Headache attributed to intracranial pressure alterations: applicability of the International Classification of Headache Disorders ICHD-3 beta version versus ICHD-2

2015 ◽  
Vol 36 (S1) ◽  
pp. 137-139 ◽  
Author(s):  
M. Curone ◽  
C. Peccarisi ◽  
G. Bussone
Cephalalgia ◽  
2017 ◽  
Vol 38 (10) ◽  
pp. 1696-1700 ◽  
Author(s):  
Evan Mullen ◽  
Mark Green ◽  
Eliza Hersh ◽  
Alfred-Marc Iloreta ◽  
Joshua Bederson ◽  
...  

Introduction The term Tolosa-Hunt Syndrome was first used more than half a century ago to describe painful ophthalmoplegia accompanied by cranial nerve palsies. In the decades since, its diagnostic criteria have evolved considerably. The beta version of the 3rd Edition of the International Classification of Headache Disorders narrows these criteria to require the demonstration of granulomatous inflammation on MRI or biopsy. We believe this may introduce challenges to accurate diagnosis. Discussion Requiring the demonstration of granulomatous inflammation for a diagnosis of Tolosa-Hunt Syndrome may introduce the potential for false negative and false positive diagnoses. Although the disorder presents secondary to granulomatous inflammation, MRI technology may not be able to identify it reliably, and biopsy is not always indicated for its symptomatology. Additionally, several cases have been reported of Tolosa-Hunt Syndrome diagnosed with MRI-confirmed granulomatous inflammation that later prove to be attributable to other pathologies. The emphasis on neuroimaging may therefore exclude some true Tolosa-Hunt Syndrome cases and include others resulting from other latent pathologies that are not visible on MRI. Conclusion We wish to offer several potential modifications to the International Classification of Headache Disorders guidelines for Tolosa-Hunt Syndrome, including making the demonstration of granulomatous inflammation on MRI or biopsy non-mandatory and lengthening patient follow-up to two years for cases in which MRI is unrevealing.


2016 ◽  
Vol 31 (1) ◽  
pp. 106 ◽  
Author(s):  
Byung-Kun Kim ◽  
Soo-Jin Cho ◽  
Byung-Su Kim ◽  
Jong-Hee Sohn ◽  
Soo-Kyoung Kim ◽  
...  

2014 ◽  
pp. 14-20
Author(s):  
Manuella Moraes Monteiro Barbosa Barros ◽  
Angélica da Silva Tonório ◽  
Thaís Ferreira Lopes Diniz Maia ◽  
Camila Carolinne Silva de Almeida ◽  
Daniella Araújo de Oliveira

Introdução: As cefaleias constituem a sintomatologia neurológica mais comum em todo o mundo; a presença desta alteração pode promover diversas consequências na biomecânica dos músculos cervicais, que podem limitar a mobilidade cervical e causar prejuízos aos pacientes que sofrem com cefaleia. Objetivo: Avaliar a relação entre a presença de cefaleia primária e a restrição na amplitude de movimento cervical. Métodos: Foi realizado um estudo piloto com 33 indivíduos (27 mulheres) com idade entre 20 e 38 anos (26 ± 5 anos). Para avaliar a mobilidade cervical ativa foi utilizado o goniômetro universal. O grau de disfunção cervical foiavaliado pelo questionário de Índice de Disfunção relacionado ao Pescoço. Para classificar a cefaleia primária foram utilizados os critérios estabelecidos pela International Classification of Headache Disorders (ICHD-III beta version, 2013). Resultados: Não houve diferenças estatísticas entre os grupos em relação ao gênero e última crise de cefaleia. A mobilidade cervical apresentou diferenças entre os grupos com cefaleia e saudáveis, mas essas não foram estatisticamente significantes. Também houve diferenças entre os grupos cefaleia e saudáveis em relação à classificação do Índice de Disfunção relacionado ao Pescoço, sem diferença significante (p<0,05). Conclusão: O estudo não demonstrou diferença entre a mobilidade cervical em pacientes com cefaleia primária, quando comparados a indivíduos saudáveis.


2018 ◽  
Vol 37 (01/02) ◽  
pp. 38-42
Author(s):  
K. Henkel

ZusammenfassungEs besteht eine hohe Komorbidität zwischen primären Kopfschmerzen und psychischen Erkrankungen. Eine gegenseitige Verstärkung und gemeinsame ätiologische Faktoren werden vermutet und wurden zum Teil nachgewiesen, so zum Beispiel bei Migräne und Depressionen. Eine nosologische Einteilung als sekundärer “Kopfschmerz zurückzuführen auf eine psychiatrische Störung“ verlangt eine hinreichende Evidenz für eine Auslösung oder wesentliche Verstärkung des Kopfschmerzes durch die psychische Erkrankung. Dieser Nachweis kann nur in Einzelfällen erfolgen. Größere systematische Untersuchungen fehlen. Die International Classification of Headache Disorders der International Headache Society erkennt auch in ihrer dritten Auflage (Beta-Version) nur die Somatisierungsstörung und die psychotische Störung als mögliche psychische Erkrankungen für die Auslösung sekundärer Kopfschmerzen an. Im Anhang der Klassifikation finden sich weitere psychische Erkrankungen, die möglicherweise sekundäre Kopfschmerzen auslösen können. Weitere prospektive und Längsschnittstudien sind nötig, um diese Zusammenhänge künftig besser beurteilen zu können.


Cephalalgia ◽  
2016 ◽  
Vol 37 (1) ◽  
pp. 20-28 ◽  
Author(s):  
Raimundo Pereira Silva-Néto ◽  
Ânderson Batista Rodrigues ◽  
Dandara Coelho Cavalcante ◽  
Pedro Henrique Piauilino Benvindo Ferreira ◽  
Ema Pereira Nasi ◽  
...  

Objectives The objective of this article is to characterize olfactory stimulation as a trigger of headaches attacks and differentiation between migraine and other primary headaches. Participants and methods The study was prospective and experimental, with comparison of groups. A total of 158 volunteers (73 men and 85 women) were diagnosed with primary headaches, according to the criteria of the International Classification of Headache Disorders, Third Edition (beta version) (ICHD-3β). The study was conducted by two examiners; one of them was assigned to diagnose the presence and type of primary headache, while the other was responsible for exposing the volunteers to odor and recording the effects of this exposure. Results Of the 158 volunteers with headache, there were 72 (45.6%) cases of migraine and 86 (54.4%) with other primary headaches. In both groups, there were differences in headache characteristics (χ2 = 4.132; p = 0.046). Headache attacks (25/72; 34.7%) and nausea (5/72; 6.9%) were triggered by odor only in patients with migraine, corresponding to 19.0% (30/158) of the sample, but in none with other primary headaches (χ2 = 43.78; p < 0.001). Headache occurred more often associated with nausea ( p = 0.146) and bilateral location ( p = 0.002) in migraineurs who had headache triggered by odor. Headache was triggered after 118 ± 24.6 min and nausea after 72.8 ± 84.7 min of exposure to odor. Conclusions The odor triggered headache attacks or nausea only in migraineurs. Therefore, headache triggered by odors may be considered a factor of differentiation between migraine and other primary headaches and this trigger seems very specific of migraine.


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