622 THE ROLE OF EPHRIN A2 RECEPTOR AND ARACHIDONIC ACID IN THE DEVELOPMENT OF PROSTATE CANCER METASTASIS

2011 ◽  
Vol 10 (2) ◽  
pp. 202
Author(s):  
T. Tawadros ◽  
C.A. Hart ◽  
M.D. Brown ◽  
N.W. Clarke
Keyword(s):  
Prostate Cancer ◽  
Arachidonic Acid ◽  
Cancer Metastasis ◽  
Prostate Cancer Metastasis

scholarly journals Role of exosomal small RNA in prostate cancer metastasis

2018 ◽  
Vol Volume 10 ◽  
pp. 4029-4038 ◽  
Author(s):  
Fei Zhan ◽  
Jingling Shen ◽  
Ruitao Wang ◽  
Liang Wang ◽  
Yao Dai ◽  
...  
Keyword(s):  
Prostate Cancer ◽  
Small Rna ◽  
Cancer Metastasis ◽  
Prostate Cancer Metastasis

280 ARACHIDONIC ACID PRIMES HUMAN BONE MARROW STROMA FOR PROSTATE CANCER METASTASIS

2010 ◽  
Vol 9 (2) ◽  
pp. 115
Author(s):  
M.D. Brown ◽  
C.A. Hart ◽  
E. Gazi ◽  
P. Gardner ◽  
N.W. Clarke
Keyword(s):  
Prostate Cancer ◽  
Bone Marrow ◽  
Arachidonic Acid ◽  
Cancer Metastasis ◽  
Human Bone ◽  
Human Bone Marrow ◽  
Bone Marrow Stroma ◽  
Prostate Cancer Metastasis ◽  
Marrow Stroma

scholarly journals New insights into the role of CXCR4 in prostate cancer metastasis

2008 ◽  
Vol 7 (11) ◽  
pp. 1849-1851 ◽  
Author(s):  
Candece L. Gladson ◽  
Danny R. Welch
Keyword(s):  
Prostate Cancer ◽  
Cancer Metastasis ◽  
Prostate Cancer Metastasis

scholarly journals Role of αVβ3 in Prostate Cancer: Metastasis Initiator and Important Therapeutic Target

2020 ◽  
Vol Volume 13 ◽  
pp. 7411-7422 ◽  
Author(s):  
Lin Tang ◽  
Meng Xu ◽  
Long Zhang ◽  
Lin Qu ◽  
Xiaoyan Liu
Keyword(s):  
Prostate Cancer ◽  
Therapeutic Target ◽  
Cancer Metastasis ◽  
Prostate Cancer Metastasis ◽  
Important Therapeutic Target

scholarly journals Role of splice variants in the metastatic progression of prostate cancer

2012 ◽  
Vol 40 (4) ◽  
pp. 870-874 ◽  
Author(s):  
Rachel M. Hagen ◽  
Michael R. Ladomery
Keyword(s):  
Prostate Cancer ◽  
Cancer Metastasis ◽  
Splice Variants ◽  
Human Cancer ◽  
Metastatic Potential ◽  
Metastatic Progression ◽  
Cellular Phenotype ◽  
Prostate Cancer Metastasis ◽  
The Relationship

AS (alternative splicing) and its role in disease, especially cancer, has come to forefront in research over the last few years. Alterations in the ratio of splice variants have been widely observed in cancer. Splice variants of cancer-associated genes have functions that can alter cellular phenotype, ultimately altering metastatic potential. As metastases are the cause of approximately 90% of all human cancer deaths, it is crucial to understand how AS is dysregulated in metastatic disease. We highlight some recent studies into the relationship between altered AS of key genes and the initiation of prostate cancer metastasis.

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