Effect of glucose on salt gland secretion in the glaucous-winged gull, Larus glaucescens

1976 ◽  
Vol 53 (3) ◽  
pp. 311-312 ◽  
Author(s):  
Maryanne Robinson Hughes
Keyword(s):  
Salt Gland ◽  
Gland Secretion ◽  
Salt Gland Secretion ◽  
Effect Of Glucose ◽  
Larus Glaucescens

The effect of crude oils on salt gland sodium secretion of orally imposed salt loads in glaucous-winged gulls, Larus glaucescens

1978 ◽  
Vol 56 (5) ◽  
pp. 1212-1213 ◽  
Author(s):  
E. H. McEwan
Keyword(s):  
Salt Gland ◽  
Gland Secretion ◽  
Crude Oils ◽  
Salt Gland Secretion ◽  
Larus Glaucescens

Oil had no effect on the salt gland secretion of seawater-adapted gulls loaded orally with 0.5 N NaCl.

Cation content of salt gland secretion and tears in the brolga, Grus rubicundus (Perry) (Aves : Gruidae)

1973 ◽  
Vol 21 (4) ◽  
pp. 515 ◽  
Author(s):  
MR Hughes ◽  
JG Blackman
Keyword(s):  
Salt Gland ◽  
Plasma Potassium ◽  
Sodium Concentration ◽  
Gland Secretion ◽  
Plasma Sodium ◽  
First Report ◽  
Gland Size ◽  
Cation Content ◽  
Salt Gland Secretion ◽  
Sodium And Potassium

This is the first report of salt gland secretion in cranes (Gruidae). The sodium and potassium concentrations of the plasma, tears, salt gland secretion, and urine of the brolga were determined. Tear sodium was equal to plasma sodium; tear plasma was four times as concentrated as plasma potassium. These values were normal for tears. The salt gland secretion sodium concentration (about 300 m-equivll) was lower than that reported for other NaC1-injected birds. This may be due to the diet, small gland size, or to insufficient stress. The salt gland secretion to plasma ratios were the same for sodium and potassium. This is unusual. The urine had a lower sodium concentration than the plasma.

Effect of volume expansion and veratrine on salt gland secretion in the goose

1977 ◽  
Vol 232 (5) ◽  
pp. R185-R189
Author(s):  
I. H. Zucker ◽  
C. Gilmore ◽  
J. Dietz ◽  
J. P. Gilmore
Keyword(s):  
Volume Expansion ◽  
Plasma Osmolality ◽  
Salt Gland ◽  
Sodium Concentration ◽  
Ringer Solution ◽  
Gland Secretion ◽  
Similar Increase ◽  
Volume Load ◽  
Salt Gland Secretion ◽  
Peripheral Receptor

The influence of acute intravascular volume expansion on salt gland secretion of conscious, adult geese was investigated. The intravenous administration of 5% dextran in Krebs-bicarbonate-Ringer solution in an amount equivalent to 30% of the estimated blood volume caused a transient but highly significant increase in salt gland secretion independent of changes in plasma osmolality or sodium concentration. Intravenous veratrine (60 microng) caused a similar increase in salt gland secretion only when administered after the volume load. Intravenous 5% NaCl always caused a prolonged and significant increase in salt gland secretion which was not potentiated by veratrine. Volume expansion and hypertonic saline caused a significant tachycardia while veratrine caused a significant bradycardia. It is concluded that a volume component may contribute to the initiation of salt gland secretion in the goose and that the peripheral receptor involved is most likely vascular in origin.

CONTROL OF THE SALT-SECRETING GLAND OF THE DUCK: I. OSMOTIC REGULATION

1967 ◽  
Vol 45 (6) ◽  
pp. 925-936 ◽  
Author(s):  
André Lanthier ◽  
Thomas Sandor
Keyword(s):  
Sodium Chloride ◽  
Sodium Bicarbonate ◽  
Intravenous Infusion ◽  
Electrolyte Concentration ◽  
Salt Gland ◽  
The Other ◽  
Gland Secretion ◽  
Osmotic Regulation ◽  
Domestic Duck ◽  
Salt Gland Secretion

The osmotic regulation of the salt gland secretion was studied in the domestic duck. The response to the intravenous infusion of 5% sodium chloride (850 meq Na per liter) was established in unanesthetized ducks. Sodium bicarbonate (892 meq Na/l) produced a salt gland secretion similar to that after 5% sodium chloride, except that it was accompanied by a moderate diuresis. Mannitol and sucrose had similar effects. On the other hand, urea, ammonium chloride, dextran, and meralluride produced only a small amount of salt-gland secretion of low electrolyte concentration. Antidiuretic doses of Pitressin did not induce secretion of the salt gland.

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