Comment on “Early treatment with activated protein C for meningococcal septic shock: case report and literature review” by Hasin et al.

2005 ◽  
Vol 31 (12) ◽  
pp. 1729-1729
Author(s):  
Gareth L. Thomas ◽  
Peter Clark
2005 ◽  
Vol 31 (7) ◽  
pp. 1002-1003 ◽  
Author(s):  
Tal Hasin ◽  
David Leibowitz ◽  
David Rot ◽  
Yoram Weiss ◽  
Tova Chajek-Shaul ◽  
...  

2005 ◽  
Vol 193 (3) ◽  
pp. 864-865 ◽  
Author(s):  
László Medve ◽  
István Kis Csitári ◽  
Zsolt Molnár ◽  
Ádám László

2021 ◽  
Vol 7 (1) ◽  
Author(s):  
Min Li ◽  
Gang Zhu ◽  
Hao Guo ◽  
Shun Nan Ge ◽  
Guo Dong Gao ◽  
...  

AbstractBackgroundCerebral fat embolism (CFE) syndrome at high altitude was rare complicated with paroxysmal sympathetic hyperactivity (PSH) syndrome and septic shock. It is a challenge to differential diagnosis and treatment at high altitude.Case presentationThis case presents a CFE with PSH and septic shock of a 23-year-old man occurred at high altitude of 3800 m above sea level, transferred by airplane successfully and cured in the department of neurosurgery, Xi’an Tangdu Hospital.ConclusionsIt is key that CFE with PSH can be rapid diagnosed and treatment bundles of septic shock should be initiated as soon as possible. Early neurological rehabilitation played an important role for good outcome.


Shock ◽  
2001 ◽  
Vol 15 (Supplement) ◽  
pp. 55-56
Author(s):  
J A Hazelzet ◽  
E D de Kleijn ◽  
R de Groot ◽  
D. J. Stearns-Kurosawa ◽  
Shinichiro Kurosawa ◽  
...  

1996 ◽  
Vol 1 (4) ◽  
pp. 275-280 ◽  
Author(s):  
Howard Daniel Hoerl ◽  
Aldo Tabares ◽  
Kandice Kottke-Marchant

Activated protein C resistance (APCR) is a recently discovered, medically important cause of venous thrombosis. More than 95% of cases are due to factor V Leiden (FVL), a mutated form of factor V that is resistant to degradation by activated protein C. The prevalence of this disorder, which is inherited in an autosomal dominant fashion, is approximately 5% among asymptomatic people of European heritage. In addition, 20 to 60% of patient cohorts with previous thrombosis demonstrate APCR, making it the most common known genetic cause of abnormal thrombophilia. Current laboratory techniques available for diagnosis include functional assays, such as the APC ratio, as well as DNA-based tests that detect the specific genetic anomaly responsible for FVL. A case report is presented, along with a review of the literature highlighting epidemiology, pathogenesis, clinical features and methods for laboratory diagnosis.


Sign in / Sign up

Export Citation Format

Share Document