scholarly journals Loose Anagen Hair Syndrome in a Saudi Girl

2021 ◽  
pp. e2021071
Author(s):  
Mahdi Al Dhafiri ◽  
Muneerah Alhumaidy
Keyword(s):  
Anagen Hair

Effect of Minoxidil Sulfate on the Growth of Human Anagen Hair Follicles Grown in Collagen Gels, and on the Colony Growth of Human Cultured Outer Root Sheath Cells in vitro.

1992 ◽  
Vol 54 (4) ◽  
pp. 727-732 ◽  
Author(s):  
Naoyuki UCHIDA ◽  
Takesi FUJIE ◽  
Seiji ARASE ◽  
Yosiroh NINOMIYA ◽  
Hideki NAKANISI ◽  
...  
Keyword(s):  
Colony Growth ◽  
Hair Follicles ◽  
Collagen Gels ◽  
Outer Root Sheath ◽  
Root Sheath ◽  
Anagen Hair ◽  
Outer Root Sheath Cells ◽  
Sheath Cells

Anagen Effluvium after Therapeutic Scalp Surgery: Unreported Phenomenon

2021 ◽  
Vol 7 (4) ◽  
pp. 311-314
Author(s):  
Leticia Arsie Contin ◽  
Leopoldo Duailibe Nogueira Santos ◽  
Ivan José Netto Pereira ◽  
Vanessa Barreto Rocha
Keyword(s):  
Hair Loss ◽  
Spontaneous Recovery ◽  
Case Reports ◽  
Practical Importance ◽  
Transient Ischemia ◽  
Melanocytic Nevi ◽  
Scarring Alopecia ◽  
Anagen Hair ◽  
Time Required

<b><i>Introduction:</i></b> Many procedures are performed on the scalp, such as excision of pilar and sebaceous cysts, melanocytic nevi, and reduction surgery for scarring alopecia, among others. In hair transplants, telogen effluvium is often reported 3 months after surgery; however, hair loss usually happens much earlier, around second week after the procedure, not compatible with the time required for hair to enter telogen and exogenous phases in normal conditions. <b><i>Case Reports:</i></b> We report 3 cases of anagen hair loss 4 weeks after surgeries, with perilesional trichoscopy suggesting anagen effluvium, with typical signs such as black dots and exclamation hairs. <b><i>Discussion:</i></b> There are only a few reports about hair loss around operated areas. The cause of this postoperative anagen effluvium is probably a transient ischemia. <b><i>Conclusion:</i></b> The practical importance of this phenomenon is to properly orient patients because most of the hair will be lost, since 85% of them are anagens, and also will have spontaneous recovery in the next 3 months.

scholarly journals Desmogleins 1 and 3 in the Companion Layer Anchor Mouse Anagen Hair to the Follicle

2004 ◽  
Vol 123 (5) ◽  
pp. 817-822 ◽  
Author(s):  
Yasushi Hanakawa ◽  
Hong Li ◽  
Chenyan Lin ◽  
John R. Stanley ◽  
George Cotsarelis
Keyword(s):  
Anagen Hair

Loose Anagen Hair Syndrome in Black-Haired Indian Children

2013 ◽  
Vol 30 (5) ◽  
pp. 579-583 ◽  
Author(s):  
Vivek Dey ◽  
Manasi Thawani
Keyword(s):  
Indian Children ◽  
Anagen Hair

Nuclear Aberrations in Anagen Hair Follicle Cells; A Novel In Vivo Assay for the Detection of Genotoxic Agents

1990 ◽  
pp. 283-293
Author(s):  
M. T. Goldberg ◽  
E. A. Goldberg ◽  
R. Church ◽  
M. H. Hardy
Keyword(s):  
Hair Follicle ◽  
Follicle Cells ◽  
In Vivo Assay ◽  
Genotoxic Agents ◽  
Anagen Hair

scholarly journals The RASopathy Family: Consequences of Germline Activation of the RAS/MAPK Pathway

2018 ◽  
Vol 39 (5) ◽  
pp. 676-700 ◽  
Author(s):  
Mylène Tajan ◽  
Romain Paccoud ◽  
Sophie Branka ◽  
Thomas Edouard ◽  
Armelle Yart
Keyword(s):  
Noonan Syndrome ◽  
Mapk Pathway ◽  
Genetic Disorders ◽  
Mapk Signaling ◽  
Mendelian Inheritance ◽  
Disease Genes ◽  
Type I ◽  
Future Directions ◽  
Legius Syndrome ◽  
Anagen Hair

Abstract Noonan syndrome [NS; Mendelian Inheritance in Men (MIM) #163950] and related syndromes [Noonan syndrome with multiple lentigines (formerly called LEOPARD syndrome; MIM #151100), Noonan-like syndrome with loose anagen hair (MIM #607721), Costello syndrome (MIM #218040), cardio-facio-cutaneous syndrome (MIM #115150), type I neurofibromatosis (MIM #162200), and Legius syndrome (MIM #611431)] are a group of related genetic disorders associated with distinctive facial features, cardiopathies, growth and skeletal abnormalities, developmental delay/mental retardation, and tumor predisposition. NS was clinically described more than 50 years ago, and disease genes have been identified throughout the last 3 decades, providing a molecular basis to better understand their physiopathology and identify targets for therapeutic strategies. Most of these genes encode proteins belonging to or regulating the so-called RAS/MAPK signaling pathway, so these syndromes have been gathered under the name RASopathies. In this review, we provide a clinical overview of RASopathies and an update on their genetics. We then focus on the functional and pathophysiological effects of RASopathy-causing mutations and discuss therapeutic perspectives and future directions.

Short anagen hair naevus: improvement after treatment with 5% topical minoxidil

2007 ◽  
Vol 46 (7) ◽  
pp. 757-759 ◽  
Author(s):  
Sylvia van Egmond ◽  
Carlijn Hoedemaker ◽  
Rodney Sinclair
Keyword(s):  
Topical Minoxidil ◽  
Anagen Hair

Short anagen hair with persistent synchronized pattern of scalp hair growth

2003 ◽  
Vol 49 (5) ◽  
pp. 949-951 ◽  
Author(s):  
Keng-Ee Thai ◽  
Rodney D Sinclair
Keyword(s):  
Hair Growth ◽  
Scalp Hair ◽  
Anagen Hair

Loose anagen hair syndrome

2004 ◽  
pp. 360-360
Keyword(s):  
Anagen Hair
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