scholarly journals Post-oral glucose stimulation of intake and conditioned flavor preference in C57BL/6J mice: A concentration–response study

2013 ◽  
Vol 109 ◽  
pp. 33-41 ◽  
Author(s):  
Steven Zukerman ◽  
Karen Ackroff ◽  
Anthony Sclafani
Keyword(s):  
Oral Glucose ◽  
Flavor Preference ◽  
Glucose Stimulation ◽  
Stimulation Of ◽  
Conditioned Flavor

scholarly journals Post-oral fat stimulation of intake and conditioned flavor preference in C57BL/6J mice: A concentration-response study

2014 ◽  
Vol 129 ◽  
pp. 64-72 ◽  
Author(s):  
Karen Ackroff ◽  
Anthony Sclafani
Keyword(s):  
Flavor Preference ◽  
Stimulation Of ◽  
Conditioned Flavor

Glucose stimulation of somatostatin and insulin release from the isolated, perfused rat pancreas

Diabetes ◽  
1980 ◽  
Vol 29 (9) ◽  
pp. 747-751 ◽  
Author(s):  
R. L. Sorenson ◽  
D. V. Lindell ◽  
R. P. Elde
Keyword(s):  
Insulin Release ◽  
Glucose Stimulation ◽  
Perfused Rat Pancreas ◽  
Rat Pancreas ◽  
Isolated Perfused Rat Pancreas ◽  
Stimulation Of

High-glucose stimulation of 64,000-Mr islet cell autoantigen expression

Diabetes ◽  
1989 ◽  
Vol 38 (10) ◽  
pp. 1326-1328 ◽  
Author(s):  
O. Kampe ◽  
A. Andersson ◽  
E. Bjork ◽  
A. Hallberg ◽  
F. A. Karlsson
Keyword(s):  
High Glucose ◽  
Islet Cell ◽  
Glucose Stimulation ◽  
Stimulation Of

scholarly journals Glucose stimulation of insulin secretion from the isolated pancreas of foetal and newborn rats

Diabetologia ◽  
1969 ◽  
Vol 5 (4) ◽  
pp. 260-262 ◽  
Author(s):  
K. Asplound ◽  
S. Westman ◽  
C. Hellerste�m
Keyword(s):  
Insulin Secretion ◽  
Newborn Rats ◽  
Glucose Stimulation ◽  
Isolated Pancreas ◽  
Stimulation Of

scholarly journals Evaluation of Peripheral Blood Neutrophil Functions after an Oral Carbohydrate Overload in Obese and Insulin Resistant Horses

2020 ◽  
Author(s):  
Constanza Salinas ◽  
Gabriel Espinosa ◽  
Natalia Morales ◽  
Claudio Henriquez ◽  
Gabriel Moran ◽  
...  
Keyword(s):  
Insulin Resistance ◽  
Oxidative Burst ◽  
Peripheral Blood ◽  
Oral Glucose ◽  
Close Monitoring ◽  
Metabolic Dysfunction ◽  
Phagocytic Capacity ◽  
Insulin Resistant ◽  
Peripheral Blood Neutrophil ◽  
Stimulation Of

Abstract Background: Obesity and insulin resistance (IR) are conditions of increasing prevalence in populations of equids worldwide. The immune impairment described in metabolic dysfunction status in humans has been extensively reported with minimal data regarding horses. The objective of the study was to evaluate the effect of obesity as an isolated factor and in association with insulin resistance on apoptosis, phagocytosis and oxidative burst activity of neutrophils isolated from peripheral blood of lean and obese adult horses with or without insulin resistance, basally and after induction of hyperglycemia through an oral glucose test. Results: No differences in apoptosis were observed between experimental groups at any time point. Phagocytic capacity was significantly diminished at baseline in the obese-IR group (P<0.05) but increased after stimulation of hyperglycemia (P=0.007). Basal reactive oxygen species production differed significantly (P=0.0001) between the obese-insulin sensitive (IS) and lean-IS or obese-IR groups, and decreased significantly after stimulation of hyperglycemia in the lean-IS and obese-IS groups (P<0.05).Conclusions: Results from this study showed that both metabolic status itself, and acute hyperglycemia, are factors that influence PMNs functionality in horses, specifically phagocytosis and oxidative burst. This indicates the need for close monitoring of immune function in horses with inflammatory disease and concurrent obesity and insulin resistance.

Regulation of the Insulin Gene by Glucose: Stimulation of Trans-Activation Potency of Human PDX-1 N-Terminal Domain

1999 ◽  
Vol 18 (6) ◽  
pp. 471-479 ◽  
Author(s):  
Etti Ben Shushan ◽  
Erol Cerasi ◽  
Danielle Melloul
Keyword(s):  
Insulin Gene ◽  
Glucose Stimulation ◽  
Terminal Domain ◽  
Stimulation Of

Formate and glucose stimulation of methane and hydrogen production in rumen liquor

1990 ◽  
Vol 20 (4) ◽  
pp. 251-254 ◽  
Author(s):  
Jayne E. Ellis ◽  
Alan G. Williams ◽  
David Lloyd
Keyword(s):  
Hydrogen Production ◽  
Glucose Stimulation ◽  
Stimulation Of

scholarly journals Dopamine D1-like receptor antagonism in amygdala impairs the acquisition of glucose-conditioned flavor preference in rats

2009 ◽  
Vol 30 (2) ◽  
pp. 289-298 ◽  
Author(s):  
Khalid Touzani ◽  
Richard J. Bodnar ◽  
Anthony Sclafani
Keyword(s):  
Flavor Preference ◽  
Receptor Antagonism ◽  
Conditioned Flavor

Hyperinsulinemia in rats with hypothalamic obesity: effects of autonomic drugs and glucose

1983 ◽  
Vol 245 (3) ◽  
pp. R372-R378 ◽  
Author(s):  
S. Inoue ◽  
Y. S. Mullen ◽  
G. A. Bray
Keyword(s):  
Adrenergic Receptors ◽  
Serum Insulin ◽  
The Other ◽  
Glucose Stimulation ◽  
Hypothalamic Obesity ◽  
Insulin Levels ◽  
Alpha Adrenergic Receptors ◽  
Beta Receptors ◽  
Increased Sensitivity ◽  
Stimulation Of

The present study examined the effects of autonomic drugs and glucose on the insulin and glucose concentrations of sham-operated rats and of rats with ventromedial hypothalamic (VMH) lesions and obesity. In the basal condition both epinephrine and atropine significantly decreased serum insulin levels in VMH-lesioned but not sham-operated rats. During glucose stimulation of insulin secretion in VMH-lesioned rats, epinephrine inhibited the increase of insulin by 83% and atropine inhibited it by 42%; whereas in sham-operated rats, epinephrine inhibited it by 70% and atropine inhibited it by 34%. Epinephrine with atropine completely blocked the increase of insulin in response to glucose in both VMH-lesioned and sham-operated rats. In the basal condition, epinephrine together with propranolol significantly decreased serum insulin levels in VMH-lesioned but not sham-operated rats. Epinephrine with phentolamine, on the other hand, markedly increased insulin in the VMH-lesioned rats and to a lesser degree in the sham-operated rats. During glucose stimulation epinephrine with propranolol inhibited the increase of insulin in both groups. Epinephrine with phentolamine or isoproterenol markedly increased serum insulin in VMH-lesioned rats. These results suggest that stimulation of the vagus nerve and increased sensitivity of the beta-receptors on the beta-cells of the islet contribute to the development of hyperinsulinemia. The sympathetic contribution may also be through suppression of alpha-adrenergic receptors.

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