The evaluation of facial nerve canal diameter in patients with ipsilateral recurrent idiopathic peripheral facial paralysis

Author(s):  
O Yagiz ◽  
Y Pekcevik ◽  
Y Arslan ◽  
I B Arslan ◽  
I Cukurova
2018 ◽  
Vol 2018 ◽  
pp. 1-3
Author(s):  
Behiye Deniz Kosovali ◽  
Asiye Yavuz ◽  
Fatma Irem Yesiler ◽  
Mustafa Kemal Bayar

Melkersson-Rosenthal Syndrome (MRS) is a rare disease characterized by persistent or recurrent orofacial oedema, relapsing peripheral facial paralysis, and furrowed tongue. Pathologically, granulomatosis is responsible for oedema of face, labia, oral cavity, and facial nerve. We present a patient with MRS admitted to our hospital with acute respiratory distress syndrome (ARDS). 45-year-old woman was admitted to an emergency department with dyspnea and swelling on her hands and face. She was intubated because of ARDS and accepted to intensive care unit (ICU). After weaning from ventilatory support, peripheral facial paralysis was diagnosed and steroid treatment was added to her therapy. On dermatologic examination, oedema on her face, pustular lesions on her skin, and fissure on her tongue were detected. The patient informed us about her recurrent and spontaneous facial paralysis in previous years. According to her history and clinical findings, MRS was diagnosed.


2002 ◽  
Vol 23 (Sup 1) ◽  
pp. S36-S37
Author(s):  
Sertac Yetiser ◽  
Mustafa Kazkayasi ◽  
Deniz Altinok ◽  
Yasemin Karadeniz

2020 ◽  
Vol 13 (8) ◽  
pp. e237146
Author(s):  
Rita Figueiredo ◽  
Vera Falcão ◽  
Maria João Pinto ◽  
Carla Ramalho

Acute facial nerve disease leading to peripheral facial paralysis is commonly associated with viral infections. COVID-19 may be a potential cause of peripheral facial paralysis and neurological symptoms could be the first and only manifestation of the disease. We report a case of a term pregnancy diagnosed with COVID-19 after presenting with isolated peripheral facial palsy.


2014 ◽  
Vol 2014 ◽  
pp. 1-3 ◽  
Author(s):  
Serhan Derin ◽  
Hatice Derin ◽  
Murat Sahan ◽  
Hüseyin Çaksen

Ramsay Hunt syndrome (RHS) is characterized by facial paralysis, inner ear dysfunction, periauricular pain, and herpetiform vesicles. The reported incidence in children is 2.7/100,000. The pathogenesis involves the reactivation of latent varicella zoster virus (VZV) in the geniculate ganglion of the facial nerve. The recovery rate is better in children than in adults. This paper discusses a 12-year-old girl with a rare case of peripheral facial paralysis caused by RHS and reviews the literature.


2021 ◽  
Author(s):  
Maurus Marques de Almeida Holanda Filho ◽  
Maurus Marques de Almeida Holanda ◽  
Camila Maria Bezerra Holanda

Background: Ramsay Hunt syndrome or herpes zoster oticus is a disorder caused by the reactivation of the varicella zoster virus in the geniculate ganglion, a bundle of nerve cells in the facial nerve. Symptoms include acute facial nerve palsy, otalgia, loss of taste in the anterior 2/3 of the tongue, dry mouth and eyes, and erythematous vesicular rash in the ear canal, tongue and / or palate. Objectives and Methods: Describe the case of a male patient, 26 years old, with complete Ramsay Hunt syndrome, reporting pain in the right ear with tinnitus, difficult to close his right eye, odynophagia and pain in the anterior 2/3 of the tongue at right side. Results: On examination, the presence of vesicles with erythema was observed in the region of the right external auditory canal and the ear, as well as on the right palate and tongue. Upon inspection, he had peripheral facial paralysis on the right, associated with loss of taste in the anterior 2/3 of the tongue. The patient was treated with antiviral and corticosteroids, followed for 3 months, obtaining partial recovery from facial paralysis. The anatomy of the facial nerve and its pathophysiology due to the involvement of herpes zoster will be discussed. Conclusion: Ramsay Hunt syndrome is often described as the presence of peripheral facial paralysis and vesicles in the auditory canal and the ear. However, this rare case presented the complete condition with the presence of vesicles on the palate and the tongue.


2021 ◽  
pp. 019459982110321
Author(s):  
Giovanna Cantarella ◽  
Riccardo F. Mazzola

Charles Bell was a talented and versatile Scottish anatomist, neurophysiologist, artist, and surgeon. On July 12, 1821, he reported his studies regarding facial innervation in the essay “On the Nerves,” read before the Royal Society in London. Since then, idiopathic peripheral facial paralysis has been named “Bell’s palsy.” He was the first author to describe the neuroanatomical basis of facial paralysis, in an essay enriched by beautifully self-made illustrations. The aim of this article is to trace the history of Bell’s description of the neuroanatomy of the facial nerve, reexamining his 1821 article, in which he stated that the lower facial expression muscles were dually innervated by both the fifth and seventh cranial nerves. In 1829, he rectified this conclusion, recognizing the exclusive role of the facial nerve, which he defined as the “respiratory nerve.” We offer a tribute to this polymath scientist on the bicentenary of his 1821 publication.


2009 ◽  
Vol 123 (11) ◽  
pp. 1193-1198 ◽  
Author(s):  
R C van de Graaf ◽  
F F A IJpma ◽  
J-P A Nicolai ◽  
P M N Werker

AbstractBell's palsy is the eponym for idiopathic peripheral facial paralysis. It is named after Sir Charles Bell (1774–1842), who, in the first half of the nineteenth century, discovered the function of the facial nerve and attracted the attention of the medical world to facial paralysis. Our knowledge of this condition before Bell's landmark publications is very limited and is based on just a few documents. In 1804 and 1805, Evert Jan Thomassen à Thuessink (1762–1832) published what appears to be the first known extensive study on idiopathic peripheral facial paralysis. His description of this condition was quite accurate. He located several other early descriptions and concluded from this literature that, previously, the condition had usually been confused with other afflictions (such as ‘spasmus cynicus’, central facial paralysis and trigeminal neuralgia). According to Thomassen à Thuessink, idiopathic peripheral facial paralysis and trigeminal neuralgia were related, being different expressions of the same condition. Thomassen à Thuessink believed that idiopathic peripheral facial paralysis was caused by ‘rheumatism’ or exposure to cold. Many aetiological theories have since been proposed. Despite this, the cold hypothesis persists even today.


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