Evaluation of the Relationship Between Lower Extremity Varicose Veins-Venous Insufficiency and Varicocele-Vulvar Varicose Veins in Our Population

2013 ◽  
Vol 22 (3) ◽  
pp. 297-302
Author(s):  
Seyhan YILMAZ ◽  
Eray AKSOY ◽  
Songül YAYLACI
Keyword(s):  
Lower Extremity ◽  
Venous Insufficiency ◽  
Varicose Veins ◽  
The Relationship

Systematic review and meta-analysis of endovascular and surgical revascularization for patients with chronic lower extremity venous insufficiency and varicose veins

2018 ◽  
Vol 196 ◽  
pp. 131-143 ◽  
Author(s):  
Sreekanth Vemulapalli ◽  
Kishan Parikh ◽  
Remy Coeytaux ◽  
Victor Hasselblad ◽  
Amanda McBroom ◽  
...  
Keyword(s):  
Systematic Review ◽  
Lower Extremity ◽  
Venous Insufficiency ◽  
Varicose Veins ◽  
Meta Analysis ◽  
Surgical Revascularization

scholarly journals Varicose Veins and Lower Extremity Venous Insufficiency

2018 ◽  
Vol 35 (01) ◽  
pp. 056-061 ◽  
Author(s):  
Eric DePopas ◽  
Matthew Brown
Keyword(s):  
Lower Extremity ◽  
Clinical Diagnosis ◽  
Saphenous Vein ◽  
Venous Insufficiency ◽  
Great Saphenous Vein ◽  
Varicose Veins ◽  
Ultrasonographic Diagnosis

AbstractLower extremity venous insufficiency and varicose veins are common conditions, affecting up to 25% of women. Herein, we review the pathophysiology of lower extremity venous insufficiency and varicose veins, the epidemiology of varicose veins, clinical diagnosis, and ultrasonographic diagnosis. We also discuss treatment rationale, algorithms, and techniques, with a focus on endovenous great saphenous vein ablation.

Prevalence and management of ovarian venous insufficiency in the presence of leg venous insufficiency

2007 ◽  
Vol 22 (1) ◽  
pp. 29-33 ◽  
Author(s):  
R Sutaria ◽  
A Subramanian ◽  
B Burns ◽  
H Hafez
Keyword(s):  
Lower Extremity ◽  
Lower Limb ◽  
Venous Insufficiency ◽  
Varicose Veins ◽  
Vena Cava ◽  
Vascular Surgeon ◽  
Venous Surgery ◽  
Magnetic Resonance Imaging Mri ◽  
The Right ◽  
Superficial Venous Insufficiency

Objective: The correlation between ovarian venous insufficiency and lower limb venous insufficiency remains poorly understood. Clinically, incompetent ovarian veins in association with lower extremity varicose veins are suspected when leg varicose veins are found in atypical distributions. Such distributions include upper lateral or posterior thigh, on the buttocks, crossing the inguinal ligament, and also in the vulval or perineal regions. The aim of this study was to determine the prevalence of ovarian venous insufficiency in those with clinically suspicious varicose veins, and to assess the effectiveness of ovarian venous embolization/ligation in treating this condition. Methods: Between June 2001 and December 2004, 424 female patients with lower limb superficial venous insufficiency were seen by a single vascular surgeon. These patients were clinically assessed, and those with atypical varicose veins were investigated with venous duplex examination and magnetic resonance imaging (MRI) venography. Patients with proven ovarian venous insufficiency were offered venography with a view to embolization or laparoscopic ligation. Results: A total of seven patients were clinically suspected of having ovarian venous insufficiency, of which three had recurrent varicose veins (42.9%). Of these, six were confirmed on MRI venography with the left side being more affected than the right; one of them had an occluded vena cava, three were treated by embolization, and two had laparoscopic ligation. Discussion: The prevalence of clinically detectable ovarian venous insufficiency in association with lower extremity varicose veins is in the region of 1.65%. Compared with the estimated prevalence of incidental ovarian venous insufficiency of 10–47%, this suggests that only a minority of incompetent ovarian veins will present with clinically detectable lower limb venous insufficiency. In our opinion, patients with signs suggestive of ovarian venous insufficiency in association with lower limb venous insufficiency should have their ovarian insufficiency controlled prior to embarking on limb venous surgery.

scholarly journals Demographic examination of lymphedema seen in the lower extremities

2021 ◽  
Vol 8 (6) ◽  
pp. 1682
Author(s):  
Ibrahim Demir ◽  
Dogan Yetut ◽  
Metin Onur Beyaz
Keyword(s):  
Lower Extremity ◽  
Venous Insufficiency ◽  
Blood Groups ◽  
Venous Disease ◽  
Lower Extremity Edema ◽  
Secondary Lymphedema ◽  
Patient Profile ◽  
The Mean ◽  
The Relationship ◽  
Perforating Vein

Background: We aimed to indicate the frequency of this disease according to blood groups, other disease types, age, weight and other demographic characteristics.Methods: We examined total of 236 patients who applied to our clinic because of lower extremity edema within 5 years. All patients had a diagnosis of lymphedema. Patients with a diagnosis of congenital lymphedema or a diagnosis secondary to cancer were excluded. Obesity, presence of venous disease, diabetes mellitus and hypertension which are considered as causes of secondary lymphedema were included in the study. Also the relationship between lymphedema patients and blood groups were evaluated.Results: 81% (n:193) of patients were women. The mean age of the patients was 50.71 (±10.28). All patients had diagnosis of lymphedema. Most of patients (n:189) had bilateral lower extremity edema. Body mass index was above 25 in 149 (63.1%) patients. Deep venous insufficiency accompanied in 75 (31.8%) patients. Perforator vein incompetance was observed with lymphedema in 96 (40.7%) patients. The number of diabetic and hypertensive patients was 64 (27.1%) and 67 (28.4%), respectively. Patients with B type blood group constituted the largest patient profile with a rate of 41.9% (n:99).Conclusions: In our study, demographic characteristics were not statistically corolated to lymphedema development, however, the rate of lymphedema in patients with perforating vein insufficiency was statistically significant (p<0.05, P=0.002). The most important point to be considered here is whether lymphedema plays a role in the development of additional pathology or do additional pathologies trigger the development of lymphedema?

The nature of lower extremity venous insufficiency in patients with primary varicose veins

1994 ◽  
Vol 8 (4) ◽  
pp. 464-471 ◽  
Author(s):  
Asterios N. Katsamouris ◽  
Demetrios G. Kardoulas ◽  
Nicholas Gourtsoyiannis
Keyword(s):  
Lower Extremity ◽  
Venous Insufficiency ◽  
Varicose Veins ◽  
Primary Varicose Veins

Chronic cerebrospinal venous insufficiency is not associated with chronic venous disorders: A case–control study

2014 ◽  
Vol 30 (10) ◽  
pp. 736-738
Author(s):  
Maurizio A Leone ◽  
Olga Raymkulova ◽  
Piergiorgio Lochner ◽  
Laura Bolamperti ◽  
Gianandrea Rivadossi ◽  
...  
Keyword(s):  
Family History ◽  
Venous Insufficiency ◽  
Ad Hoc ◽  
Varicose Veins ◽  
Color Doppler ◽  
Chronic Cerebrospinal Venous Insufficiency ◽  
History Of ◽  
The Relationship ◽  
Venous Disorders ◽  
Control Study

Objectives To evaluate the relationship between chronic cerebrospinal venous insufficiency (CCSVI) and the presence of a Chronic Venous Disorder (CVD). Method We included 55 subjects with CCSVI aged >18 years, and 186 controls without CCSVI. Each subject was evaluated with color Doppler sonography in accordance with Zamboni’s five criteria, examined by two neurologists and interviewed with an ad-hoc designed form. The neurologists and the sonographers were mutually blinded. CVD were classified according to CEAP. Results Mean age was 42 years (SD = 9) in cases and 43 years (10) in controls ( p = ns). The odds ratios in subjects CCSVI were 0.6 (0.2–2.2) for CEAP 1, 0.9 (0.2–4.5) for CEAP 2, and 1.0 (0.6–1.9) for family history of varicose veins. The prevalence of CVD and, family history of varicose veins, was similar between cases and controls for each Zamboni criterion. Conclusions We found no association of CCSVI with the presence of CVD or family history of varicose veins.

Invasive therapeutic options in truncal varicosity of the great saphenous vein

VASA ◽  
2006 ◽  
Vol 35 (3) ◽  
pp. 157-166 ◽  
Author(s):  
Hach-Wunderle ◽  
Hach
Keyword(s):  
Minimally Invasive ◽  
Saphenous Vein ◽  
Venous Insufficiency ◽  
Great Saphenous Vein ◽  
Varicose Veins ◽  
Venous Pressure ◽  
Stage Iii ◽  
High Ligation ◽  
Surgical Trauma ◽  
Disease Stages

It is known from current pathophysiology that disease stages I and II of truncal varicosity of the great saphenous vein do not cause changes in venous pressure on dynamic phlebodynamometry. This is possibly also the case for mild cases of the disease in stage III. In pronounced cases of stage III and all cases of stage IV, however, venous hypertension occurs which triggers the symptoms of secondary deep venous insufficiency and all the complications of chronic venous insufficiency. From these facts the therapeutic consequence is inferred that in stages I and II and perhaps also in very mild cases of stage III disease, it is enough "merely" to remove varicose veins without expecting there to be any other serious complications in the patient’s further life caused by the varicosity. Recurrence rates are not included in this analysis. In marked cases of disease stages III and IV of the great saphenous vein, however, secondary deep venous insufficiency is to be expected sooner or later. The classical operation with saphenofemoral high ligation ("crossectomy") and stripping strictly adheres to the recognized pathophysiologic principles. It also takes into account in the greatest detail aspects of minimally invasive surgery and esthetics. In the past few years, developments have been advanced to further minimize surgical trauma and to replace the stripping maneuver using occlusion of the trunk vein which is left in place. Obliteration of the vessel is subsequently performed via transmission of energy through an inserted catheter. This includes the techniques of radiofrequency ablation and endovenous laser treatment. High ligation is not performed as a matter of principle. In a similar way, sclerotherapy using microfoam is minimally invasive in character. All these procedures may be indicated for disease stages I and II, and with reservations also in mild forms of stage III disease. Perhaps high ligation previously constituted overtreatment in some cases. Targeted studies are still needed to prove whether secondary deep venous insufficiency can be avoided in advanced stages of varicose vein disease without high ligation and thus without exclusion of the whole recirculation circuit.

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