scholarly journals Mitochondrial Creatine Kinase Attenuates ROS Emission and Improves Myocyte Survival after ROS in the Failing Heart

2019 ◽  
Vol 116 (3) ◽  
pp. 156a
Author(s):  
Gizem Keceli ◽  
Joevin Sourdon ◽  
Ashish Gupta ◽  
Carlo G. Tocchetti ◽  
Bongsoo Park ◽  
...  
1989 ◽  
Vol 264 (5) ◽  
pp. 2890-2897
Author(s):  
R C Haas ◽  
C Korenfeld ◽  
Z F Zhang ◽  
B Perryman ◽  
D Roman ◽  
...  

JAMA ◽  
1980 ◽  
Vol 243 (20) ◽  
pp. 2058 ◽  
Author(s):  
C. Jack Bark

1998 ◽  
Vol 254 (1) ◽  
pp. 172-180 ◽  
Author(s):  
Frank Norbert Gellerich ◽  
Fanny Dorine Laterveer ◽  
Bernard Korzeniewski ◽  
Stephan Zierz ◽  
Klaas Nicolay

Author(s):  
J Smeitink ◽  
W Ruitenbeek ◽  
T v Lith ◽  
R Sengers ◽  
F Trijbels ◽  
...  

We studied pre- and postnatal changes in total creatine kinase (CK) activity, mitochondrial creatine kinase (Mi-CK) activity and immunochemical reactivity with anti-Mi-CK antibodies in skeletal muscle specimens from 12 infants, 10 of them preterm born, after a pregnancy varying between 28 and 40 weeks. Our results demonstrate that Mi-CK is present in fetal human quadriceps muscle and that the specific activity of Mi-CK increases during prenatal development from week 28 to 40 by a factor about two. Generally, adult levels have not been reached at birth, indicating a further postnatal increase of the activity of the enzyme. The Mi-CK protein content also increases during prenatal development. These results suggest that in human skeletal muscle the expression and accumulation of Mi-CK starts at mid-gestation, later than is known to occur for cytosolic CK.


1986 ◽  
Vol 32 (1) ◽  
pp. 236-236
Author(s):  
W L Chandler ◽  
J S Fine ◽  
K J Clayson

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