Hypovitaminosis a from Intestinal Infestations

1975 ◽  
pp. 77-78
Author(s):  
I. S. Roy ◽  
E. Ahmed
Keyword(s):  
Author(s):  
J.C.S. Kim ◽  
M.G. Jourden ◽  
E.S. Carlisle

Chronic exposure to nitrogen dioxide in rodents has shown that injury reaches a maximum after 24 hours, and a reparative adaptive phase follows (1). Damage occurring in the terminal bronchioles and proximal portions of the alveolar ducts in rats has been extensively studied by both light and electron microscopy (1).The present study was undertaken to compare the response of lung tissue to intermittent exposure to 10 ppm of nitrogen dioxide gas for 4 hours per week, while the hamsters were on a vitamin A deficient diet. Ultrastructural observations made from lung tissues obtained from non-gas exposed, hypovitaminosis A animals and gas exposed animals fed a regular commercially prepared diet have been compared to elucidate the specific effect of vitamin A on nitrogen dioxide gas exposure. The interaction occurring between vitamin A and nitrogen dioxide gas has not previously been investigated.


1994 ◽  
Vol 124 (suppl_8) ◽  
pp. 1467S-1472S ◽  
Author(s):  
Barbara A. Underwood
Keyword(s):  

2020 ◽  
Vol 8 (1) ◽  
pp. e001038
Author(s):  
Georgina Hall ◽  
Craig Breheny ◽  
Zohra Khan ◽  
Tobias Schwarz ◽  
Richard J Mellanby

A seven-month-old male entire Bulldog presented for a three-week history of progressive lethargy, exercise intolerance, hindlimb paresis, muscle atrophy and hyperaesthesia. The dog had initially been raised on the bitch’s milk before maternal illness resulted in him transitioning onto milk replacer. He was weaned onto commercial puppy food until four months of age before being transitioned onto a homecooked diet. This diet comprised of raw meat, collected weekly from a local abattoir, homecooked cereals and vegetables. The patient was oxygen-dependent on presentation, requiring 40 per cent inspired oxygen concentration to maintain normoxaemia. Investigations revealed marked osteopenia with concurrent severe hypovitaminosis D, hypocalcaemia and hyperparathyroidism; hypovitaminosis A; hypothyroidism and concurrent severe pneumonia. Despite intensive medical care, nutritional interventions and escalating oxygen therapy, the dog was euthanased due to a deterioration in respiratory function. This case report highlights the severe clinical complications associated with the consumption of a nutritionally incomplete diet.


1970 ◽  
Vol 23 (2) ◽  
pp. 231-241 ◽  
Author(s):  
J. G. CHOPRA ◽  
J. KEVANY
Keyword(s):  

1988 ◽  
Vol 65 (5) ◽  
pp. 158-160 ◽  
Author(s):  
MJ CARRIGAN ◽  
JRW GLASTONBURY ◽  
JV EVERS
Keyword(s):  

2020 ◽  
Vol 25 (6) ◽  
pp. 194-198
Author(s):  
Vicki Baldrey

Respiratory disease is common in avian species. Rapid diagnosis and treatment are essential as birds tend to hide clinical signs of illness until disease is advanced and cases can deteriorate rapidly. Risk factors are frequently related to diet and environment, and include hypovitaminosis A and inhaled irritants. Birds presenting with respiratory signs should initially be stabilised with oxygen supplementation and supportive care before further diagnostics are pursued. Work up of these cases includes blood sampling for biochemistry and haematology, imaging including radiography and rigid endoscopy, and culture and polymerase chain reaction testing of respiratory secretions. Chlamydia and aspergillosis are common diagnoses encountered in practice. Treatment includes systemic antimicrobials, non-steroidal anti-inflammatory drugs and inhaled medications administered via nebulisation. Correction of underlying risk factors is also essential for a successful outcome. This article gives an overview of avian respiratory disease and describes a systematic approach to the investigation and treatment of these cases, achievable in general practice.


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