Inferring the Subsurface Geometry and Strength of Slow‐moving Landslides using 3D Velocity Measurements from the NASA/JPL UAVSAR

2021 ◽  
Author(s):  
A. L. Handwerger ◽  
A. M. Booth ◽  
M.‐H. Huang ◽  
E. J. Fielding
Keyword(s):  
Velocity Measurements ◽  
Slow Moving ◽  
Subsurface Geometry

scholarly journals Velocity measurements on Engabreen, Norway

2005 ◽  
Vol 42 ◽  
pp. 29-34 ◽  
Author(s):  
Miriam Jackson ◽  
Ian A. Brown ◽  
Hallgeir Elvehøy
Keyword(s):  
Aerial Photography ◽  
Satellite Images ◽  
Horizontal Velocity ◽  
Aerial Photographs ◽  
Velocity Measurements ◽  
Measurable Velocity ◽  
Slow Moving

AbstractHorizontal velocity measurements on the lower part of Engabreen, Norway, were made from repeat aerial photography. IMCORR software, which has been widely used to measure velocities from satellite images, was used to make the measurements. This is the first known successful use of IMCORR on aerial photographs. Supplementary horizontal velocity measurements were made from repeat measurements of stakes, giving velocities over different periods and also in areas that are too slow-moving to register a measurable velocity after only a few days.

Timing in Rather Slow-Moving Pottos and Other Timely Issues

1983 ◽  
Vol 28 (11) ◽  
pp. 848-849
Author(s):  
Charles P. Shimp
Keyword(s):  
Slow Moving

Antithrombin III Alger: A New Homozygous AT III Variant

1986 ◽  
Vol 55 (02) ◽  
pp. 218-221 ◽  
Author(s):  
A M Fischer ◽  
P Cornu ◽  
C Sternberg ◽  
F Mériane ◽  
M D Dautzenberg ◽  
...  
Keyword(s):  
Family History ◽  
Antithrombin Iii ◽  
Antigen Concentration ◽  
Crossed Immunoelectrophoresis ◽  
The Family ◽  
At Iii ◽  
Molecular Abnormality ◽  
Slow Moving ◽  
Cofactor Activity ◽  
Plasma Heparin

SummaryA qualitative abnormality of antithrombin III (AT III) was found in the plasma of a 41-year old patient. The plasmatic AT III antigen concentration was 130% and the progressive anti-F IIa and anti-F Xa activities were normal (105% and 137%). The plasma heparin cofactor activity was less than 10%, when measured by F Ila or F Xa inhibition. Crossed immunoelectrophoresis of AT III in the presence of heparin revealed in the plasma an abnormal slow-moving peak. When tested by affinity chromatography on heparin Sepharose, this abnormal AT III did not bind to heparin. Among the investigated relatives, 5 subjects had normal AT III levels, whatever the test used, the nine others having reduced levels of antithrombin heparin cofactor activity (45-61%) but normal levels of immunoreactive AT III (97-122%). Consanguinity was found in the family history. We therefore considered our patient as homozygous for an AT III molecular abnormality affecting the binding site for heparin.

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