scholarly journals Acute Cerebral Infarction Related to Stenosis of Accessory Middle Cerebral Artery

2012 ◽  
Vol 14 (1) ◽  
pp. 43 ◽  
Author(s):  
Sang-Hyeon Son ◽  
Hye-Yeon Choi ◽  
Sang-Beom Kim ◽  
Won-Chul Shin ◽  
Key-Chung Park ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Acute Cerebral Infarction ◽  
Accessory Middle Cerebral Artery

scholarly journals A case of acute cerebral infarction associated with an accessory middle cerebral artery in a patient who underwent thrombectomy

2019 ◽  
Vol 7 (1) ◽  
Author(s):  
Ichiro Deguchi ◽  
Takashi Osada ◽  
Hiroaki Kimura ◽  
Naoko Saito ◽  
Eiichi Arai ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Acute Cerebral Infarction ◽  
Accessory Middle Cerebral Artery

Patterns of Vascular Pathology in Acute, First-Ever Cerebral Infarction

1993 ◽  
Vol 38 (2) ◽  
pp. 41-44 ◽  
Author(s):  
A.R. Naylor ◽  
P.A.G. Sandercock ◽  
R.J. Sellar ◽  
C.P. Warlow
Keyword(s):  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Indirect Evidence ◽  
Acute Cerebral Infarction ◽  
Vascular Pathology ◽  
Clinical Classification ◽  
Anterior Circulation ◽  
Major Vessel ◽  
Preliminary Study

This was a preliminary study to see whether patterns of vascular pathology correlated with a simple clinical classification of acute cerebral infarction. Thirty-two patients with acute, first-ever hemispheric cerebral infarction were prospectively studied clinically together with an extra- and transcranial Doppler ultrasound. All 14 patients with the triad of ipsilateral motor/sensory deficit, homonymous hemianopia and higher cortical dysfunction (Total Anterior Circulation Infarction,[TACI]) had occlusion of the symptomatic middle cerebral artery and/or internal carotid artery, or a severe middle cerebral artery stenosis. Three patients with lacunar infarction had no evidence of major vessel occlusion. Eleven of thirteen patients with Partial Anterior Circulation, Infarction (PACI) I(i.e. only one or two clinical features of the TACI triad) had patent symptomatic major vessels, with indirect evidence to suggest distal branch occlusion(s) of the middle cerebral artery in six. The remaining two PACI patients had major vessel occlusions. Two patients were not clinically classifiable, but both had significant vascular pathology on ultrasound. The findings of this preliminary study therefore suggest that a simple clinical classification was generally capable of predicting patterns of vascular pathology inpatients with acute cerebral infarction, which could have implications for the selection of patients for clinical trials of thrombolytic therapy.

Acute cerebral infarction and changes of regional cerebral blow flow (rCBF) following experimental middle cerebral artery (MCA) occlusion

1988 ◽  
Vol 10 (4) ◽  
pp. 203-212 ◽  
Author(s):  
Dae Hee Han ◽  
Hee-Won Jung ◽  
Sun Ho Lee ◽  
Hyun Jip Kim ◽  
Kil Soo Choi ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Acute Cerebral Infarction ◽  
Mca Occlusion

High-Resolution MRI to Evaluate Benefit of Middle Cerebral Artery Stenosis in Secondary Prevention in Patients with Acute Cerebral Infarction

2021 ◽  
Vol 11 (7) ◽  
pp. 1911-1919
Author(s):  
Jingwei Wu ◽  
Ying Xiao ◽  
Chuanzi Li ◽  
Zhongshi Nie ◽  
Desheng Wang ◽  
...  
Keyword(s):  
Magnetic Resonance ◽  
High Resolution ◽  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Artery Stenosis ◽  
Acute Cerebral Infarction ◽  
Vascular Stenosis ◽  
Middle Cerebral Artery Stenosis ◽  
Cerebral Vascular

High-resolution magnetic resonance (HRMRI) combined with magnetic resonance angiography (MRA) was used to analyze the correlation between the degree of infarction and the degree of cerebral vascular stenosis, and these patients are all have the trend to contain acute infarction. All patients were examined by CT cerebral angiography, which was analysed by conventional magnetic resonance imaging (MRI), high-Resolution (HRMRI) imaging, and cerebral vascular (MRA) imaging. The infarct size of each patient with cerebral infarction was calculated. The degree of middle cerebral artery stenosis was evaluated. Moreover, agnosis of acute cerebral infarction in middle cerebral artery region was calculated, and the correlation between acute cerebral infarction in middle cerebral artery region and the degree of cerebral vascular stenosis was analyzed. The specificity, sensitivity, and accuracy of magnetic resonance angiography (MRA) in the diagnosis of acute cerebral infarction in the blood supply area of the middle cerebral artery were 90.48%, 94.87%, and 93.94%. The coincidence rates for the degree of middle cerebral artery stenosis were 90.91% for mild stenosis, 92.31% for moderate stenosis, 93.75% for severe stenosis, and 85.71% for vascular occlusion, respectively. The K value was in good agreement with the results of CTA diagnosis (≥0.75). In conclusion, HRMRI can clearly indicated that the location of lesions with acute cerebral infarction, and MRA can show the degree of vascular stenosis, there is a close relationship between them.

scholarly journals Acute cerebral infarction with adenomyosis in a young woman: a case report

2019 ◽  
Author(s):  
yuan zhao ◽  
Yongbo Zhang ◽  
Yishu Yang
Keyword(s):  
Computed Tomography ◽  
Magnetic Resonance ◽  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Young Woman ◽  
Anticoagulant Therapy ◽  
Cerebral Artery ◽  
Acute Cerebral Infarction ◽  
Menstrual Phase ◽  
D Dimer

Abstract Background Acute cerebral infarction with adenomyosis in a young woman has been rarely reported. Case presentation We describe a 34-year-old young woman who presented headache and fever (38°C) for 4 days and left limb weakness for 1 day during her menstrual phase. Laboratory test data showed: Hemoglobin (HGB) (112g/L, normal: 120-150 g/L), Carcinoembryonic antigen 125 (CA125) (937.70U/ml, normal: 0-35 U/ml), D-Dimer (27.4mg/L, normal: 0-1.5mg/L). Magnetic resonance imaging (MRI) indicated acute cerebral infarction in right basal ganglia and subcortical region of right frontotemporal lobe. Further, brain computed tomography angiography (CTA) showed that the M1 segment of right middle cerebral artery was strictured and the distal branches of right middle cerebral artery were significantly less than those on the opposite side. No obvious abnormality was found in cranial magnetic resonance venogram (MRV). She had a 5-year history of adenomyosis. No tumors were found by whole body positron emission tomography-computed tomography (PET-CT). We treated this patient by using anti-infective therapy for 1 week and using anticoagulant therapy with low molecular weight heparin for 2 weeks. Subsequently, the anticoagulant therapy was discontinued and replaced by antiplatelet therapy with poliovir. We followed up this patient for 4 months, and no recurrence of cerebral infarction was found. Conclusions Acute cerebral infarction with adenomyosis may be related to elevated D-Dimer, elevated CA125, anemia, menstruation and fever. Our report suggests that acute cerebral infarction with adenomyosis can occur not only in middle-aged women but also in young women, and fever during menstrual phase in a woman with adenomyosis may be a factor leading to acute cerebral infarction.

scholarly journals Acute middle cerebral artery stroke in a patient with a patent middle cerebral artery

2019 ◽  
Vol 9 (3) ◽  
pp. 250-255 ◽  
Author(s):  
Jamie Cooke ◽  
Julian Maingard ◽  
Ronil V. Chandra ◽  
Lee-Anne Slater ◽  
Mark Brooks ◽  
...  
Keyword(s):  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Anatomical Variation ◽  
Clinical Importance ◽  
Signs And Symptoms ◽  
Endovascular Management ◽  
Accessory Middle Cerebral Artery ◽  
Excellent Functional Outcome ◽  
Middle Cerebral Artery Stroke

Purpose of reviewKnowledge of cerebrovascular anatomical variants is vital for clinicians working with patients presenting with signs and symptoms of cerebral infarction, particularly in the era of endovascular clot retrieval.Recent findingsWe provide an overview of a cerebrovascular anatomical variation and detail a patient presenting with cerebral infarction secondary to occlusion of their anomalous vessel who underwent successful endovascular clot retrieval with excellent functional outcome. We also include technical descriptions.SummaryGiven the clinical importance of the areas supplied by the accessory middle cerebral artery, knowledge of this vessel is not only important for diagnosis but also for neurosurgical or endovascular management of patients with this variant.

scholarly journals ME3277, a GPIIb/IIIa Antagonist Reduces Cerebral Infarction without Enhancing Intracranial Hemorrhage in Photothrombotic Occlusion of Rabbit Middle Cerebral Artery

2000 ◽  
Vol 20 (6) ◽  
pp. 988-997 ◽  
Author(s):  
Ken-Ichi Kawano ◽  
Kazuyuki Fujishima ◽  
Yasuhiko Ikeda ◽  
Kazunao Kondo ◽  
Kazuo Umemura
Keyword(s):  
Arachidonic Acid ◽  
Platelet Aggregation ◽  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Intracranial Hemorrhage ◽  
Cerebral Artery ◽  
Photochemical Reaction ◽  
Bleeding Time ◽  
Infarct Volume ◽  
Acute Cerebral Infarction

GPIIb/IIIa antagonists are expected to have a beneficial effect on acute cerebral infarction, however, the occurrence of intracranial hemorrhage has not been as widely investigated. A rabbit focal thrombotic occlusion model of the middle cerebral artery was established by creating a photochemical reaction between green light and Rose Bengal. Hemorrhagic transformation was common in the area of cerebral infarction. Using this model, the effect of a GPIIb/IIIa antagonist, ME3277 (low dose, (L); 0.15 mg/kg + 0.125 mg/kg·h, middle dose, (M); 0.3 mg/kg + 0.25 mg/kg·h and high dose, (H); 0.6 mg/kg + 0.5 mg/kg·h), aspirin (20 mg/kg) and sodium ozagrel (thromboxane A2 synthase inhibitor, 1 mg/kg + 2 mg/kg·h) were evaluated. Drugs were intravenously administrated 30 minutes after the photochemical reaction for 24 hours. Aspirin inhibited the ex vivo platelet aggregation induced by arachidonic acid and collagen but not by adenosine diphosphate (ADP), while sodium ozagrel only inhibited the arachidonic acid-induced aggregation. ME3277 dose-dependently inhibited the platelet aggregation induced by all the inducers (approximately 60% in L, 80% in M, and 90% in H). At 24 hours of middle cerebral artery (MCA) occlusion, infarct volume was significantly reduced by aspirin and each dose of ME3277. These agents improved neurologic deficits, with ME3277 being more potent than aspirin. Sodium ozagrel did not alter the infarct volume nor neurologic deficits. No drug was found to worsen hemorrhage volume despite increasing bleeding time (2–3 fold) in the skin. In this model, the occluded artery was spontaneously recanalized and re-thrombosed frequently. One mechanism by which antiplatelet agents reduced infarct volume was inhibition of rethrombosis of the MCA. These results suggest that treatment with a GPIIb/IIIa antagonist is a useful intervention for acute cerebral infarction prolonging dose bleeding time to 3 times the basal value.

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