Neuroprotective effects of ethyl pyruvate on brain energy metabolism after ischemia depend on glial cells: A 31P-NMR study

2007 ◽  
Vol 58 ◽  
pp. S240
Author(s):  
Osamu Tokumaru ◽  
Chihiro Kuroki ◽  
Takaaki Kitano ◽  
Naoko Nisimaru ◽  
Isao Yokoi
Keyword(s):  
Energy Metabolism ◽  
Glial Cells ◽  
31P Nmr ◽  
Ethyl Pyruvate ◽  
Brain Energy Metabolism ◽  
Neuroprotective Effects ◽  
Nmr Study ◽  
Brain Energy

Depolarizing high-[K+] load model of rat brain energy metabolism: α 31P-NMR study

2009 ◽  
Vol 65 ◽  
pp. S128
Author(s):  
Osamu Tokumaru ◽  
Chihiro Kuroki ◽  
Kazue Ogata ◽  
Takaaki Kitano ◽  
Isao Yokoi
Keyword(s):  
Energy Metabolism ◽  
Rat Brain ◽  
31P Nmr ◽  
Brain Energy Metabolism ◽  
Load Model ◽  
Nmr Study ◽  
High K ◽  
Brain Energy

Effects of increased ICP on brain phosphocreatine and lactate determined by simultaneous 1H and 31P NMR spectroscopy

1987 ◽  
Vol 67 (3) ◽  
pp. 381-386 ◽  
Author(s):  
Leslie N. Sutton ◽  
Alan C. McLaughlin ◽  
William Kemp ◽  
M. D. Schnall ◽  
Byung-Ku Cho ◽  
...  
Keyword(s):  
Energy Metabolism ◽  
Cerebral Perfusion ◽  
31P Nmr ◽  
Perfusion Pressure ◽  
Brain Energy Metabolism ◽  
Resonance Spectroscopy ◽  
31P Nmr Spectroscopy ◽  
Content Type ◽  
Brain Energy ◽  
Increased Icp

✓ In order to study the metabolic events surrounding ischemia induced by the graded increase of cerebrospinal fluid (CSF) pressure, the technique of simultaneous phosphorus-31- and hydrogen-1-enhanced nuclear magnetic resonance spectroscopy was applied to five cats as intracranial pressure (ICP) was gradually raised by the instillation of mock CSF. Threshold lactate rose at an average cerebral perfusion pressure (CPP) of 49 torr, and, in general, preceded a threshold decrease in phosphocreatine, which was observed at an average CPP of 29 torr. There was considerable variation among cats in the CPP at which failure of brain energy metabolism occurred, however, suggesting differences in the autoregulatory curves. It is concluded that, with elevated ICP, there is no universally “safe” CPP at which brain energy metabolism may be assumed to be uncompromised.

scholarly journals Brain Energy Metabolism: Conserved Functions of Glycolytic Glial Cells

2015 ◽  
Vol 22 (3) ◽  
pp. 361-363 ◽  
Author(s):  
Andrea Trevisiol ◽  
Klaus-Armin Nave
Keyword(s):  
Energy Metabolism ◽  
Glial Cells ◽  
Brain Energy Metabolism ◽  
Brain Energy

Effects of fasting, intermittent feeding, or continuous parenteral nutrition on rat liver and brain energy metabolism as assessed by 31P-NMR

1995 ◽  
Vol 58 (3) ◽  
pp. 521-527 ◽  
Author(s):  
Gyorgy Bodoky ◽  
Zhong-Jin Yang ◽  
Michael M. Meguid ◽  
Alessandro Laviano ◽  
Nikolaus Szeverenyi
Keyword(s):  
Energy Metabolism ◽  
Parenteral Nutrition ◽  
Rat Liver ◽  
31P Nmr ◽  
Brain Energy Metabolism ◽  
Intermittent Feeding ◽  
Brain Energy
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