scholarly journals Correction to: Wave intensity, an index of ventriculo-arterial interaction, increases in hypertensive subjects but decreases in normotensive subjects during the cold pressor test

2021 ◽  
Author(s):  
Midori Tanaka ◽  
Motoaki Sugawara ◽  
Kiyomi Niki ◽  
Yasuo Ogasawara
Keyword(s):  
Cold Pressor Test ◽  
Cold Pressor ◽  
Wave Intensity ◽  
Pressor Test ◽  
Normotensive Subjects

scholarly journals Wave intensity, an index of ventriculo-arterial interaction, increases in hypertensive subjects but decreases in normotensive subjects during the cold pressor test

2021 ◽  
Author(s):  
Midori Tanaka ◽  
Motoaki Sugawara ◽  
Kiyomi Niki ◽  
Yasuo Ogasawara
Keyword(s):  
Blood Pressure ◽  
Cardiovascular Reactivity ◽  
Cold Pressor Test ◽  
Cold Pressor ◽  
Wave Intensity ◽  
Point Of View ◽  
Chi Square ◽  
Pressor Test ◽  
Ultrasonic Equipment ◽  
Normotensive Subjects

Abstract Purpose Cardiovascular reactivity to the cold pressor test (CPT) is considered to be a marker for apparent and potential hypertension. We aimed to elucidate the association between the changes in wave intensity (WI) during CPT and hypertension. Methods We recruited 85 volunteers, 33 of whom were hypertensive and 52 normotensive. Using ultrasonic equipment during CPT, we measured carotid arterial WI, which is defined in terms of blood pressure and velocity in the carotid artery. Results The peak WI (W1) increased during CPT in 70.6% of hypertensive individuals, but decreased in 72.6% of normotensive individuals. The chi-square (χ2) test showed that the association between the direction of change in W1 (increase or decrease) and the blood pressure (hypertensive or normotensive) was very strong (P < 0.0001). Conclusion Direction of change in W1 during CPT is a clear marker to discriminate cardiovascular reactivity that does not vary depending on each investigator’s subjective point of view.

Elevated Plasma Adrenaline Reflects Sympathetic Overactivity and Enhanced α-Adrenoceptor-Mediated Vasoconstriction in Essential Hypertension

1981 ◽  
Vol 61 (s7) ◽  
pp. 161s-164s ◽  
Author(s):  
P. Bolli ◽  
F. W. Amann ◽  
L. Hulthén ◽  
W. Kiowski ◽  
F. R. Bühler
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Blood Flow ◽  
Essential Hypertension ◽  
Forearm Blood Flow ◽  
Cold Pressor Test ◽  
Cold Pressor ◽  
Plasma Adrenaline ◽  
Pressor Test ◽  
Normotensive Subjects

1. Stressful sympathetic stimulation (cold pressor test) was applied to 18 patients with essential hypertension and 15 normotensive subjects. Intra-arterial blood pressure, heart rate, plasma adrenaline and noradrenaline concentrations as well as forearm blood flow were measured before and during the cold pressor test; tests were repeated after regional postsynaptic α1-adrenoceptor blockade with prazosin. 2. Under basal conditions mean blood pressure (P &lt; 0.001), heart rate (P &lt; 0.01), forearm blood flow (P &lt; 0.001) as well as adrenaline concentration (P &lt; 0.01), but not noradrenaline, was higher in patients with essential hypertension. 3. During the cold pressor test, mean blood pressure, heart rate, plasma adrenaline and noradrenaline concentrations increased and forearm flow decreased (all P &lt; 0.001). 4. Stress-stimulated plasma adrenaline was higher in essential hypertensive patients than in normotensive subjects (P &lt; 0.01). In the former the stress-induced increase in plasma adrenaline correlated with the increase in mean blood pressure (r = 0.514; P &lt; 0.05). 5. Prazosin increased forearm blood flow more in essential hypertension (P &lt; 0.001). This increase correlated with the resting plasma adrenaline in the hypertensive (r = 0.710; P &lt; 0.001), but not in normotensive, subjects. 6. When the cold pressor test was repeated during postsynaptic α1-adrenoceptor blockade forearm blood flow did not decrease; instead it increased further in both groups (P &lt; 0.05). 7. Thus in essential hypertension elevated plasma adrenaline concentration reflects sympathetic overactivity as also expressed by enhanced α-adrenoceptor-mediated vasoconstriction.

P1.13 ENHANCED VASCULAR REACTIVITY TO COLD PRESSOR TEST IN AFRICAN NORMOTENSIVE SUBJECTS

2008 ◽  
Vol 2 (3) ◽  
pp. 95
Author(s):  
D. Adamopoulos ◽  
W. Ngatsou ◽  
D. Lemogoum ◽  
B. Biholong ◽  
S. Beloka ◽  
...  
Keyword(s):  
Vascular Reactivity ◽  
Cold Pressor Test ◽  
Cold Pressor ◽  
Pressor Test ◽  
Normotensive Subjects

scholarly journals Different LF and HF HRV responses to cold pressor test in normotensive and prehypertensive men

Biologija ◽  
2019 ◽  
Vol 64 (4) ◽  
Author(s):  
Vera E. Shekh ◽  
Emeka Idot-Eyin Ofoegbu ◽  
Ebunoluwa Grace Adenuga ◽  
Larisa S. Shchyrova
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Linear Regression Analysis ◽  
Cold Pressor Test ◽  
Low Frequency ◽  
Cold Pressor ◽  
Multiple Linear Regression Analysis ◽  
Total Power ◽  
Pressor Test ◽  
Normotensive Subjects

Hypertension is a global health problem and a risk factor of cardiovascular disease, cerebrovascular and kidney disease. The cold pressor test (CPT) is commonly used as a sympathoexcitatory manoeuvre. Heart rate variability (HRV) is a useful tool for assessment of autonomic nervous system activity. The aim of this study was to evaluate cardiovascular and HRV responsiveness to CPT in 46 normotensive and 25 prehypertensive (PHT) young man. In ECG was recorded at rest and CPT for 5 min each to evaluate heart rate (HR), total power (TP), low frequency (LF) and high frequency (HF) power. General linear model was used to test for effect of prehypertension and CPT on HR, systolic blood pressure (SBP), diastolic blood pressure (DBP) and HRV variables and their responses to cold. The stepwise multiple linear regression analysis was used to reveal predictors of ∆HR, ∆SBP and ∆DBP. All analyses were conducted using SPSS 22. Blood pressure responsiveness to CPT was not associated with PHT; instead, it was associated with BMI. The ∆HR, ∆SBP, and ∆DBP were not significantly different between normotensive and prehypertensive subjects. However, ∆LnTP, ∆LnLF, and ∆LnHF were significantly higher in normotensives than in prehypertensives: LnTP, LnLF, and LnHF increased in normotensive subjects while these indices decreased in prehypertensives.

GENDER-RELATED CARDIOVASCULAR RESPONSES TO COLD PRESSOR TEST IN NORMOTENSIVE SUBJECTS

1996 ◽  
Vol 3 (6) ◽  
pp. 419-422
Author(s):  
Mario Blanco ◽  
Ana Aleman ◽  
Ingrid Jelambi ◽  
Northan Hurtado ◽  
Tatiana Franco ◽  
...  
Keyword(s):  
Cold Pressor Test ◽  
Cold Pressor ◽  
Cardiovascular Responses ◽  
Pressor Test ◽  
Normotensive Subjects

Repercussões pressóricas do frio local: a crioterapia deve ser administrada com precaução em pacientes hipertensos?

2016 ◽  
Vol 14 (4) ◽  
Author(s):  
Marcelo Coertjens
Keyword(s):  
Cold Pressor Test ◽  
Cold Pressor ◽  
Pressor Test

Introdução: A crioterapia é um recurso que diminui a temperatura corporal local com finalidades terapêuticas. Uma importante repercussão é a vasoconstrição local, que seria o desencadeador de um possível aumento na pressão arterial (PA). Entretanto, não existem trabalhos que comprovem essa suposição. Nossa hipótese é que os resultados das pesquisas de Cold Pressor Test (CPT) avaliando PA acabaram historicamente fundamentando as precauções da crioterapia em relação a pacientes hipertensos. Objetivo: Realizar uma revisão de literatura a respeito das pesquisas que sustentam a precaução da crioterapia em indivíduos hipertensos e verificar sua relação com estudos que utilizaram o CPT. Material e métodos: Trata-se de uma revisão de literatura que utilizou as bases de dados online Medline, Scielo, Lilacs e Google Acadêmico para a realização da pesquisa. Resultado: Apesar de não serem unânimes, diversas pesquisas que utilizaram o CPT encontraram significativos aumentos da atividade nervosa simpática muscular e da PA em normotensos e hipertensos, entretanto não encontramos estudos que tenham comprovado respostas significativas de PA com o uso da crioterapia, principalmente, em hipertensos. Conclusão: Não existem evidências científicas que comprovem a precaução da crioterapia em indivíduos hipertensos. Além disso, os estudos com CPT não são unânimes em relação aos aumentos pressóricos em indivíduos normotensos e hipertensos.Palavras-chave: crioterapia, hemodinâmica, hipertensão, pressão arterial. 

scholarly journals The expanding role of the cold pressor test: a brief history

2021 ◽  
Author(s):  
Guillaume Lamotte ◽  
Christopher J. Boes ◽  
Phillip A. Low ◽  
Elizabeth A. Coon
Keyword(s):  
Cold Pressor Test ◽  
Cold Pressor ◽  
Pressor Test

The tolerance to stretch is linked with endogenous modulation of pain

2021 ◽  
Vol 0 (0) ◽  
Author(s):  
Morten Pallisgaard Støve ◽  
Rogerio Pessoto Hirata ◽  
Thorvaldur Skuli Palsson
Keyword(s):  
Range Of Motion ◽  
Pain Sensitivity ◽  
Cold Pressor Test ◽  
Cold Pressor ◽  
Static Stretch ◽  
Pain Thresholds ◽  
Pressure Pain ◽  
Pressor Test ◽  
Pressure Pain Thresholds ◽  
Exercise Induced

Abstract Objectives The effect of stretching on joint range of motion is well documented, and although sensory perception has significance for changes in the tolerance to stretch following stretching the underlining mechanisms responsible for these changes is insufficiently understood. The aim of this study was to examine the influence of endogenous pain inhibitory mechanisms on stretch tolerance and to investigate the relationship between range of motion and changes in pain sensitivity. Methods Nineteen healthy males participated in this randomized, repeated-measures crossover study, conducted on 2 separate days. Knee extension range of motion, passive resistive torque, and pressure pain thresholds were recorded before, after, and 10 min after each of four experimental conditions; (i) Exercise-induced hypoalgesia, (ii) two bouts of static stretching, (iii) resting, and (iv) a remote, painful stimulus induced by the cold pressor test. Results Exercise-induced hypoalgesia and cold pressor test caused an increase in range of motion (p<0.034) and pressure pain thresholds (p<0.027). Moderate correlations in pressure pain thresholds were found between exercise-induced hypoalgesia and static stretch (Rho>0.507, p=0.01) and exercise-induced hypoalgesia and the cold pressor test (Rho=0.562, p=0.01). A weak correlation in pressure pain thresholds and changes in range of motion were found following the cold pressor test (Rho=0.460, p=0.047). However, a potential carryover hypoalgesic effect may have affected the results of the static stretch. Conclusions These results suggest that stretch tolerance may be linked with endogenous modulation of pain. Present results suggest, that stretch tolerance may merely be a marker for pain sensitivity which may have clinical significance given that stretching is often prescribed in the rehabilitation of different musculoskeletal pain conditions where reduced endogenous pain inhibition is frequently seen.

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