Formate and glucose stimulation of methane and hydrogen production in rumen liquor

1990 ◽  
Vol 20 (4) ◽  
pp. 251-254 ◽  
Author(s):  
Jayne E. Ellis ◽  
Alan G. Williams ◽  
David Lloyd

Diabetes ◽  
1980 ◽  
Vol 29 (9) ◽  
pp. 747-751 ◽  
Author(s):  
R. L. Sorenson ◽  
D. V. Lindell ◽  
R. P. Elde


Diabetes ◽  
1989 ◽  
Vol 38 (10) ◽  
pp. 1326-1328 ◽  
Author(s):  
O. Kampe ◽  
A. Andersson ◽  
E. Bjork ◽  
A. Hallberg ◽  
F. A. Karlsson




Diabetologia ◽  
1969 ◽  
Vol 5 (4) ◽  
pp. 260-262 ◽  
Author(s):  
K. Asplound ◽  
S. Westman ◽  
C. Hellerste�m


1999 ◽  
Vol 18 (6) ◽  
pp. 471-479 ◽  
Author(s):  
Etti Ben Shushan ◽  
Erol Cerasi ◽  
Danielle Melloul


1983 ◽  
Vol 245 (3) ◽  
pp. R372-R378 ◽  
Author(s):  
S. Inoue ◽  
Y. S. Mullen ◽  
G. A. Bray

The present study examined the effects of autonomic drugs and glucose on the insulin and glucose concentrations of sham-operated rats and of rats with ventromedial hypothalamic (VMH) lesions and obesity. In the basal condition both epinephrine and atropine significantly decreased serum insulin levels in VMH-lesioned but not sham-operated rats. During glucose stimulation of insulin secretion in VMH-lesioned rats, epinephrine inhibited the increase of insulin by 83% and atropine inhibited it by 42%; whereas in sham-operated rats, epinephrine inhibited it by 70% and atropine inhibited it by 34%. Epinephrine with atropine completely blocked the increase of insulin in response to glucose in both VMH-lesioned and sham-operated rats. In the basal condition, epinephrine together with propranolol significantly decreased serum insulin levels in VMH-lesioned but not sham-operated rats. Epinephrine with phentolamine, on the other hand, markedly increased insulin in the VMH-lesioned rats and to a lesser degree in the sham-operated rats. During glucose stimulation epinephrine with propranolol inhibited the increase of insulin in both groups. Epinephrine with phentolamine or isoproterenol markedly increased serum insulin in VMH-lesioned rats. These results suggest that stimulation of the vagus nerve and increased sensitivity of the beta-receptors on the beta-cells of the islet contribute to the development of hyperinsulinemia. The sympathetic contribution may also be through suppression of alpha-adrenergic receptors.



1986 ◽  
Vol 233 (1) ◽  
pp. 287-289 ◽  
Author(s):  
C S Hii ◽  
J Stutchfield ◽  
S L Howell

The phorbol ester 4 beta-phorbol 12-myristate 13-acetate (PMA), at concentrations of 0.1 microM and above, stimulated secretion of glucagon and of insulin from isolated rat islets of Langerhans incubated in the presence of 5.5 mM-glucose. Stimulation of secretion of both hormones by 1 microM-PMA persisted in the absence of external Ca2+, and could be abolished by incubating the islets at 4 degrees C. These findings suggest a role of protein kinase C in the alpha-cell (and beta-cell) secretory mechanism.



Life Sciences ◽  
2010 ◽  
Vol 87 (23-26) ◽  
pp. 667-671 ◽  
Author(s):  
Mossaad Abdel-Ghany ◽  
Geoffrey W.G. Sharp ◽  
Susanne G. Straub


Sign in / Sign up

Export Citation Format

Share Document