Primary structure of bovine Hageman factor (blood coagulation factor XII): comparison with human and guinea pig molecules

1994 ◽  
Vol 1206 (1) ◽  
pp. 63-70 ◽  
Author(s):  
Yoko Shibuya ◽  
Umeko Semba ◽  
Hiroaki Okabe ◽  
Takeshi Kambara ◽  
Tetsuro Yamamoto
Keyword(s):  
Guinea Pig ◽  
Blood Coagulation ◽  
Primary Structure ◽  
Coagulation Factor ◽  
Factor Xii ◽  
Hageman Factor

Rabbit blood coagulation factor XI. Mechanism of activation by rabbit Hageman factor (factor XII)

1979 ◽  
Vol 15 (3-4) ◽  
pp. 487-495 ◽  
Author(s):  
Roger C. Wiggins ◽  
Charles G. Cochrane ◽  
John H. Griffin
Keyword(s):  
Blood Coagulation ◽  
Coagulation Factor ◽  
Factor Xii ◽  
Factor Xi ◽  
Hageman Factor ◽  
Rabbit Blood

Quantitative immunoblotting assay of blood coagulation factor XII

1986 ◽  
Vol 41 (6) ◽  
pp. 747-759 ◽  
Author(s):  
Bernhard Lämmle ◽  
Mauro Berrettini ◽  
Hans Peter Schwarz ◽  
Mary Jo Heeb ◽  
John B. Griffin
Keyword(s):  
Blood Coagulation ◽  
Coagulation Factor ◽  
Factor Xii ◽  
Immunoblotting Assay ◽  
Quantitative Immunoblotting

A hitherto undescribed plasma factor acting at the contact phase of blood coagulation (Flaujeac factor): case report and coagulation studies

Blood ◽  
1975 ◽  
Vol 46 (5) ◽  
pp. 761-768 ◽  
Author(s):  
MJ Lacombe ◽  
B Varet ◽  
JP Levy
Keyword(s):  
Blood Coagulation ◽  
Coagulation Factor ◽  
Old French ◽  
Bleeding Tendency ◽  
Contact Phase ◽  
Hageman Factor ◽  
Coagulation Defect ◽  
Plasma Factor ◽  
Fletcher Factor

Abstract This paper reports an asymptomatic coagulation defect responsible for an abnormality at the contact phase of blood coagulation in vitro, distinct from Hageman factor and Fletcher factor deficiencies. Coagulation studies in a 50-yr-old French woman without bleeding tendency revealed the following results: whole-blood clotting time in glass tubes and activated partial thromboplastin time with kaolin and ellagic acid were greatly prolonged; one-stage prothrombin was normal; no circulating anticoagulant was detected, and the infusion of normal plasma corrected the coagulation defect with an estimated half-life of 6.5 days; the levels of factor VIII, IX, XI, and XII were normal; mutual correction was obtained with a Fletcher factor-deficient plasma; the level of whole complement was normal. Studies of the contact phase of blood coagulation and contact-induced fibrinolysis showed the same abnormalities as in Hageman factor- and Fletcher-deficient plasmas. These results indicate that the patient's plasma is deficient in a previously undescribed coagulation factor, which participates in the initial stage of the blood coagulation process in vitro. Family studies revealed consanguinity in the propositus' parents. The assay of this newly described factor in the propositus' children revealed a partial defect, compatible with a heterozygous state, in three of the four tested children. This indicates a recessive inheritance of this new blood coagulation defect.

scholarly journals Blood coagulation factor XII drives adaptive immunity during neuroinflammation via CD87-mediated modulation of dendritic cells

2016 ◽  
Vol 7 (1) ◽  
Author(s):  
Kerstin Göbel ◽  
Susann Pankratz ◽  
Chloi-Magdalini Asaridou ◽  
Alexander M. Herrmann ◽  
Stefan Bittner ◽  
...  
Keyword(s):  
Dendritic Cells ◽  
Blood Coagulation ◽  
Adaptive Immunity ◽  
Coagulation Factor ◽  
Factor Xii

Acceleration of surface-dependent autocatalytic activation of blood coagulation factor XII by divalent metal ions

Biochemistry ◽  
1987 ◽  
Vol 26 (8) ◽  
pp. 2250-2258 ◽  
Author(s):  
Joseph D. Shore ◽  
Duane E. Day ◽  
Paul E. Bock ◽  
Steven T. Olson
Keyword(s):  
Metal Ions ◽  
Blood Coagulation ◽  
Coagulation Factor ◽  
Divalent Metal ◽  
Factor Xii ◽  
Divalent Metal Ions ◽  
Autocatalytic Activation
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