Phosphatidylcholine-Dependent Phospholipase C in Rat Liver Chromatin

1999 ◽  
Vol 265 (3) ◽  
pp. 640-643 ◽  
Author(s):  
Elisabetta Albi ◽  
Mariapia Viola Magni
Keyword(s):  
Phospholipase C ◽  
Rat Liver ◽  
Liver Chromatin

Purification and properties of a neutral protease from rat liver chromatin

Biochemistry ◽  
1974 ◽  
Vol 13 (25) ◽  
pp. 5128-5134 ◽  
Author(s):  
Ming Ta Chong ◽  
William T. Garrard ◽  
James Bonner
Keyword(s):  
Rat Liver ◽  
Neutral Protease ◽  
Purification And Properties ◽  
Liver Chromatin

Association of the thyroid hormone receptor with rat liver chromatin

Biochemistry ◽  
1981 ◽  
Vol 20 (24) ◽  
pp. 6781-6789 ◽  
Author(s):  
Donald B. Jump ◽  
Steven Seelig ◽  
Harold L. Schwartz ◽  
Jack H. Oppenheimer
Keyword(s):  
Thyroid Hormone ◽  
Rat Liver ◽  
Hormone Receptor ◽  
Thyroid Hormone Receptor ◽  
Liver Chromatin

Protein Synthesis in Rat Liver Chromatin and Chromatin Fractions

1966 ◽  
Vol 5 (6) ◽  
pp. 592-592 ◽  
Author(s):  
C. E. Sekeris ◽  
W. Schmid ◽  
D. Gallwitz ◽  
I. Lukacs
Keyword(s):  
Protein Synthesis ◽  
Rat Liver ◽  
Liver Chromatin

Early alterations in rat liver chromatin structure after a single dose of diethylnitrosamine

1983 ◽  
Vol 3 (2) ◽  
pp. 185-188
Author(s):  
Karl Letnansky ◽  
Hratschik R. Vardapetjan
Keyword(s):  
Single Dose ◽  
Rat Liver ◽  
Chromatin Structure ◽  
Chemical Modifications ◽  
Lower Melting ◽  
Melting Temperatures ◽  
Liver Chromatin ◽  
Rat Livers ◽  
Rapid Shift ◽  
Good Agreement

In the chromatin of 24-h regenerating rat livers, derivative melting profiles are characterized by a high proportion of transitions above 90°C. After the injection of diethylnitrosamine there is a rapid shift to lower melting temperatures. This is due to a rearrangement of the chromatin to higher amounts of nucleosomal components but possibly also a consequence of chemical modifications and conformational alterations of the DNA. In the nonregenerating liver essentially the same observations can be made, although reactions proceed significantly slower. These results are in good agreement with the observation that carcinogens are more active in tissues stimulated to rapid proliferation as compared to resting tissues.

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