Facial Nerve Paralysis

Author(s):  
Donald W. Winnicott

In this case, Winnicott presents the history of facial nerve paralysis in a very young child. In this case, the symptoms gradually fade and appear not to return.

Author(s):  
Donald W. Winnicott

In this case, Winnicott presents the family history of a seven-year-old, one of twins suffering from facial nerve paralysis associated with fits.


1989 ◽  
Vol 103 (8) ◽  
pp. 768-770 ◽  
Author(s):  
Ronald A. Hoffman ◽  
Bruce Horten

AbstractA 69-year-old woman with severe rheumatoid arthritis presented with a history of chronic otitis and a facial nerve paralysis. She was found to have a rheumatoid nodule involving the mastoid and mesotympanum. This is believed to be the first reported case of a rheumatoid nodule involving the temporal bone.


Author(s):  
Ignacio Calvo ◽  
Irene Espadas ◽  
Gawain Hammond ◽  
Kathryn Pratschke

A 7-year-old male entire West Highland white terrier was referred to the Small Animal Hospital at the University of Glasgow for bilateral, chronic, medically unresponsive otitis media and externa. A history of cranio-mandibular osteopathy was also reported. Bilateral total ear canal ablation and lateral bulla osteotomy was performed with the aid of a pneumatic burr. Extensive bone proliferation was present bilaterally originating from the caudal mandibular ramus and tympanic bulla which incorporated the horizontal canal on each side. The right facial nerve was identified leaving the stylomastoid foramen and running in a cranial direction through a 1.5 cm diameter cuff of bone surrounding the horizontal canal and external acoustic meatus. Despite careful dissection, a facial nerve neurotmesis ensued which required microsurgical epineurial repair. Neurologic examination performed 12 h post-operatively revealed abnormalities consistent with right facial nerve paralysis. At 3 months, the facial nerve function was found to have improved significantly and was assessed to be normal four months after surgery. To the authors’ knowledge, this clinical communication described the first reported clinical case where unilateral facial nerve paralysis resulting from iatrogenic facial nerve neurotmesis was successfully treated by microsurgical epineurial repair.


2020 ◽  
pp. 239719832095689
Author(s):  
Catherine B Xie ◽  
Shawn Cowper ◽  
Ian D Odell

Morphea, also known as localized scleroderma, is characterized by inflammation and fibrosis of the skin. The exact pathogenesis of morphea is unknown, but generally includes genetic predisposition to autoimmunity combined with an environmental insult. Previous cases have been associated with active Borrelia infection; however, Borrelia infection as a direct cause of morphea was not generalizable to most patients. Within endemic areas, Borrelia burgdorferi is the most common cause of facial nerve paralysis, another autoimmune phenomenon. We report a case of facial morphea in a young man with family history of autoimmune disease who developed morphea in the same location as two previous episodes of Borrelia-induced facial nerve palsy. This case is remarkable because it suggests Borrelia burgdorferi induced loss of local immune tolerance to host antigens, first with facial nerve palsy and followed years later by development of morphea.


2019 ◽  
Vol 13 (3) ◽  
pp. 44-48
Author(s):  
B K Bhattacharya ◽  
◽  
Subhajit Sarkar ◽  

2019 ◽  
Vol 6 ◽  
pp. 52
Author(s):  
Yayun Siti Rochmah

Background: Chronic osteomyelitis mandibula is one of the complications from dental extraction. Inadequate wound handling can have an impact on the spread of infection in the surrounding tissue like nerve which results in facial nerve paralysis. The purpose is to present a rare case that facilitative nerve paralysis as a result of the spread of osteomyelitis infectionCase Management: A 69 years old woman with chief complains numbness onher lips accompanied by pus out beside the lower teeth. No sistemic disease. Panoramic radiograph showed abnormal bone-like sequester. Extraoral examination appeared the bluish color on the right cheek and there was right facial muscle paralysis. Debridement, sequesterectomy by general anesthesia and medication using ceftriaxone intravenous, ketorolac injection, multivitamin, and corticosteroid, physiotherapy for facial nerve paralyze, also.Discussion: Pathogenesis mandibular osteomyelitis involves contiguous spreadfrom an odontogenic focus infection. The bacteria produce an exotoxin, which, while unable to cross the blood-brain barrier, can have deleterious effects on thePeripheral Nerve System (Fasialis Nerve) in up to 75% of cases, with the severity of presentation correlating with the severity of the infection.Conclusion: Chronic mandibular osteomyelitis can spread the infection to around another anatomy oral cavity like facials nerves.


1993 ◽  
Vol 103 (12) ◽  
pp. 1326???1333 ◽  
Author(s):  
John R. Austin ◽  
Steven P. Peskind ◽  
Sara G. Austin ◽  
Dale H. Rice

2017 ◽  
Vol 69 (1) ◽  
pp. 58-61 ◽  
Author(s):  
Sriranga Prasad ◽  
K. V. Vishwas ◽  
Swetha Pedaprolu ◽  
R. Kavyashree

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