Three-Dimensional Computed Tomographic Analysis of a Rare Left Coronary Artery to Left Ventricle Fistula

2012 ◽  
Vol 34 (3) ◽  
pp. 774-776 ◽  
Author(s):  
Kelvin Lee ◽  
Gary H. Danton ◽  
Richard E. Kardon
Keyword(s):  
Coronary Artery ◽  
Left Ventricle ◽  
Left Coronary Artery ◽  
Three Dimensional ◽  
Computed Tomographic ◽  
Tomographic Analysis

Anatomical & Imaging Measures of the Left Ventricular Apical Thinning

2013 ◽  
Vol 749 ◽  
pp. 144-148
Author(s):  
Ping Zhang ◽  
Yao Hui Zhu ◽  
Chee Mun Lum ◽  
Shao Yin Duan
Keyword(s):  
Coronary Artery ◽  
Left Ventricle ◽  
Volume Rendering ◽  
Left Coronary Artery ◽  
Heart Diseases ◽  
Three Dimensional ◽  
Left Ventricular ◽  
Multiplanar Reconstruction ◽  
Imaging Data ◽  
Anatomical Imaging

Doing the heart three-dimensional CT imaging (3DCT), the writers found 3DCT can clearly show the left ventricular apical thinning (LVAT). Purpose: To observe the shape of LVAT and measure related parameters in the end-systolic and end-diastolic phases. Methods: 12 cadaveric heart specimens were observed, and the thickness of LVAT was measured, as well as the thickest myocardium of left ventricle (TMLV). There are 69 cases imaging data of the end-systolic and end-diastolic phases without heart diseases from PACS in our hospital, with multiplanar reconstruction (MPR), Volume rendering (VR), the LVAT was clearly shown and measured. Measuring parameters include the thickness of LVAT, TMLV and the distance between the LVAT and the anterior descending branch of left coronary artery (DBLCA). Statistical comparisons were made. Results: In all cadaveric heart specimens of 12 cases were found the LVAT, the thickness of LVAT, TMLV was 1.74 mm ± 0.32 mm, 13.07 mm ± 1.48 mm. 3DCT clearly showed the LVAT in the 69 cases, whose thickness was 1.17 mm ± 0.43 mm in the diastole phase and 1.19 mm ± 0.48 mm in the systole phase. The thickness of TMLV was 12.02 mm ± 1.66mm, and the distance between the LVAT and DBLCA was 13.70mm ± 3.78 mm in the diastole phase. There were not significant differences in the LVAT thickness between systole and diastole phases (t = 0.366, p > 0.5), but there are significant differences in measuring the thickness of myocardium between the anatomy and 3DCT (t = 2.210, 0.01< P<0.05). Conclusion: The LVAT can be clearly shown by anatomy and 3DCT, and its thickness does not change in the end-systolic and end-diastolic phases.

scholarly journals A RARE CASE OF LARGE DIFFUSE BILATERAL RIGHT AND LEFT CORONARY ARTERY FISTULAS TERMINATING IN THE LEFT VENTRICLE

2021 ◽  
Vol 77 (18) ◽  
pp. 2434
Author(s):  
Iyad Farouji ◽  
Omar Alradaideh ◽  
Hossam Abed ◽  
Zaid Amin ◽  
Dilesha Kumanayaka ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Left Ventricle ◽  
Rare Case ◽  
Left Coronary Artery ◽  
Coronary Artery Fistulas

Right and left coronary artery to left ventricle fistula detected by color Doppler flow mapping

1987 ◽  
Vol 114 (4) ◽  
pp. 890-894 ◽  
Author(s):  
Toshio Nishikimi ◽  
Hisao Oku ◽  
Kazuyoshi Hirota ◽  
Kayoko Murai ◽  
Takahiko Kawarabayashi ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Left Ventricle ◽  
Left Coronary Artery ◽  
Color Doppler ◽  
Flow Mapping ◽  
Doppler Flow ◽  
Color Doppler Flow

Algorithm for diagnosis and typing of ALCAPA syndrome

2021 ◽  
Vol 3 (2) ◽  
pp. 01-07
Author(s):  
Mariela Céspedes Almira ◽  
Adel Eladio González Morejón ◽  
Giselle Serrano Ricardo ◽  
Tania Rosa González Rodríguez ◽  
Judith Escobar Bermúdez
Keyword(s):  
Coronary Artery ◽  
Left Ventricle ◽  
Left Coronary Artery ◽  
Cross Sectional Study ◽  
Left Ventricular ◽  
Volume Index ◽  
Cross Sectional ◽  
End Diastolic Volume ◽  
Congenital Origin ◽  
Diagnostic Modalities

ALCAPA syndrome was characterized by anomalous origin of left coronary artery from pulmonary artery. Its clinical presentation is varied and although it is an anomaly of congenital origin, it is not exclusive to pediatric ages. Its epidemiological documentation is difficult. We aimed to make the non-invasive diagnosis of the ALCAPA syndrome and its variants. An observational, prospective and cross-sectional study was conducted with 31 patients with a positive echocardiographic diagnosis of ALCAPA syndrome at Pediatric Cardio Center “William Soler” from 2005 to 2018. The variables with significance for diagnosis were the echocardiographic visualization of the anomalous connection and the reversed flow in the left coronary artery. The variables with significance for typing were age at diagnosis, ischemia in the electrocardiogram, echocardiographic visualization of left ventricle papillary muscles fibrosis, presence of severe mitral regurgitation, left ventricle spheroidal remodeling, left ventricle ejection fraction, left ventricular end-diastolic volume index, and left ventricular end-diastolic diameter index. An algorithm integrated by various diagnostic modalities associated with echocardiography as a tool for the detection of ALCAPA was developed. The documentation of the diagnostic and classificatory aspects of the syndrome is possible by detecting echocardiographic elements in conjunction with electrocardiographic and radiological aspects.

scholarly journals Heart anatomy of Giraffa camelopardalis rothschildi: a case report

2008 ◽  
Vol 53 (No. 3) ◽  
pp. 165-168 ◽  
Author(s):  
W. Perez ◽  
M. Lima ◽  
G. Pedrana ◽  
F. Cirillo
Keyword(s):  
Case Report ◽  
Coronary Artery ◽  
Left Ventricle ◽  
Left Coronary Artery ◽  
Left Ventricular ◽  
Left Auricle ◽  
Papillary Muscles ◽  
Atrioventricular Valve ◽  
Heart Anatomy ◽  
The Right

In the present study the most outstanding anatomical findings of the heart of a giraffe are described. Two papillary muscles were found in the right ventricle, namely magnus and subarterial. There were no papillary parvi muscles. The supraventricular crest gave insertion to various tendinous chords. These chords fixed the angular cusp of the right atrioventricular valve. The pectinate muscles were better developed in the left auricle than in the right one. Within the left ventricle two big papillary muscles were found as well as a notorious septomarginal trabecula. The left coronary artery irrigated the majority of the heart’s territory. It gave origin to the interventricular paraconal branch and to the circumflex branch. The latter gave off the branch of the left ventricular border and the interventricular subsinosal branch.

Patient-specific three-dimensional simulation of LDL accumulation in a human left coronary artery in its healthy and atherosclerotic states

2009 ◽  
Vol 296 (6) ◽  
pp. H1969-H1982 ◽  
Author(s):  
Ufuk Olgac ◽  
Dimos Poulikakos ◽  
Stefan C. Saur ◽  
Hatem Alkadhi ◽  
Vartan Kurtcuoglu
Keyword(s):  
Shear Stress ◽  
Coronary Artery ◽  
Left Coronary Artery ◽  
Three Dimensional ◽  
Arterial Blood Flow ◽  
Patient Specific ◽  
Computed Tomography Images ◽  
Arterial Blood ◽  
Diseased State ◽  
Stress Dependent

We calculate low-density lipoprotein (LDL) transport from blood into arterial walls in a three-dimensional, patient-specific model of a human left coronary artery. The in vivo anatomy data are obtained from computed tomography images of a patient with coronary artery disease. Models of the artery anatomy in its healthy and diseased states are derived after segmentation of the vessel lumen, with and without the detected plaque, respectively. Spatial shear stress distribution at the endothelium is determined through the reconstruction of the arterial blood flow field using computational fluid dynamics. The arterial endothelium is represented by a shear stress-dependent, three-pore model, taking into account blood plasma and LDL passage through normal junctions, leaky junctions, and the vesicular pathway. Intraluminal pressures of 70 and 120 mmHg are employed as the normal and hypertensive operating pressures, respectively. By applying our model to both the healthy and diseased states, we show that the location of the plaque in the diseased state corresponds to one of the two sites with predicted high-LDL concentration in the healthy state. We further show that, in the diseased state, the site with high-LDL concentration has shifted distal to the plaque, which is in agreement with the clinical observation that plaques generally grow in the downstream direction. We also demonstrate that hypertension leads to increased number of regions with high-LDL concentration, elucidating one of the ways in which hypertension may promote atherosclerosis.

Myocardial infarction in closed-chest dogs: a simplified method for production

1975 ◽  
Vol 39 (5) ◽  
pp. 831-833 ◽  
Author(s):  
H. A. Lappin ◽  
E. H. Botvinick ◽  
W. W. Parmley ◽  
J. V. Tyberg
Keyword(s):  
Myocardial Infarction ◽  
Coronary Artery ◽  
Left Ventricle ◽  
Left Coronary Artery ◽  
Sodium Pentobarbital ◽  
Simple Method ◽  
Closed Chest ◽  
Simplified Method ◽  
Coaxial Catheter

Due to our need, we sought a simple method to reliably create myocardial infarction in the closed-chest dog. Previous techniques were dangerous, time consuming, unreliable, and costly. Here we described a new coaxial catheter method by which occluded catheter plugs are embolized selectively to branches of the left coronary artery in closed chest dogs anesthetized with sodium pentobarbital (10 mg/lb). Infarcts varying in size from 3 to 27 g, 2–27% of the left ventricle, were reliably created in dogs weighing 26–70 lb. Complications were rare with only a single fatality in the last 15 procedures. The method proved safe, simple, quick, versatile, reproducible, and inexpensive.

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