Therapeutic and prophylactic effects of isometamidium chloride (Samorin) against the hemoflagellate Cryptobia salmositica in chinook salmon ( Oncorhynchus tshawytscha ) and the effects of the drug on uninfected rainbow trout ( O. mykiss )

2001 ◽  
Vol 87 (1) ◽  
pp. 18-26 ◽  
Author(s):  
B. F. Ardelli ◽  
P. T. K. Woo
1986 ◽  
Vol 43 (6) ◽  
pp. 1149-1155 ◽  
Author(s):  
J. W. Hilton ◽  
S. J. Slinger

The digestibility study indicated that dry matter digestibility of canola meal (CM) was low (56.2–60.4%), probably due to the high fiber content of this product and its relatively high level of complex carbohydrates. Energy and protein digestibility of CM were 72.4 and 83.2%, respectively, and the essential amino acid digestibilities were similar to those of protein in the CM. CM cannot successfully replace either soybean meal or fish meal in practical-type diets for young rainbow trout (Salmo gairdneri) without sacrificing growth. Supplementation with additional arginine, lysine and zinc, or energy to CM-based diets did not overcome the growth depressing effects. CM supplementation to the trout diet affected the thyroid gland of the fish, producing a diffuse, microfollicular hyperplasia that increased in severity with increasing CM supplementation. Young rainbow trout appear to be more sensitive to dietary glucosinolates than chinook salmon (Oncorhynchus tshawytscha), and total glucosinolate levels of 158 μg/g or more are deleterious to these fish. We conclude that CM should not, at this time, be included as a feed ingredient in commercial trout diets for young rainbow trout.


2007 ◽  
Vol 64 (5) ◽  
pp. 813-818 ◽  
Author(s):  
Anthony L Fritts ◽  
Jennifer L Scott ◽  
Todd N Pearsons

We tested whether one generation of state-of-the-art hatchery culture influenced the vulnerability of Chinook salmon (Oncorhynchus tshawytscha) fry to predators. Size-matched hatchery and wild origin spring Chinook salmon fry were exposed to rainbow trout (Oncorhynchus mykiss) and torrent sculpin (Cottus rhotheus) predators in 10.8 m3 net pens. The hatchery origin fry were the offspring of first generation hatchery-reared broodstock, and the wild origin fry had no history of hatchery culture; both originated from the same stock. Wild origin fry were found to have a 2.2% (p = 0.016) survival advantage over hatchery origin fry during 2 years of predation challenges. The most important findings of this study are (i) domestication can affect the susceptibility to predators after only one generation of state-of-the-art hatchery culture practices, and (ii) the domestication effect was very small.


2020 ◽  
Vol 43 (7) ◽  
pp. 719-728 ◽  
Author(s):  
Maureen K. Purcell ◽  
Rachel L. Powers ◽  
Torunn Taksdal ◽  
Doug McKenney ◽  
Carla M. Conway ◽  
...  

<EM>ABSTRACT. </EM>The potential for <em>Myxobolus cerebralis</em>, the cause of salmonid whirling disease, to affect resident populations of spring chinook salmon <em>Oncorhynchus tshawytscha </em>in the Lostine River, Oregon, was investigated in this study. Spring chinook salmon and rainbow trout <em>O. mykiss </em>fry were held in the Lostine River for 14 d in late March 1999, when resident chinook salmon alevins naturally emerge. After exposure, fry were held in pathogen-free water in the laboratory. The prevalence of infection at 5 months postexposure, as determined by PCR, was equivalent in both species (37.5% and 41%, respectively). Only rainbow trout developed cranial lesions (average lesion severity 0.4 on a 5-point scale; 4 of 10 fish examined were positive), and no spores were detected in homogenates of cartilage from fish of either species. Comparison of data on chinook salmon spawning sites (1996–2000) with known distribution of <em>M. cerebralis </em>in the Lostine River demonstrated that the majority of chinook salmon spawn in the middle section of the river, where levels of <em>M. cerebralis </em>exposure were reduced. Results of this study indicate that juvenile chinook salmon may become infected with <em>M. cerebralis</em>, when naturally exposed to the parasite, but suggest that the timing and location of their emergence may mitigate the negative impacts of <em>M. cerebralis </em>infection in this river.


1983 ◽  
Vol 40 (1) ◽  
pp. 101-102 ◽  
Author(s):  
G. L. Bullock ◽  
G. Maestrone ◽  
C. Starliper ◽  
B. Schill

Effective control of enteric redmouth disease was obtained during artificial infection of rainbow trout (Salmo gairdneri) and in natural outbreaks in rainbow trout and chinook salmon (Oncorhynchus tshawytscha) with a potentiated sulfonamide, Ro5-O037, a combination of five parts sulfadimethoxine and one part ormetoprim. Treatment was given at the rate of 50 mg/kg of fish for 5 d.Key words: potentiated sulfonamide, enteric redmouth, therapy, salmonids


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