In Vivo Kinematics of Functional Ankle Instability Patients and Lateral Ankle Sprain Copers During Stair Descent

2019 ◽  
Vol 37 (8) ◽  
pp. 1860-1867 ◽  
Author(s):  
Shengxuan Cao ◽  
Chen Wang ◽  
Xin Ma ◽  
Xu Wang ◽  
Jiazhang Huang ◽  
...  
Keyword(s):  
Ankle Sprain ◽  
Ankle Instability ◽  
Functional Ankle Instability ◽  
Lateral Ankle Sprain ◽  
Lateral Ankle ◽  
In Vivo Kinematics ◽  
Stair Descent

scholarly journals In Vivo Kinematics of the Tibiotalar Joint After Lateral Ankle Instability

2009 ◽  
Vol 37 (11) ◽  
pp. 2241-2248 ◽  
Author(s):  
Adam M. Caputo ◽  
Jun Y. Lee ◽  
Chuck E. Spritzer ◽  
Mark E. Easley ◽  
James K. DeOrio ◽  
...  
Keyword(s):  
Ankle Instability ◽  
Lateral Ankle ◽  
Tibiotalar Joint ◽  
In Vivo Kinematics ◽  
Lateral Ankle Instability

Predictors of chronic ankle instability after an index lateral ankle sprain: A systematic review

2014 ◽  
Vol 17 (6) ◽  
pp. 568-573 ◽  
Author(s):  
Fereshteh Pourkazemi ◽  
Claire E. Hiller ◽  
Jacqueline Raymond ◽  
Elizabeth J. Nightingale ◽  
Kathryn M. Refshauge
Keyword(s):  
Systematic Review ◽  
Ankle Sprain ◽  
Ankle Instability ◽  
Chronic Ankle Instability ◽  
Lateral Ankle Sprain ◽  
Lateral Ankle

In Vivo Kinematics of the Talocrural and Subtalar Joints With Functional Ankle Instability During Weight-Bearing Ankle Internal Rotation

2013 ◽  
Vol 6 (3) ◽  
pp. 178-184 ◽  
Author(s):  
Takumi Kobayashi ◽  
Yumi No ◽  
Kei Yoneta ◽  
Masashi Sadakiyo ◽  
Kazuyoshi Gamada
Keyword(s):  
Internal Rotation ◽  
Ankle Instability ◽  
Weight Bearing ◽  
Functional Ankle Instability ◽  
In Vivo Kinematics

scholarly journals Motor-Neuron Pool Excitability of the Lower Leg Muscles After Acute Lateral Ankle Sprain

2011 ◽  
Vol 46 (3) ◽  
pp. 263-269 ◽  
Author(s):  
Lindsey W. Klykken ◽  
Brian G. Pietrosimone ◽  
Kyung-Min Kim ◽  
Christopher D. Ingersoll ◽  
Jay Hertel
Keyword(s):  
Motor Neuron ◽  
Ankle Sprain ◽  
Tibialis Anterior ◽  
Ankle Instability ◽  
Ankle Sprains ◽  
Lateral Ankle Sprain ◽  
Lateral Ankle ◽  
Leg Muscles ◽  
Motor Neuron Pool ◽  
Neuron Pool

Context: Neuromuscular deficits in leg muscles that are associated with arthrogenic muscle inhibition have been reported in people with chronic ankle instability, yet whether these neuromuscular alterations are present in individuals with acute sprains is unknown. Objective: To compare the effect of acute lateral ankle sprain on the motor-neuron pool excitability (MNPE) of injured leg muscles with that of uninjured contralateral leg muscles and the leg muscles of healthy controls. Design: Case-control study. Setting: Laboratory. Patients or Other Participants: Ten individuals with acute ankle sprains (6 females, 4 males; age = 19.2 ± 3.8 years, height = 169.4 ± 8.5 cm, mass = 66.3 ±11.6 kg) and 10 healthy individuals (6 females, 4 males; age = 20.6 ± 4.0 years, height = 169.9 ± 10.6 cm, mass = 66.3 ± 10.2 kg) participated. Intervention(s): The independent variables were group (acute ankle sprain, healthy) and limb (injured, uninjured). Separate dependent t tests were used to determine differences in MNPE between legs. Main Outcome Measure(s): The MNPE of the soleus, fibularis longus, and tibialis anterior was measured by the maximal Hoffmann reflex (Hmax) and maximal muscle response (Mmax) and was then normalized using the Hmax:Mmax ratio. Results: The soleus MNPE in the ankle-sprain group was higher in the injured limb (Hmax:Mmax = 0.63; 95% confidence interval [CI], 0.46, 0.80) than in the uninjured limb (Hmax:Mmax = 0.47; 95% CI, 0.08, 0.93) (t6 = 3.62, P = .01). In the acute ankle-sprain group, tibialis anterior MNPE tended to be lower in the injured ankle (Hmax:Mmax = 0.06; 95% CI, 0.01, 0.10) than in the uninjured ankle (Hmax:Mmax = 0.22; 95% CI, 0.09, 0.35), but this finding was not different (t9 = −2.01, P = .07). No differences were detected between injured (0.22; 95% CI, 0.14, 0.29) and uninjured (0.25; 95% CI, 0.12, 0.38) ankles for the fibularis longus in the ankle-sprain group (t9 = −0.739, P = .48). We found no side-to-side differences in any muscle among the healthy group. Conclusions: Facilitated MNPE was present in the involved soleus muscle of patients with acute ankle sprains, but no differences were found in the fibularis longus or tibialis anterior muscles.

scholarly journals Systematic Review of Postural Control and Lateral Ankle Instability, Part I: Can Deficits Be Detected With Instrumented Testing?

2008 ◽  
Vol 43 (3) ◽  
pp. 293-304 ◽  
Author(s):  
Patrick O. McKeon ◽  
Jay Hertel
Keyword(s):  
Postural Control ◽  
Ankle Sprain ◽  
Ankle Instability ◽  
Chronic Ankle Instability ◽  
Effect Sizes ◽  
Ankle Sprains ◽  
Lateral Ankle Sprain ◽  
Lateral Ankle ◽  
Increased Risk ◽  
Clinical Questions

Abstract Objective: To answer the following clinical questions: (1) Is poor postural control associated with increased risk of a lateral ankle sprain? (2) Is postural control adversely affected after acute lateral ankle sprain? (3) Is postural control adversely affected in those with chronic ankle instability? Data Sources: PubMed and CINAHL entries from 1966 through October 2006 were searched using the terms ankle sprain, ankle instability, balance, chronic ankle instability, functional ankle instability, postural control, and postural sway. Study Selection: Only studies assessing postural control measures in participants on a stable force plate performing the modified Romberg test were included. To be included, a study had to address at least 1 of the 3 clinical questions stated above and provide adequate results for calculation of effect sizes or odds ratios where applicable. Data Extraction: We calculated odds ratios with 95% confidence intervals for studies assessing postural control as a risk factor for lateral ankle sprains. Effect sizes were estimated with the Cohen d and associated 95% confidence intervals for comparisons of postural control performance between healthy and injured groups, or healthy and injured limbs, respectively. Data Synthesis: Poor postural control is most likely associated with an increased risk of sustaining an acute ankle sprain. Postural control is impaired after acute lateral ankle sprain, with deficits identified in both the injured and uninjured sides compared with controls. Although chronic ankle instability has been purported to be associated with altered postural control, these impairments have not been detected consistently with the use of traditional instrumented measures. Conclusions: Instrumented postural control testing on stable force plates is better at identifying deficits that are associated with an increased risk of ankle sprain and that occur after acute ankle sprains than at detecting deficits related to chronic ankle instability.

scholarly journals Ultrasound measures of intrinsic foot muscle size and motor activation following lateral ankle sprain and chronic ankle instability. A cross-sectional study.

2020 ◽  
Author(s):  
John J Fraser ◽  
Rachel M Koldenhoven ◽  
Jay Hertel
Keyword(s):  
Young Adults ◽  
Ankle Sprain ◽  
Ankle Instability ◽  
Linear Regression Analysis ◽  
Chronic Ankle Instability ◽  
Cross Sectional ◽  
Lateral Ankle Sprain ◽  
Lateral Ankle ◽  
Intrinsic Foot Muscle ◽  
Injury Status

Objectives: To assess the effects of ankle injury status on intrinsic foot muscle (IFM) size at rest and during contraction in young adults with and without a history of lateral ankle sprain (LAS) and chronic ankle instability (CAI). Methods: Foot Posture Index (FPI), Foot Mobility Magnitude (FMM), and ultrasonographic cross-sectional area of the abductor hallucis (AbdH), flexor digitorum brevis (FDB), quadratus plantae (QP), and flexor hallucis brevis (FHB) were assessed at rest, and during non-resisted and resisted contraction in 22 healthy (13 females, BMI: 22.5±3.2, FPI: 4.2±3.9, FMM: 2.5±1.8), 17 LAS (9 females, BMI: 24.1±3.7, FPI: 2.5±3.4, FMM: 2.7±1.7), 21 Copers (13 females, BMI: 23.7±2.9, FPI: 3.6±4.1, FMM: 1.8±1.3), and 20 CAI (15 females, BMI: 25.1±4.5, FPI: 4.4±3.6., FMM: 2.3±1.1). Results: A multiple linear regression analysis assessing group, sex, BMI, FPI, and FMM on resting and contracted IFM size found sex (p<.001), BMI (p=.01), FPI (p=.05), and FMM*FPI interaction (p=.008) accounted for 19% of the variance (p=.002) in resting AbdH measures. Sex (p<.001) and BMI (p=.02) explained 24% of resting FDB measures (p<.001). Having a recent LAS (p=.03) and FMM (p=.02) predicted 11% of non-resisted QP contraction measures (p=.04), with sex (p<.001) explaining 13% of resting QP measures (p=.02). Both sex (p=.01) and FMM (p=.03) predicted 16% of resting FDB measures (p=.01). There were no other statistically significant findings. Conclusions: IFM resting ultrasound measures were primarily determined by sex, BMI, and foot phenotype and not injury status. The clinical utility of these IFM ultrasonographic assessments in young adults with LAS and CAI may be limited.

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