Postoperative Liver Dysfunction in Living Donors After Left-Sided Graft Hepatectomy: Portal Venous Occlusion of the Medial Segment After Lateral Segmentectomy and Hepatic Venous Congestion After Left Lobe Hepatectomy

2012 ◽  
Vol 44 (2) ◽  
pp. 332-337 ◽  
Author(s):  
K. Kumamoto ◽  
S. Mizuno ◽  
N. Kuriyama ◽  
I. Ohsawa ◽  
M. Kishiwada ◽  
...  
2009 ◽  
Vol 87 (5) ◽  
pp. 698-703 ◽  
Author(s):  
Toru Ikegami ◽  
Mitsuo Shimada ◽  
Satoru Imura ◽  
Yuji Soejima ◽  
Tomoharu Yoshizumi ◽  
...  

1965 ◽  
Vol 209 (6) ◽  
pp. 1106-1114 ◽  
Author(s):  
Hermes A. Kontos ◽  
H. Page Mauck ◽  
John L. Patterson

Reactive hyperemia was studied in the hindlimb and forelimb of 81 anesthetized dogs. Reactive hyperemia volume in response to 5-sec arterial occlusions correlated moderately well with the decrease in intravascular pressure. This correlation was poor for 30-sec arterial occlusions. Venous congestion induced by venous occlusion decreased or abolished the vasodilator response to 5-sec arterial occlusions and decreased the response to 30-sec occlusions. Evidence is presented supporting the view that this effect of venous congestion was related to reduction in the fall in intravascular pressure during arterial occlusion. The response to 30-sec occlusions was decreased during acutely induced hypocapnia and increased following acetazolamide administration. The data support the view that both physical and metabolic factors contribute to the production of reactive hyperemia. They further suggest that the accumulation of CO2 in the ischemic tissues contributes to the production of reactive hyperemia.


1958 ◽  
Vol 194 (1) ◽  
pp. 65-71 ◽  
Author(s):  
F. Eugene Yates ◽  
John Urquhart ◽  
Arthur L. Herbst

Acute hepatic venous congestion severely impairs the enzymatic reduction of ring A of desoxycorticosterone, cortisone and hydrocortisone in male and female rats. Preliminary experiments with aldosterone indicate a similar impairment in metabolism. Sham operations do not have this effect. The defect in the inactivation of these hormones persists for as long as 9 months after the onset of hepatic venous congestion, although some recovery is evident. It is suggested that the secondary aldosteronism of congestive heart failure may be accounted for in part by this enzymatic lesion.


Radiology ◽  
2004 ◽  
Vol 232 (1) ◽  
pp. 173-180 ◽  
Author(s):  
Bong Soo Kim ◽  
Tae Kyoung Kim ◽  
Jung-Sun Kim ◽  
Moon-Gyu Lee ◽  
Jung Hoon Kim ◽  
...  

Kanzo ◽  
1997 ◽  
Vol 38 (5) ◽  
pp. 337-342 ◽  
Author(s):  
Takeshi MORIOKA ◽  
Hiroshi MAKINO ◽  
Kazunori ARAI ◽  
Joji HARATAKE
Keyword(s):  

Hepatology ◽  
2019 ◽  
Vol 70 (3) ◽  
pp. 1085-1086
Author(s):  
Haisu Dai ◽  
Kaicheng Shen ◽  
Xingxing Su ◽  
Yishi Yang ◽  
Yan Jiang ◽  
...  

1957 ◽  
Vol 191 (2) ◽  
pp. 339-341 ◽  
Author(s):  
Wilmot C. Ball ◽  
James O. Davis

Four dogs were studied before and after constriction of the inferior vena cava immediately above the entrance of the adrenolumbar veins. Despite a high venous pressure below the constricting ligature, the dogs failed to show increased aldosterone excretion, sustained sodium retention or ascites formation. These changes, which are observed regularly in dogs with constriction of the inferior vena cava in the thorax, are apparently related to hepatic venous congestion rather than elevated adrenal venous pressure.


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