scholarly journals Time contraction caused by a distractor in children and adults: The influence of inhibition capacities

2020 ◽  
Vol 210 ◽  
pp. 103186
Author(s):  
Quentin Hallez
Keyword(s):  
2006 ◽  
Vol 15 (03) ◽  
pp. 755-760
Author(s):  
GERALD MOTT

Using only the descriptions and the results of the 'thought experiment' contained in Einstein's seminal 1905 paper, proofs are offered which show that the transformation equations of Einstein's special relativity apply only to the joint use in his experiment of point sources of light and point reflectors. Further, it is shown that two different special relativities could have been invented by Einstein and, because they possess differing space and time contraction factors, they cannot co-exist and, therefore, both must be discarded.


1991 ◽  
Vol 9 (2) ◽  
pp. 363-370 ◽  
Author(s):  
L. Bergé ◽  
PH. Dousseau ◽  
G. Pelletier ◽  
D. Pesme

Two spherically symmetric versions of a self-similar collapse are investigated within the framework of the Zakharov equations, namely, one relative to a vectorial electric field and the other corresponding to a scalar modeling of the Langmuir field. Singular solutions of both of them depend on a linear time contraction rate Ξ(t) = V(t* – t), where t* and V = – Ξ denote, respectively, the collapse time and the constant collapse velocity. We show that under certain conditions, only the scalar model admits self-similar solutions, varying regularly as a function of the control parameter V from the subsonic (V ≪ 1) to the supersonic (V ≫ 1) regime.


1993 ◽  
Vol 264 (2) ◽  
pp. H512-H519 ◽  
Author(s):  
S. Fukuda ◽  
M. Morioka ◽  
T. Tanaka ◽  
K. Taga ◽  
K. Shimoji

To investigate the modulation of CO2 and endothelium of vascular contraction induced by various agonists, we studied the influence of high PCO2 (PCO2 = 91 mmHg, pH = 6.99) on the response of endothelium-intact and -rubbed rat aortic preparations to KCl, phenylephrine (PE), and human-porcine endothelin-1 (ET-1). Response of endothelium-intact aortic preparations to KCl was not influenced by both high PCO2 and the pH-matched acidotic solution (7.00) with normal PCO2, whereas that of endothelium-rubbed preparations was attenuated solely by high PCO2. With cyclooxygenase inhibitors or a thromboxane A2 receptor antagonist, high PCO2 attenuated the respose of both preparations to KCl. The dose-response curve of endothelium-intact and -rubbed preparations to PE was shifted to the right by both high PCO2 and the pH-matched acidotic solution with normal PCO2. The maximal response of endothelium-intact preparation to PE was attenuated by high PCO2. Indomethacin augmented the inhibitory action of high PCO2 on the PE-induced contraction. Contractile responses of endothelium-intact and -rubbed preparations to ET-1 were not influenced by high PCO2. With indomethacin, high PCO2 also had no influence on the ET-1-induced contraction of endothelium-intact preparations. Endothelium modified the high PCO2 effects on the time-contraction responses to the three agonists. CO2 and endothelium may variously modify the responses of rat aorta to different agonists. Cyclooxygenase-related eicosanoid(s) may be involved in the effects of high PCO2 on the response of rat aortic smooth muscle cells to KCl and PE.


2012 ◽  
Vol 12 (7) ◽  
pp. 8-8 ◽  
Author(s):  
N. Binetti ◽  
F. Lecce ◽  
F. Doricchi

2006 ◽  
Vol 84 (11) ◽  
pp. 1133-1138 ◽  
Author(s):  
Atanas D. Krustev ◽  
Mariana D. Argirova ◽  
Damianka P. Getova ◽  
Valentin I. Turiiski ◽  
Natalia A. Prissadova

Tacrine, a non-competitive reversible acetylcholinesterase and butyrylcholineserase inhibitor, caused a concentration-dependent tonic contraction of gastric smooth muscle preparations in the concentration range 1 × 10−7 mol/L – 1 × 10−5 mol/L, whereas concentrations higher than 2 × 10−5 mol/L induced a biphasic effect; a short-time contraction was followed by a prolonged relaxation. To shed some light on the mechanism underlying this untypical relaxation, the amplitude of mechanical reactions caused by tacrine were compared with those of tacrine in the presence of atropine, ipratropium, metrifonate, TTX, nifedipine, D-600, caffeine, apamin, and charybdotoxin. The results obtained revealed that the relaxation was neither cholinergic in nature, nor mediated by the influence of the drug on intramural neuronal structures. It was not influenced by processes inducing changes in cytosolic Ca2+ levels. This assumption was confirmed by experiments with permeabilized muscle preparations that were pre-contracted in a solution with pCa 5.5. Tacrine relaxed the smooth muscles in spite of the constant intracellular Ca2+ concentration resulting from the permeabilization. These findings argue that tacrine at concentrations higher than 2 × 10−5 mol/L has a desensitizing effect on the contractile apparatus of gastric corpus smooth muscle preparations towards Ca2+.


1995 ◽  
Vol 80 (3_suppl) ◽  
pp. 1103-1107 ◽  
Author(s):  
Lutz Jäncke ◽  
Harald Hefter ◽  
Karl Theodor Kalveram

10 adult stutterers and 10 nonstutterers were required to extend as fast as possible their right and left index fingers under isometric conditions in response to an auditory signal. Force developed during finger extension was measured continuously during each trial. From these force records reaction time, contraction time, and peak force were measured. For stutterers, contraction times were longer (about 36 msec.) than for nonstutterers. Possible reasons for this result are discussed.


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