scholarly journals Lung function and ventilatory response to exercise in asymptomatic elite soccer players positive for COVID-19.

Pulmonology ◽  
2021 ◽  
Author(s):  
Adriano Di Paco ◽  
Stefano Mazzoleni ◽  
Michele Vitacca ◽  
Laura Comini ◽  
Nicolino Ambrosino
Keyword(s):  
Lung Function ◽  
Ventilatory Response ◽  
Soccer Players ◽  
Response To Exercise

scholarly journals Ventilatory response to exercise of elite soccer players

2014 ◽  
Vol 9 (1) ◽  
Author(s):  
Adriano Di Paco ◽  
Giosuè A Catapano ◽  
Guido Vagheggini ◽  
Stefano Mazzoleni ◽  
Matteo Levi Micheli ◽  
...  
Keyword(s):  
Ventilatory Response ◽  
Soccer Players ◽  
Response To Exercise

scholarly journals Ventilatory response to exercise of elite soccer players

2014 ◽  
Vol 9 ◽  
Author(s):  
Adriano Di Paco ◽  
Giosuè A. Catapano ◽  
Guido Vagheggini ◽  
Stefano Mazzoleni ◽  
Matteo Levi Micheli ◽  
...  
Keyword(s):  
Maximal Exercise ◽  
Ventilatory Response ◽  
Minute Ventilation ◽  
Lung Function Test ◽  
Peak Oxygen Uptake ◽  
Forced Expiratory Volume ◽  
Soccer Players ◽  
Ventilatory Parameters ◽  
Response To Exercise

Background: The purpose of this study was to evaluate the role of ventilatory parameters in maximal exercise performance in elite soccer players. Methods: From September 2009 to December 2012, 90 elite soccer players underwent evaluation of lung function test and ergospirometry by means of an incremental symptom-limited treadmill test. Results were analyzed according to i) maximal exercise velocity performed (Hi-M: high-performers, >18.65 km/h; Lo-M: low-performers, <18.65 km/h) and ii) usual role in the team. Results: Hi-M showed higher peak minute ventilation (V_ Epeak: 158.3 ± 19.5 vs 148.0 ± 18.54 L/min, p = 0.0203), and forced expiratory volume at first second (5.28 ± 0.50 vs 4.89 ± 0.52 liters, p < 0.001) than Lo-M, independently of playing role. Moreover, a significant correlation between peak oxygen uptake and V_ E (r = 0.57, p < 0.001) was found. Conclusions: Ventilatory response plays a role in the assessment of exercise capacity in elite soccer players.

scholarly journals Ventilatory response to exercise in patients with left-to-right shunts.

Heart ◽  
1966 ◽  
Vol 28 (5) ◽  
pp. 590-598 ◽  
Author(s):  
N Gazetopoulos ◽  
H Davies
Keyword(s):  
Ventilatory Response ◽  
Response To Exercise

Role of ventilatory response to exercise in determining exercise capacity in COPD

1995 ◽  
Vol 79 (6) ◽  
pp. 1870-1877 ◽  
Author(s):  
O. Bauerle ◽  
M. Younes
Keyword(s):  
Body Mass Index ◽  
Oxygen Consumption ◽  
Exercise Capacity ◽  
Body Mass ◽  
Ventilatory Response ◽  
Inspiratory Flow ◽  
Peak Oxygen Consumption ◽  
Chronic Obstructive ◽  
Inspiratory Capacity ◽  
Response To Exercise

The progression of chronic obstructive pulmonary disease (COPD) is generally associated with decreased exercise capacity. Differences in forced expired volume in 1 s (FEV1) among patients account for only a fraction of the variability in maximal oxygen consumption (VO2max). We hypothesized that variability in ventilatory response to exercise and in inspiratory mechanics and body mass index contributes importantly to variability in VO2max in this disease. We analyzed the files of 53 patients with established diagnosis of COPD who underwent a recent symptom-limited exercise test. We used inspiratory capacity and maximum inspiratory flow as measures of variability in inspiratory mechanics. The minute ventilation (VE) at the subject's VO2max was divided by the predicted in a normal subject at the same VO2 to obtain a ratio (VE,max/VE,pred). The ventilatory response during exercise provided the best correlation with peak VO2 (r = 0.62). FEV1 and inspiratory capacity also correlated with peak oxygen consumption but not as well as the ventilatory response (r = 0.49 and r = 0.46, respectively). Maximum inspiratory flow and body mass index showed only weak positive correlations (r = 0.23, not significant). The stepwise analysis generated the following equation: VO2max (%predicted) = (77.26 x VE,pred/VE,max) + [0.45 x FEV1 (%predicted)] - 23.66; r = 0.76, P < 0.001. We conclude that variability in the ventilatory response during exercise is one of the main determinants of variability in exercise capacity in COPD patients.

scholarly journals Neuro-hormonal activation predicts ventilatory response to exercise and functional capacity in patients with heart failure

2005 ◽  
Vol 8 (1) ◽  
pp. 46-53 ◽  
Author(s):  
Claudio Passino ◽  
Roberta Poletti ◽  
Francesca Bramanti ◽  
Concetta Prontera ◽  
Aldo Clerico ◽  
...  
Keyword(s):  
Heart Failure ◽  
Functional Capacity ◽  
Ventilatory Response ◽  
Patients With Heart Failure ◽  
Hormonal Activation ◽  
Response To Exercise

Ventilatory Response to Exercise after Intracardiac Repair of Tetralogy of Fallot

1991 ◽  
Vol 144 (4) ◽  
pp. 833-836 ◽  
Author(s):  
Geordie P. Grant ◽  
Robert P. Garofano ◽  
Anthony L. Mansell ◽  
Harris B. Leopold ◽  
Welton M. Gersony
Keyword(s):  
Tetralogy Of Fallot ◽  
Ventilatory Response ◽  
Intracardiac Repair ◽  
Response To Exercise

Effect of mild hypoxia on ventilation during exercise

1962 ◽  
Vol 17 (2) ◽  
pp. 239-242 ◽  
Author(s):  
Thomas F. Hornbein ◽  
Albert Roos
Keyword(s):  
Steady State ◽  
Ventilatory Response ◽  
Human Subjects ◽  
Sympathetic Innervation ◽  
Mild Degree ◽  
Flow Through ◽  
Neural Discharge ◽  
Response To Exercise ◽  
Mild Hypoxia ◽  
Rest And Exercise

Hypoxia of mild degree (PaOO2 above 60 mm Hg) produces little or no ventilatory response in resting man during the steady state. To evaluate the possibility that the effectiveness of a hypoxic chemoreceptor drive might be enhanced by exercise, the ventilatory response to mild hypoxia was measured in two human subjects during rest and exercise. Though no significant increase in ventilation occurred at rest above a PaOO2 of 60 mm Hg, a decrease in PaOO2from 100 to 94 mm Hg produced a statistically significant increase in steady state ventilation during moderate exercise. In addition, temporary block of the sympathetic innervation to the carotid and aortic bodies in one subject resulted in a diminution of work hyperpnea. This suggests that increased sympathetic tone during exercise, by reducing blood flow through the chemoreceptors, might result in increased neural discharge and hence increased ventilation even though arterial POO2 is the same as at rest. Thus, activity of the chemoreceptors as modified by sympathetic control of their blood supply may be an important determinant of the ventilatory response to exercise. Since work hyperpnea is enhanced by even mild hypoxia, this ventilatory response may be sufficient to initiate respiratory acclimatization to altitudes so low that resting ventilation on acute exposure is unaffected. Submitted on July 31, 1961

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