The role of the proton motive force and electron flow in light-driven solute transport in Rhodopseudomonas sphaeroides

1984 ◽  
Vol 50 (1) ◽  
pp. 88-88
Author(s):  
Marieke G. L. Elferink ◽  
Klaas J. Hellingwerf ◽  
Wil N. Konings
2005 ◽  
Vol 129 (3) ◽  
pp. 583-587 ◽  
Author(s):  
Marieke G. L. ELFERINK ◽  
Ilan FRIEDBERG ◽  
Klaas J. HELLINGWERF ◽  
Wil N. KONINGS

1985 ◽  
Vol 153 (1) ◽  
pp. 161-165 ◽  
Author(s):  
Marieke G. L. ELFERINK ◽  
Klaas J. HELLINGWERF ◽  
Wil N. KONINGS

2002 ◽  
Vol 43 (2) ◽  
pp. 335-354 ◽  
Author(s):  
K. H. Rohde ◽  
A. F. Gillaspy ◽  
M. D. Hatfield ◽  
L. A. Lewis ◽  
D. W. Dyer

Microbiology ◽  
2016 ◽  
Vol 85 (4) ◽  
pp. 506-508
Author(s):  
D. V. Eroshenko ◽  
T. V. Polyudova ◽  
V. P. Korobov

2009 ◽  
Vol 58 (10) ◽  
pp. 1309-1313 ◽  
Author(s):  
Mona Anoushiravani ◽  
Tahereh Falsafi ◽  
Vahid Niknam

The aim of this study was to evaluate the role of proton motive force (PMF)-dependent efflux in resistance of Helicobacter pylori to tetracycline (Tet). Tet MIC was determined by agar dilution in the presence and absence of carbonyl cyanide m-chlorophenylhydrazone (CCCP), an inhibitor of PMF. Antibiotic accumulation was conducted in the presence or absence of CCCP and the fluorescence of the accumulated antibiotic was measured by spectrofluorometry. In the presence of CCCP, antibiotic accumulation was increased by 2–17-fold in 17/20 Tetr isolates and by 3–10-fold in four of five high-level-resistant mutants. Correlation was observed between this increase and diminution of MIC with CCCP. PMF-dependent efflux mechanisms therefore appear to play an important role in the resistance of clinical isolates of H. pylori to Tet.


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