Role of Cerebral Blood Flow Imaging in the Evaluation of Subarachnoid Hemorrhage and Intracranial Arteriovenous Malformations

1992 ◽  
pp. 84-93 ◽  
Author(s):  
H. H. Batjer
2003 ◽  
Vol 60 (6) ◽  
pp. 549-552 ◽  
Author(s):  
Yasunari Otawara ◽  
Kuniaki Ogasawara ◽  
Hirotsugu Yukawa ◽  
Nobuhiko Tomitsuka ◽  
Yoshitaka Kubo ◽  
...  

1988 ◽  
Vol 153 (3) ◽  
pp. 399-402 ◽  
Author(s):  
J. A. O. Besson ◽  
K. P. Ebmeier ◽  
H. G. Gemmell ◽  
P. F. Sharp ◽  
M. McFadyen ◽  
...  

A patient with spasms of the neck, occurring when he turned his head to the left, responded to treatment with benzhexol. Cerebral blood flow imaging demonstrated reduced uptake in the right corpus striatum compared with the left. The study demonstrates the presence of an abnormality in the basal ganglia; it also illustrates response to drug treatment. Cerebral blood flow imaging may be useful in the detection of basal ganglia abnormalities in spasmodic torticollis and assist in the selection of cases which should be targeted for treatment with drugs.


1982 ◽  
Vol 10 (3) ◽  
pp. 215
Author(s):  
Bernard H. Holzman ◽  
Richard G. Curless ◽  
George N. Sfakianakis ◽  
Cosimo Ajmone-Marsan ◽  
Jorge E. Montes

1990 ◽  
Vol 15 (9) ◽  
pp. 623-629 ◽  
Author(s):  
WEI-JEN SHIH ◽  
STEVEN T. DEKOSKY ◽  
JOHN J. COUPAL ◽  
GUY SIMMONS ◽  
CALIXTO PULMANO ◽  
...  

1997 ◽  
Vol 6 (6) ◽  
pp. 436
Author(s):  
T Yamashita ◽  
T Nagamitsu ◽  
O Hayashida ◽  
H Yasuda ◽  
S Kashiwagi

2006 ◽  
Vol 290 (1) ◽  
pp. R84-R89 ◽  
Author(s):  
Kazuhiko Takeuchi ◽  
Noriyuki Miyata ◽  
Marija Renic ◽  
David R. Harder ◽  
Richard J. Roman

Recent studies have indicated that 20-hydroxyeicosatetraenoic acid (20-HETE) contributes to the fall in cerebral blood flow (CBF) after subarachnoid hemorrhage (SAH), but the factors that stimulate the production of 20-HETE are unknown. This study examines the role of vasoactive factors released by clotting blood vs. the scavenging of nitric oxide (NO) by hemoglobin (Hb) in the fall in CBF after SAH. Intracisternal (icv) injection of blood produced a greater and more prolonged (120 vs. 30 min) decrease in CBF than that produced by a 4% solution of Hb. Pretreating rats with Nω-nitro-l-arginine methyl ester (l-NAME; 10 mg/kg iv) to block the synthesis of NO had no effect on the fall in CBF produced by an icv injection of blood. l-NAME enhanced rather than attenuated the fall in CBF produced by an icv injection of Hb. Blockade of the synthesis of 20-HETE with TS-011 (0.1 mg/kg iv) prevented the sustained fall in CBF produced by an icv injection of blood and the transient vasoconstrictor response to Hb. Hb (0.1%) reduced the diameter of the basilar artery (BA) of rats in vitro by 10 ± 2%. This response was reversed by TS-011 (100 nM). Pretreatment of vessels with l-NAME (300 μM) reduced the diameter of BA and blocked the subsequent vasoconstrictor response to the addition of Hb to the bath. TS-011 returned the diameter of vessels exposed to l-NAME and Hb to that of control. These results suggest that the fall in CBF after SAH is largely due to the release of vasoactive factors by clotting blood rather than the scavenging of NO by Hb and that 20-HETE contributes the vasoconstrictor response of cerebral vessels to both Hb and blood.


2011 ◽  
Vol 19 (17) ◽  
pp. 15777 ◽  
Author(s):  
Nengyun Feng ◽  
Jianjun Qiu ◽  
Pengcheng Li ◽  
Xiaoli Sun ◽  
Cui Yin ◽  
...  

2009 ◽  
Vol 34 (S1) ◽  
pp. 153-154
Author(s):  
M. Bellotti ◽  
S. Bertini ◽  
V. Raspaolo ◽  
C. Bulfoni ◽  
S. Dalzero ◽  
...  

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