Ventricular tachycardia due to bundle branch reentry: Induction by spontaneous atrial premature beats

1988 ◽  
Vol 116 (2) ◽  
pp. 552-555 ◽  
Author(s):  
Mani Nallasivan ◽  
Ruth A. Appel ◽  
William J. Welch ◽  
Robert A. Bauernfeind
Keyword(s):  
Ventricular Tachycardia ◽  
Premature Beats ◽  
Bundle Branch Reentry

scholarly journals Re-entrant ventricular tachycardia as a complication of ablation of idiopathic ventricular premature beats from the right outflow tract: a case report

2020 ◽  
Author(s):  
Nam Van Tran ◽  
Samuel Rotman ◽  
Patrice Carroz ◽  
Etienne Pruvot
Keyword(s):  
Ventricular Tachycardia ◽  
Low Voltage ◽  
Perfect Match ◽  
Right Ventricular Outflow Tract ◽  
Outflow Tract ◽  
Ventricular Premature Beats ◽  
First Case ◽  
The Right ◽  
Premature Beats ◽  
Pace Mapping

Abstract Background We report an unusual case of non-sustained ventricular tachycardia (NSVT) from the epicardial part of the right ventricular outflow tract (RVOT). Case summary A 37-year-old woman who underwent in 2006 an ablation for idiopathic ventricular premature beats (VPBs) from the RVOT presented with pre-syncopal NSVT in 2016. A cardiac workup showed no coronary disease, normal biventricular function, and no enhancement on cardiac magnetic resonance imaging. A metabolic positron emission tomography scan excluded inflammation. Biopsies revealed normal desmosomal proteins. An endocardial mapping revealed an area of low voltage potential (<0.5 mV) at the antero-septal aspect of the RVOT corresponding to the initial site of ablation from 2006. Activation mapping revealed poor prematurity and pace-mapping showed unsatisfactory morphologies in the RVOT, the left ventricle outflow tract and the right coronary cusp. An epicardial map revealed a low voltage area at the antero-septal aspect of the RVOT with fragmented potentials opposite to the endocardial scar. Pace-mapping demonstrated perfect match. An NSVT was induced and local electrocardiogram showed mid-diastolic potentials. Ablation was applied epicardially and endocardially without any complication. The patient was arrhythmia free at 4-year follow-up. Discussion Cardiac workup allowed to exclude specific conditions such as arrhythmogenic cardiomyopathy, tetralogy of Fallot, sarcoidosis, or myocarditis as a cause for NSVT from the RVOT. The epi and endocardial map showed residual scar subsequent to the first ablation which served as substrate for the re-entrant NSVT. This is the first case which describes NSVT from the epicardial RVOT as a complication from a previous endocardial ablation for idiopathic VPB.

Sustained monomorphic ventricular tachycardia: the role of catheter ablation

ESC CardioMed ◽  
2018 ◽  
pp. 2279-2288
Author(s):  
Tilman Maurer ◽  
William G. Stevenson ◽  
Karl-Heinz Kuck
Keyword(s):  
Heart Disease ◽  
Ventricular Tachycardia ◽  
Catheter Ablation ◽  
Sudden Cardiac Death ◽  
Cardiac Death ◽  
Therapeutic Option ◽  
Structural Heart Disease ◽  
Premature Ventricular Contractions ◽  
Monomorphic Ventricular Tachycardia ◽  
Premature Beats

Monomorphic ventricular tachycardia (VT) may occur in the presence or absence of structural heart disease. The standard therapy for patients with structural heart disease at high risk of sudden cardiac death due to VT is the implantable cardioverter defibrillator (ICD). While ICDs effectively terminate VT and prevent sudden cardiac death, they do not prevent recurrent episodes of VT, since the underlying arrhythmogenic substrate remains unchanged. However, shocks from an ICD increase mortality and impair quality of life. These limitations as well as continuous advancements in technology have made catheter ablation an important treatment strategy for patients with structural heart disease presenting with VT. Idiopathic ventricular arrhythmias include premature ventricular contractions and VT occurring in the absence of overt structural heart disease. In this setting, catheter ablation has evolved as the primary therapeutic option for symptomatic ventricular premature beats and sustained VTs and is curative in most cases. This chapter presents an overview of the principles of invasive diagnosis and treatment of monomorphic VTs in patients with and without structural heart disease and delineates the clinical outcome of catheter ablation. Finally, the chapter provides an outlook to the future, discussing potential directions and upcoming developments in the field of catheter ablation of monomorphic VT.

scholarly journals Bundle-branch reentry ventricular tachycardia after transcatheter aortic valve replacement

2015 ◽  
Vol 15 (5) ◽  
pp. 251-254
Author(s):  
Adriana de la Rosa Riestra ◽  
José Amador Rubio Caballero ◽  
Alfonso Freites Estévez ◽  
Javier Alonso Belló ◽  
Javier Botas Rodríguez
Keyword(s):  
Ventricular Tachycardia ◽  
Aortic Valve ◽  
Aortic Valve Replacement ◽  
Valve Replacement ◽  
Transcatheter Aortic Valve Replacement ◽  
Transcatheter Aortic Valve ◽  
Bundle Branch Reentry

scholarly journals Outpatient evaluation and management of patients with ventricular premature beats or non-sustained ventricular tachycardia

EP Europace ◽  
2012 ◽  
Vol 14 (2) ◽  
pp. 294-296 ◽  
Author(s):  
J. H. Svendsen ◽  
A. Goette ◽  
D. Dobreanu ◽  
G. Marinskis ◽  
P. Mabo ◽  
...  
Keyword(s):  
Ventricular Tachycardia ◽  
Sustained Ventricular Tachycardia ◽  
Ventricular Premature Beats ◽  
Premature Beats

Ischemia- and reperfusion-induced ventricular arrhythmias in dogs: effects of estrogen

1995 ◽  
Vol 268 (6) ◽  
pp. H2569-H2573 ◽  
Author(s):  
N. A. McHugh ◽  
S. M. Cook ◽  
J. L. Schairer ◽  
M. M. Bidgoli ◽  
G. F. Merrill
Keyword(s):  
Ventricular Tachycardia ◽  
Ventricular Arrhythmias ◽  
Coronary Perfusion Pressure ◽  
Ventricular Premature Beat ◽  
Coronary Perfusion ◽  
Ischemia And Reperfusion ◽  
Myocardial Ischemia And Reperfusion ◽  
Exogenous Estrogen ◽  
Life Threatening ◽  
Premature Beats

The purpose of this investigation was to determine if exogenous estrogen could attenuate the ventricular arrhythmias caused by myocardial ischemia and reperfusion. Conjugated equine estrogen, administered as an intravenous bolus injection (100 micrograms) to anesthetized, instrumented beagles of both genders, significantly attenuated the incidence of ventricular arrhythmias during a 20-min period of ischemia (2 +/- 1 vs. 19 +/- 16% ectopy) and in the first 5 min of reperfusion (15 +/- 9 vs. 69 +/- 20% ectopy). By 15-20 min of ischemia, ventricular salvos and nonsustained ventricular tachycardia were frequently observed in nontreated dogs. One dog in this group fibrillated during ischemia. In contrast, estrogen-treated dogs exhibited only an occasional ventricular premature beat during the same period of ischemia. When compared with baseline values, the percent ectopy during ischemia in estrogen-treated dogs was insignificant. During reperfusion, nontreated dogs displayed severe, life-threatening arrhythmias such as sustained ventricular tachycardia. In two of these dogs ventricular tachycardia deteriorated to ventricular fibrillation. In comparison, estrogen-treated dogs displayed only innocuous ventricular arrhythmias during reperfusion, i.e., ventricular premature beats, ventricular salvos, and ventricular bigeminy. In addition to the effect of estrogen on arrhythmias, there was a gradual increase in coronary blood flow on reperfusion in estrogen-treated dogs. This effect of estrogen was preceded by a significantly higher coronary perfusion pressure during ischemia (31 +/- 2 vs. 18 +/- 4 mmHg, P < 0.05). In conclusion, our findings suggest that antiarrhythmic effects of estrogen treatment might stabilize ventricular rhythmicity during ischemia and reperfusion.

Bundle branch reentry ventricular tachycardia

2018 ◽  
pp. 397-412
Author(s):  
John M. Miller ◽  
Mithilesh K. Das ◽  
Douglas P. Zipes
Keyword(s):  
Ventricular Tachycardia ◽  
Bundle Branch Reentry
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