scholarly journals Binary Bose-Einstein condensates in a disordered time-dependent potential

2021 ◽  
Author(s):  
Karima Abbas ◽  
Abdelaali Boudjemaa
Keyword(s):  
Time Evolution ◽  
Random Potential ◽  
Time Dependent ◽  
Interspecies Interactions ◽  
Closed Set ◽  
System Parameters ◽  
Dependent Potential ◽  
Time Propagation ◽  
Bose Einstein

Abstract We study the non-equilibrium evolution of binary Bose-Einstein condensates in the presence of weak random potential with a Gaussian correlation function using the time-dependent perturbation theory. We apply this theory to construct a closed set of equations that highlight the role of the spectacular interplay between the disorder and the interspecies interactions in the time evolution of the density induced by disorder in each component. It is found that this latter increases with time favoring localization of both species. The time scale at which the theory remains valid depends on the respective system parameters. We show analytically and numerically that such a system supports a steady state that periodically changing during its time propagation. The obtained dynamical corrections indicate that disorder may transform the system into a stationary out-of-equilibrium states. Understanding this time evolution is pivotal for the realization of Floquet condensates.

Nonlinear tunneling and chaos between two Bose–Einstein condensates trapped in time-dependent potential

2001 ◽  
Vol 291 (1) ◽  
pp. 17-21 ◽  
Author(s):  
B.-Y. Ou ◽  
X.-G. Zhao ◽  
J. Liu ◽  
S.-G. Chen
Keyword(s):  
Time Dependent ◽  
Dependent Potential ◽  
Bose Einstein

scholarly journals The time-dependent angular analysis of Bd → KSℓℓ, a new benchmark for new physics

2021 ◽  
Vol 2021 (2) ◽  
Author(s):  
Sébastien Descotes-Genon ◽  
Martín Novoa-Brunet ◽  
K. Keri Vos
Keyword(s):  
Time Evolution ◽  
Form Factors ◽  
New Physics ◽  
Time Dependent ◽  
Angular Analysis ◽  
Time Dependent Analysis ◽  
Tensor Operators

Abstract We consider the time-dependent analysis of Bd→ KSℓℓ taking into account the time-evolution of the Bd meson and its mixing into $$ {\overline{B}}_d $$ B ¯ d . We discuss the angular conventions required to define the angular observables in a transparent way with respect to CP conjugation. The inclusion of time evolution allows us to identify six new observables, out of which three could be accessed from a time-dependent tagged analysis. We also show that these observables could be obtained by time-integrated measurements in a hadronic environment if flavour tagging is available. We provide simple and precise predictions for these observables in the SM and in NP models with real contributions to SM and chirally flipped operators, which are independent of form factors and charm-loop contributions. As such, these observables provide robust and powerful cross-checks of the New Physics scenarios currently favoured by global fits to b → sℓℓ data. In addition, we discuss the sensitivity of these observables with respect to NP scenarios involving scalar and tensor operators, or CP-violating phases. We illustrate how these new observables can provide a benchmark to discriminate among the various NP scenarios in b → sμμ. We discuss the extension of these results for Bs decays into f0, η or η′.

scholarly journals Endothelial cell-related autophagic pathways in Sugen/hypoxia-exposed pulmonary arterial hypertensive rats

2017 ◽  
Vol 313 (5) ◽  
pp. L899-L915 ◽  
Author(s):  
Fumiaki Kato ◽  
Seiichiro Sakao ◽  
Takao Takeuchi ◽  
Toshio Suzuki ◽  
Rintaro Nishimura ◽  
...  
Keyword(s):  
Flow Cytometry ◽  
Endothelial Cell ◽  
Vascular Remodeling ◽  
Time Dependent ◽  
Causative Role ◽  
Dependent Manner ◽  
Vascular Endothelial ◽  
Pulmonary Vascular Remodeling ◽  
Pulmonary Arterial

Pulmonary arterial hypertension (PAH) is characterized by progressive obstructive remodeling of pulmonary arteries. However, no reports have described the causative role of the autophagic pathway in pulmonary vascular endothelial cell (EC) alterations associated with PAH. This study investigated the time-dependent role of the autophagic pathway in pulmonary vascular ECs and pulmonary vascular EC kinesis in a severe PAH rat model (Sugen/hypoxia rat) and evaluated whether timely induction of the autophagic pathway by rapamycin improves PAH. Hemodynamic and histological examinations as well as flow cytometry of pulmonary vascular EC-related autophagic pathways and pulmonary vascular EC kinetics in lung cell suspensions were performed. The time-dependent and therapeutic effects of rapamycin on the autophagic pathway were also assessed. Sugen/hypoxia rats treated with the vascular endothelial growth factor receptor blocker SU5416 showed increased right ventricular systolic pressure (RVSP) and numbers of obstructive vessels due to increased pulmonary vascular remodeling. The expression of the autophagic marker LC3 in ECs also changed in a time-dependent manner, in parallel with proliferation and apoptotic markers as assessed by flow cytometry. These results suggest the presence of cross talk between pulmonary vascular remodeling and the autophagic pathway, especially in small vascular lesions. Moreover, treatment of Sugen/hypoxia rats with rapamycin after SU5416 injection activated the autophagic pathway and improved the balance between cell proliferation and apoptosis in pulmonary vascular ECs to reduce RVSP and pulmonary vascular remodeling. These results suggested that the autophagic pathway can suppress PAH progression and that rapamycin-dependent activation of the autophagic pathway could ameliorate PAH.

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