Online and Offline Diagnosis of Motor Power Cables Based on 1D CNN and Periodic Burst Signal Injection

We introduce a new approach for online and offline soft fault diagnosis in motor power cables, utilizing periodic burst injection and nonintrusive capacitive coupling. We focus on diagnosing soft faults because local cable modifications or soft faults that occur without any indication while the cable is still operational can eventually develop into hard faults; furthermore, advance diagnosis of soft faults is more beneficial than the later diagnosis of hard faults, with respect to preventing catastrophic production stoppages. Both online and offline diagnoses with on-site diagnostic ability are needed because the equipment in the automated lines operates for 24 h per day, except during scheduled maintenance. A 1D CNN model was utilized to learn high-level features. The advantages of the proposed method are that (1) it is suitable for wiring harness cables in automated factories, where the installed cables are extremely short; (2) it can be simply and identically applied for both online and offline diagnoses and to a variety of cable types; and (3) the diagnosis model can be directly established from the raw signal, without manual feature extraction and prior domain knowledge. Experiments conducted with various fault scenarios demonstrate that this method can be applied to practical cable faults.

Pacemaker Potentials for the Periodic Burst Discharge in the Heart Ganglion of a Stomatopod, Squilla oratoria

From somata of the pacemaker neurons in the Squilla heart ganglion, pacemaker potentials for the spontaneous periodic burst discharge are recorded with intracellular electrodes. The electrical activity is composed of slow potentials and superimposed spikes, and is divided into four types, which are: (a) "mammalian heart" type, (b) "slow generator" type, (c) "slow grower" type, and (d) "slow deficient" type. Since axons which are far from the somata do not produce slow potentials, the soma and dendrites must be where the slow potentials are generated. Hyperpolarization impedes generation of the slow potential, showing that it is an electrically excitable response. Membrane impedance increases on depolarization. Brief hyperpolarizing current can abolish the plateau but brief tetanic inhibitory fiber stimulation is more effective for the abolition. A single stimulus to the axon evokes the slow potential when the stimulus is applied some time after a previous burst. Repetitive stimuli to the axon are more effective in eliciting the slow potential, but the depolarization is not maintained on continuous stimulation. Synchronization of the slow potential among neurons is achieved by: (a) the electrotonic connections, with periodic change in resistance of the soma membrane, (b) active spread of the slow potential, and (c) synchronization through spikes.