Vein of galen aneurysm presenting with recurrent aseptic meningitis and subsequent spontaneous thrombosis

1990 ◽  
Vol 33 (5) ◽  
pp. 325-328 ◽  
Author(s):  
John J. Collins ◽  
W.S. Fisher
2006 ◽  
Vol 64 (3b) ◽  
pp. 862-864 ◽  
Author(s):  
Robinson M. Marques ◽  
Carlos A.F. Lobão ◽  
Viviane S. Sassaki ◽  
Luiz R. Aguiar

Vein of Galen aneurysm is a rare pathology, representing less than 1% of intracranial vascular malformations. We report on a 65 years-old man who experienced a generalized tonic-clonic seizure. Brain imaging showed a large calcified expanding mass in the pineal region, confirming the diagnosis of a vein of Galen aneurysm. Because of the spontaneous thrombosis of the malformation, there was no need for microsurgical or endovascular treatment and he is been regularly followed since that.


2009 ◽  
Vol 50 (7) ◽  
pp. 812-815
Author(s):  
M. Irfan ◽  
B. Lohman ◽  
A. M. McKinney

Spontaneous thrombosis of a vein of Galen aneurysmal malformation (VOGM) is rare. We describe a 2-month-old patient with a patent VOGM and hydrocephalus, also confirmed patent at 6 months, but with subsequent lack of filling on preembolization catheter digital subtraction angiography (DSA) at 9 months’ age. Due to the presence of T1- and T2-bright signal, noncontrast T1-weighted images (T1WI), T2-weighted images (T2WI), two-dimensional (2D) time-of-flight (TOF) magnetic resonance venography (MRV), and postcontrast T1WI were ambiguous for patency. However, subtracting the pre- from the postcontrast MRV images confirmed closure compared to subtracted images at 6 months’ age. The factors contributing to thrombosis were likely a combination of a disproportionately small straight sinus, ventriculostomy, and contrast medium from DSA.


Neurosurgery ◽  
1988 ◽  
Vol 22 (5) ◽  
pp. 908-910 ◽  
Author(s):  
Jane Matjasko ◽  
Walker Robinson ◽  
Daniel Eudaily

Abstract A 12-day-old infant in intractable cardiac failure due to a vein of Galen malformation was treated successfully with serial ligation of the majority of the vessels feeding the malformation. Despite some residual vascular supply to the malformation, the congestive heart failure has disappeared and growth and development have been normal over a 3-year follow-up period.


DICP ◽  
1991 ◽  
Vol 25 (11) ◽  
pp. 1183-1184 ◽  
Author(s):  
Babette B. Weksler ◽  
Anne Marie Lehany

2014 ◽  
Vol 16 (suppl 5) ◽  
pp. v48-v49
Author(s):  
N. Majd ◽  
E. Neil ◽  
R. Warnick ◽  
J. Tew ◽  
R. Curry

Neurosurgery ◽  
1982 ◽  
Vol 10 (2) ◽  
pp. 258-262 ◽  
Author(s):  
Michael S. Olin ◽  
Atef A. Eltomey ◽  
Rembrandt H. Dunsmore ◽  
Melville P. Roberts

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