Hyperbaric oxygen therapy for the prevention of arterial gas embolism in food grade hydrogen peroxide ingestion

2017 ◽  
Vol 35 (5) ◽  
pp. 809.e5-809.e8 ◽  
Author(s):  
Stephen M. Hendriksen ◽  
Nicholas L Menth ◽  
Bjorn C. Westgard ◽  
Jon B. Cole ◽  
Joseph W. Walter ◽  
...  
2021 ◽  
pp. 177-186
Author(s):  
Stella Pak ◽  
◽  
Christopher Lee ◽  
Joseph Lach ◽  
George Ortiz ◽  
...  

A 52-year-old male accidentally ingested approximately 100 mL of 35% hydrogen peroxide (H2O2), resulting in the sudden onset of gastrointestinal and neurologic symptoms. Non-contrast abdominal CT revealed extensive portal venous gas and gastric pneumatosis. The patient was treated with hyperbaric oxygen therapy which resulted in complete resolution of symptoms. The case highlights the therapeutic value of hyperbaric oxygen therapy in the treatment of vascular gas embolism and mitigation of concentrated H2O2 ingestion toxicity.


2019 ◽  
Vol 68 (7) ◽  
pp. 456-460
Author(s):  
L. Hellinger ◽  
A. M. Keppler ◽  
H. Schoeppenthau ◽  
J. Perras ◽  
R. Bender

2017 ◽  
Vol 18 (2) ◽  
pp. 134-136 ◽  
Author(s):  
Louise Segan ◽  
Fiona Permezel ◽  
Wei Ch’ng ◽  
Ian Millar ◽  
Mark Brooks ◽  
...  

Cerebral arterial gas embolism is a recognised complication of endovascular intervention with an estimated incidence of 0.08%. Its diagnosis is predominantly clinical, supported by neuroimaging. The treatment relies on alleviating mechanical obstruction and reversing the proinflammatory processes that contribute to tissue ischaemia. Hyperbaric oxygen therapy is an effective treatment and has multiple mechanisms to reverse the pathological processes involved in cerebral arterial gas embolism. Symptomatic cerebral arterial gas embolism is a rare complication of endovascular intervention for acute ischaemic stroke. Although there are no previous descriptions of its successful treatment with hyperbaric oxygen therapy following mechanical thrombectomy, this is likely to become more common as mechanical thrombectomy is increasingly used worldwide to treat acute ischaemic stroke.


2012 ◽  
Vol 18 (2) ◽  
pp. 228-233 ◽  
Author(s):  
Wondwossen G. Tekle ◽  
Cheryl D. Adkinson ◽  
Saqib A. Chaudhry ◽  
Vikram Jadhav ◽  
Ameer E. Hassan ◽  
...  

2014 ◽  
Vol 46 (2) ◽  
pp. 171-175 ◽  
Author(s):  
Brendan Byrne ◽  
Robert Sherwin ◽  
Cheryl Courage ◽  
Alfred Baylor ◽  
Bram Dolcourt ◽  
...  

2021 ◽  
pp. 187-193
Author(s):  
Je-seop Lee ◽  
◽  
Youn Sung Cha ◽  

Hydrogen peroxide (H2O2) ingestion can cause vascular gas embolism (GE). Hyperbaric oxygen therapy (HBO2) is known to improve neurological abnormalities in patients with arterial gas embolism (AGE). Previously, HBO2 based on the U.S. Navy Table 6 diving protocol has been adopted for treating AGE and preventing the progression of portal venous GE, caused by H2O2 ingestion, to AGE. However, the indication and protocol for HBO2 have not been established for GE related to H2O2 ingestion. Herein, we describe a case in which GE caused by H2O2 ingestion was treated using HBO2 with a short protocol. A 69-year-old female patient presented with abdominal pain, vomiting, and transient loss of consciousness after ingesting 35% H2O2. Computed tomography revealed gastric wall and portal venous gas. She was administered an HBO2 protocol with 2.8-atmosphere absolute (ATA) compression for 45 minutes. This was followed by a 2.0-ATA treatment for 60 minutes with a five-minute air break, after which all gas bubbles disappeared. After HBO2 treatment, brain magnetic resonance imaging revealed focal cytotoxic edema lesions; however, the patient was discharged without additional symptoms.


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