Effects of intravenous nitroglycerin combined with dopamine on intracranial pressure and cerebral arteriovenous oxygen difference in patients with acute subarachnoid haemorrhage

1995 ◽  
Vol 136 (3-4) ◽  
pp. 175-180 ◽  
Author(s):  
H. Iwanaga ◽  
K. Okuchi ◽  
N. Koshimae ◽  
K. Goda ◽  
M. Imanishi ◽  
...  
Keyword(s):  
Intracranial Pressure ◽  
Subarachnoid Haemorrhage ◽  
Intravenous Nitroglycerin ◽  
Oxygen Difference

Clinical evaluation of intravenous nitroglycerin for neurosurgery

1978 ◽  
Vol 48 (5) ◽  
pp. 704-711 ◽  
Author(s):  
Julian S. Chestnut ◽  
Maurice S. Albin ◽  
Evelyn Gonzalez-Abola ◽  
Philippa Newfield ◽  
Joseph C. Maroon
Keyword(s):  
Blood Pressure ◽  
Intracranial Pressure ◽  
Intravenous Nitroglycerin ◽  
Control Blood Pressure ◽  
Content Type ◽  
Cerebrovascular Autoregulation ◽  
Ideal Agent ◽  
Neurosurgical Patient ◽  
Moment Control ◽  
The Ideal

✓ Moment-to-moment control of blood pressure is important in the management of the neurosurgical patient. The ideal agent to control blood pressure or induce hypotension should be non-toxic, maintain cerebrovascular autoregulation, and not alter cardiac output or change intracranial pressure. Intravenous nitroglycerin has been used to control blood pressure in 54 neurosurgical cases. This agent produces a rapid, controllable, but not precipitous fall in blood pressure without rebound, is non-toxic, may not alter cerebrovascular autoregulation, and does not raise intracranial pressure. Our clinical experience with intravenous nitroglycerin indicates that it has an important role as a hypotensive agent for the neurosurgical patient.

Neurologic Emergencies (DRAFT)

2018 ◽  
Author(s):  
Tamara Kaplan ◽  
Tracey Milligan
Keyword(s):  
Status Epilepticus ◽  
Intracranial Pressure ◽  
Subarachnoid Haemorrhage ◽  
Vascular Malformation ◽  
Reversible Cerebral Vasoconstriction Syndrome ◽  
Increased Intracranial Pressure ◽  
Cerebral Vasoconstriction ◽  
Neurologic Emergencies

The video in this chapter discusses neurologic emergencies, including the symptoms of increased intracranial pressure leading to herniation, subarachnoid haemorrhage (can be due to aneurysm, vascular malformation, or reversible cerebral vasoconstriction syndrome, and definition and management of status epilepticus.

Neurology

2013 ◽  
Author(s):  
Martin Beed ◽  
Richard Sherman ◽  
Ravi Mahajan
Keyword(s):  
Traumatic Brain Injury ◽  
Intensive Care ◽  
Status Epilepticus ◽  
Brain Injury ◽  
Intracranial Pressure ◽  
Subarachnoid Haemorrhage ◽  
Metabolic Disorders ◽  
Raised Intracranial Pressure ◽  
Thromboembolic Stroke ◽  
Neurological State

Decreased consciousnessSeizures and status epilepticusStroke/thromboembolic strokeIntracerebral haemorrhageSubarachnoid haemorrhageTraumatic brain injuryRaised intracranial pressureMeningitis and encephalitisAgitation/confusion/aggressionAlcohol withdrawalNeuromuscular weakness and paralysisGuillain–Barré syndromeMyasthenia gravis↓consciousness occurs in many diseases requiring admission to intensive care, and is often a cause for admission in its own right. Changes in neurological state may be related to intracranial pathology, or may occur in response to respiratory, circulatory, or metabolic disorders....

Effects of intravenous nitroglycerin on the intracranial pressure and volume pressure response

1983 ◽  
Vol 58 (4) ◽  
pp. 562-565 ◽  
Author(s):  
Ghaleb A. Ghani ◽  
Yung Fong Sung ◽  
Michael S. Weinstein ◽  
George T. Tindall ◽  
Alan S. Fleischer
Keyword(s):  
Blood Pressure ◽  
Intracranial Pressure ◽  
Fluid Pressure ◽  
Pressure Response ◽  
Mean Blood Pressure ◽  
Intravenous Nitroglycerin ◽  
Content Type ◽  
Ventricular Fluid Pressure ◽  
The Mean ◽  
Fine Print

✓ Ventricular fluid pressure (VFP) and volume-pressure response were measured during nitroglycerin (NTG) infusion in nine patients anesthetized with N2O and fentanyl. The patients' ventilation was controlled, and PaCO2 was kept at 32 ± 4 mm Hg. When an infusion of 0.01% NTG was given intravenously to decrease the mean blood pressure to 95.1%, 84.7%, and 78.2% of control, the VFP increased from control levels of 9.94 ± 2.14 mm Hg to 12.89 ± 2.25, 15.6 ± 2.85, and 14.43 ± 3.45 mm Hg, respectively. The volume-pressure response showed a significant increase when blood pressure decreased to 84.7% and 78.2% of control. These results suggest that intravenous NTG caused an increase in the intracranial pressure and a decrease in the intracranial compliance.

Intracranial pressure and pressure volume relation in patients with subarachnoid haemorrhage (SAH)

1978 ◽  
Vol 44 (1-2) ◽  
pp. 69-80 ◽  
Author(s):  
U. Hase ◽  
H. -J. Reulen ◽  
A. Fenske ◽  
K. Sch�rmann
Keyword(s):  
Intracranial Pressure ◽  
Subarachnoid Haemorrhage ◽  
Volume Relation

Haemodynamic, intracranial pressure and electrocardiographic changes following subarachnoid haemorrhage in rats

1992 ◽  
Vol 115 (3-4) ◽  
pp. 118-122 ◽  
Author(s):  
J. Verlooy ◽  
J. Van Reempts ◽  
M. Haseldonckx ◽  
M. Borgers ◽  
P. Selosse
Keyword(s):  
Intracranial Pressure ◽  
Subarachnoid Haemorrhage ◽  
Electrocardiographic Changes

Drugs for rapidly lowering blood pressure

1979 ◽  
Vol 17 (16) ◽  
pp. 61-63
Keyword(s):  
Myocardial Infarction ◽  
Blood Pressure ◽  
Intracranial Pressure ◽  
Subarachnoid Haemorrhage ◽  
Severe Hypertension ◽  
Dissecting Aneurysm ◽  
Hypertensive Encephalopathy ◽  
Acute Pulmonary Oedema ◽  
Raised Intracranial Pressure ◽  
Old People

In most patients with severe hypertension it is safest to lower the blood pressure gradually over an hour or so using oral medication, e. g. with hydralazine + propranolol. The lowering of blood pressure within minutes is necessary only when life is threatened by hypertensive encephalopathy, dissecting aneurysm of the aorta, continuing cerebral or subarachnoid haemorrhage, acute pulmonary oedema secondary to severe hypertension, or eclampsia. If hypertension is due to raised intracranial pressure it should not be treated unless the intracranial pressure has first been reduced. Rapid or excessive reduction of blood pressure can precipitate cerebral or myocardial infarction, particularly in old people, and in patients with long-standing hypertension.

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