Kung
Keyword(s):  
Case Report ◽  
Bell’S Palsy ◽  
Facial Nerve Paralysis ◽  
Bell's Palsy ◽  
Unique Case ◽  
Nerve Paralysis ◽  
Csf Analysis ◽  
History Of ◽  
Sheer Number ◽  
Rule Out

Objective. To describe a unique case of familial Bell’s palsy and summarize the current literature regarding possible hereditary influences.Design. Case report.Main Outcome Measures. Clinical exam, CSF analysis, and family history provided per the patient.Results. We report the case of a 58-year-old female who presented with recurrent and bilateral episodes of facial palsy. The patient underwent multiple CSF investigations to rule out a possible infectious and rheumatologic etiology that were all negative. Further questioning revealed she was one of seven family members with a history of unilateral facial nerve paralysis.Conclusion. The sheer number of similar case studies to date suggests that familial clustering of Bell’s palsy is a real, noncoincidental phenomenon. Our case represents a unique and perplexing example of one such family. Familial Bell’s palsy may represent an autoimmune disease secondary to inherited HLA alloantigens or a structural predisposition to disease based on the dimensions of the facial canal.

Scleroderma Mimicking Bell's Palsy: Case Report

2015 ◽  
Vol 120 (2) ◽  
pp. e57
Author(s):  
IGOR HENRIQUE MORAIS SILVA ◽  
SYLVIA SAMPAIO PEIXOTO ◽  
VÂNIA RIBEIRO ◽  
LUIZ ALCINO MONTEIRO GUEIROS ◽  
JAIR CARNEIRO LEÃO ◽  
...  
Keyword(s):  
Case Report ◽  
Bell’S Palsy ◽  
Bell's Palsy

Not all facial paralysis is bell's palsy: A case report

1999 ◽  
Vol 80 (7) ◽  
pp. 857-859 ◽  
Author(s):  
Jennifer S. Brach ◽  
Jessie M. Vanswearingen
Keyword(s):  
Case Report ◽  
Facial Paralysis ◽  
Bell’S Palsy ◽  
Bell's Palsy

Time-Dependent Recovery of Human Synovial Membrane Mesenchymal Stem Cell Function After High-Dose Steroid Therapy: Case Report and Laboratory Study

2017 ◽  
Vol 46 (3) ◽  
pp. 695-701 ◽  
Author(s):  
Yukihiko Yasui ◽  
David A. Hart ◽  
Norihiko Sugita ◽  
Ryota Chijimatsu ◽  
Kota Koizumi ◽  
...  
Keyword(s):  
Stem Cell ◽  
Case Report ◽  
Mesenchymal Stem Cell ◽  
Steroid Therapy ◽  
Laboratory Study ◽  
Expression Profiles ◽  
Bell’S Palsy ◽  
High Dose ◽  
Bell's Palsy ◽  
High Dose Steroid

Background: The use of mesenchymal stem cells from various tissue sources to repair injured tissues has been explored over the past decade in large preclinical models and is now moving into the clinic. Purpose: To report the case of a patient who exhibited compromised mesenchymal stem cell (MSC) function shortly after use of high-dose steroid to treat Bell’s palsy, who recovered 7 weeks after therapy. Study Design: Case report and controlled laboratory study. Methods: A patient enrolled in a first-in-human clinical trial for autologous implantation of a scaffold-free tissue engineered construct (TEC) derived from synovial MSCs for chondral lesion repair had a week of high-dose steroid therapy for Bell’s palsy. Synovial tissue was harvested for MSC preparation after a 3-week recovery period and again at 7 weeks after therapy. Results: The MSC proliferation rates and cell surface marker expression profiles from the 3-week sample met conditions for further processing. However, the cells failed to generate a functional TEC. In contrast, MSCs harvested at 7 weeks after steroid therapy were functional in this regard. Further in vitro studies with MSCs and steroids indicated that the effect of in vivo steroids was likely a direct effect of the drug on the MSCs. Conclusion: This case suggests that MSCs are transiently compromised after high-dose steroid therapy and that careful consideration regarding timing of MSC harvest is critical. Clinical Relevance: The drug profiles of MSC donors and recipients must be carefully monitored to optimize opportunities to successfully repair damaged tissues.

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